Abnormal muscle tone/spasticity Flashcards
flaccidity
complete lack of resistance to passive stretch
hypotonia
abnormally low/less than normal resistance to passive stretch
hypertonia subtypes
spasticity + rigidity
spasticity vs rigidity
spasticity is velocity dependent and rigidity is velocity independent
LMNL typically present as hyper or hypotonia
hypotonia or flaccidity
myasthenia gravis presents as hyper or hypotonia
hypotonia
when can UMNL present with flaccidity or hypotonia
during spinal shock (temporary state)
however, it then presents as hypertonic after a few months
UMNL typically present as hyper or hypotonia?
hypertonia
is cogwheel part of spasticity or rigidity
rigidity
cogwheel
start-stop resistance to movement as limb is passively moved through ROM
lead-pipe
constant resistance to movement throughout ROM
gegenhalten (and where is it most seen)
involuntary resistance to passive movement
seen in dyspraxia, apraxia, dementia
decerebrate rigidity positioning vs decorticate
decerebrate: elbow EXTENSION
decorticate: elbow FLEXION
decerebrate rigidity cause
damage of brainstem between midbrain and pons
decorticate rigidity cause
damage of superior midbrain
or
severe bilateral lesions of cerebral cortex
how does an EMG look in spasticity
it INCREASES linearly with speed of movement
clonus
involuntary, repetitive rhythmic reflex contraction of a single muscle group
clasp knife phenomenon with catch/give
initial resistance to passive stretch/movement followed by decreased resistance as movement continues
is all spasticity bad?
no, can help with posture, muscle mass, gait, etc.
what type of neurophysiological contribution of spasticity involves pyramidal tracts and extrapyramidal tracts?
supra-segmental contribution
how do pyramidal tracts contribute to spasticity
inhibition not working on the corticospinal tract causes exaggerated movement and tone
how do extrapyramidal tracts like DRT, VMRST, and VST contribute to spasticity
DRT = inhibitory center
VMRST + VST = excitatory centers
what 2 areas of the cortex must simultaneously have a legion to produce spasticity
primary motor cortex
premotor cortex
what happens when a pt has a legion only in the primary motor cortex
weakness and decreased reflexes