Abnormal muscle tone/spasticity Flashcards

1
Q

flaccidity

A

complete lack of resistance to passive stretch

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2
Q

hypotonia

A

abnormally low/less than normal resistance to passive stretch

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3
Q

hypertonia subtypes

A

spasticity + rigidity

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4
Q

spasticity vs rigidity

A

spasticity is velocity dependent and rigidity is velocity independent

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5
Q

LMNL typically present as hyper or hypotonia

A

hypotonia or flaccidity

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6
Q

myasthenia gravis presents as hyper or hypotonia

A

hypotonia

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7
Q

when can UMNL present with flaccidity or hypotonia

A

during spinal shock (temporary state)

however, it then presents as hypertonic after a few months

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8
Q

UMNL typically present as hyper or hypotonia?

A

hypertonia

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9
Q

is cogwheel part of spasticity or rigidity

A

rigidity

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10
Q

cogwheel

A

start-stop resistance to movement as limb is passively moved through ROM

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11
Q

lead-pipe

A

constant resistance to movement throughout ROM

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12
Q

gegenhalten (and where is it most seen)

A

involuntary resistance to passive movement

seen in dyspraxia, apraxia, dementia

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13
Q

decerebrate rigidity positioning vs decorticate

A

decerebrate: elbow EXTENSION
decorticate: elbow FLEXION

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14
Q

decerebrate rigidity cause

A

damage of brainstem between midbrain and pons

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15
Q

decorticate rigidity cause

A

damage of superior midbrain
or
severe bilateral lesions of cerebral cortex

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16
Q

how does an EMG look in spasticity

A

it INCREASES linearly with speed of movement

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17
Q

clonus

A

involuntary, repetitive rhythmic reflex contraction of a single muscle group

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18
Q

clasp knife phenomenon with catch/give

A

initial resistance to passive stretch/movement followed by decreased resistance as movement continues

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19
Q

is all spasticity bad?

A

no, can help with posture, muscle mass, gait, etc.

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20
Q

what type of neurophysiological contribution of spasticity involves pyramidal tracts and extrapyramidal tracts?

A

supra-segmental contribution

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21
Q

how do pyramidal tracts contribute to spasticity

A

inhibition not working on the corticospinal tract causes exaggerated movement and tone

22
Q

how do extrapyramidal tracts like DRT, VMRST, and VST contribute to spasticity

A

DRT = inhibitory center
VMRST + VST = excitatory centers

23
Q

what 2 areas of the cortex must simultaneously have a legion to produce spasticity

A

primary motor cortex
premotor cortex

24
Q

what happens when a pt has a legion only in the primary motor cortex

A

weakness and decreased reflexes

25
a legion to what part of the internal capsule can cause spasticity
ANTERIOR limb
26
the ventromedial reticular formation is also known as the....
inhibitory system
27
where is the ventromedial reticular formation located? what controls it?
medulla premotor cortex
28
function of ventromedial reticular formation
inhibit spinal stretch reflex via descending output of dorsal (lateral) reticulospinal tract
29
excitatory system FACILITATES _____ and _____, while INHIBITING ______
facilitates = stretch reflex + extensor tone inhibits = flexors
30
lesions of primary motor cortex must also involve ____(2)____ to cause spasticity
premotor and supplementary motor areas of the brain
31
what type of neurophysiological contribution of spasticity involves renshaw cell inhibition
segmental spinal contribution
32
with increased fusimotor drive, there is often an overactive ____ motor system. what does this cause?
gamma (Y) this causes muscles to respond excessively to stimuli
33
spinal shock
within 24 hrs of a SCI, no DTR can be elicited
34
gamma (Y) motor neurons are also known as
fusimotor neurons
35
1a presynaptic inhibition
constant release of GABA decreases 1a afferent ability to release neurotransmitters
36
how is immobilization beneficial in 1a presynaptic inhibition
casting can help reduce spasticity by long-term stretching out the spastic muscle
37
1a reciprocal inhibition
alpha MNs cant tell antagonist muscle to relax this inhibits co-contraction relationship of the agonist/antagonist
38
1b non-reciprocal inhibition (autogenic inhibition)
GTOs are unable to communicate to its muscle belly to relax when the tendon is being stretched
39
renshaw cell (interneuron) inhibition
inhibits gamma (Y) MNs and 1a interneurons (recurrent inhibition)
40
what neurotransmitter excites renshaw cells
ACh
41
neuroplasticity
functional reorganization within spinal circuits
42
UMN injury neuroplasticity presentation
abnormal motor control to try to adapt to new circumstances --> contributes to spasticity
43
what type of changes can occur to constantly contracted soft tissue
contractures (which WORSENS spasticity)
44
non-nociceptive stimuli that can trigger spasticity
touch/pressure (cutaneous) bladder, bowel, infection, etc (visceral)
45
what tool is most commonly used to measure spasticity
Modified Ashworth Scale (MAS)
46
1 vs 1+ on MAS
1 releases or has minimal resistance through end ROM vs 1+ has minimal resistance throughout the remainder of ROM (no release)
47
2 on MAS
increased muscle tone through most ROM but part is easily moved
48
4 on MAS
affected part is rigid in flexion or extension
49
3 on MAS
considerable increase in muscle tone AND pROM is difficult
50
spasticity presentation on NCVs vs EMGs
NCVs show normal EMGs show increase (measures central issues)