9 Flashcards

1
Q

Describe the orbital cavity

A
  • pyramid shaped with apex pointing posteriorly
  • four bony walls: frontal bone, sphenoid bone, zygomatic bone, thmoid bone, lacrimal bone, palatine bone

See S9 anatomy slide 3

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2
Q

What is the weakest part of the orbit, and what happens if it is fractured?

A
  • inferior (floor) and medial walls
  • vulnerable to fracture
  • leads to sudden increase in intraorbital pressure
  • often the floor that fractures (orbital blow-out fracture)
  • orbital contents can bleed into maxillary sinus and prevent upward gaze on the affect side
  • pt. Will have history of trauma to eye, periorbital swelling, painful, double vision, numbness over cheek, lower eyelid and upper lip (b/c infraorbital nerve is affected)
  • usually not medial wall because ethmoid air cells act as buttresses and give extra strength to the medial wall but the air cells are prone to infection which can cause orbital cellulitis

See S9 anatomy slide 6-7

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3
Q

What are the blood vessels and nerves of the orbit and what openings do they go through?

A
  • main arterial supply is ophthalmic artery (from internal carotid) and its subsequent branches and it goes through optic canal
  • opthalmic veins (superior (superior orbital fissure) and inferior (inferior orbital fissure)) drain venous blood into cavernous sinus, pterygoid plexus and facial vein
  • general sensory from the eye (including conjunctiva, cornea): trigeminal ophthalmic division which goes through superior orbital fissure
  • special sensory vision from retina: optic nerve which goes through optic canal
  • motor nerve to muscles: oculomotor, trochlear, abducens which all go through superior orbital fissure

See S9 anatomy slide 4

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4
Q

What are the anatomical relation for the orbit and their implications?

A
  • paranasal air sinuses (maxillary and ethmoid)
  • nasal cavity (nasolacrimal duct, connects it to orbital fossa)
  • anterior cranial fossa

Implications for

  • orbital trauma
  • spread of infection

See S9 anatomy slide 5

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5
Q

Describe the eyelid

A
  • consists of skin, subcutaneous tissue, tarsal plate, muscles
  • tarsal plates: dense CT which strengthens the eyelid and gives their shape, contains tarsal glands
  • contains lacrimal glands which protect eye and prevent it from drying out
  • muscles: orbicularis oculi (palpebral part) which closes eyelid and levator palpebrae superioris which retracts eyelid
  • contains glands: meibomian glands which secrete oily substance that lubricate the eyelid and prevent tears evaportating too quickly and sebaceous glands associated with lash follicle

See S9 anatomy slide 8

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6
Q

What pathologies can occur in the eyelid?

A

Stye

  • if the glands or hair follicles are blocked then a stye is produced
  • they are a self-limiting so can use a warm compress to resolve it
  • more superficial and close to edge
  • painful

Meibomian cyst

  • occurs when meibomian gland is blocked
  • oily secretions are built up into a lump
  • deeper within eyelid

Blepharitis

  • inflammation of edges of the lid
  • pt. Will experience crusting
  • includes skin, lashes and meibomian glands

See S9 anatomy slide 9

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7
Q

Describe the orbital septum

A
  • thin sheet of fibrous tissue originating from orbital rim periosteum bleeds with tarsal plates
  • directly underneath the orbicularis oculi
  • orbital septum and tarsal plates separate subcutaneous tissue or eyelid and orbicularis oculi form intra-orbital contents
  • acts as a barrier against superficial infection spreading from the pre-septal to post-septal space (orbital cavity proper)

See S9 anatomy slide 10

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8
Q

What is periorbital (pre-septal) cellulitis?

A
  • infection occuring WITHIN eyelid tissue, SUPERFICAL to orbital septum
  • secondary to superficial infections (ex. Bites, wounds)
  • may be secondary to bacterial sinusitis in kids
  • confined to tissues superficial to orbital septum (and tarsal plates) so deeper structures are unaffected
  • ocular function is unaffected
  • can be difficult to differentiate between peri-orbital and the more severe orbital cellulitis
  • if any doubt, urgently refer IV and surgical drainage

See S9 anatomy slide 11

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9
Q

What is orbital (post-septal) cellulitis?

A
  • infection WITHIN the orbit (not globe of eye) posterior or deep to the orbital septum
  • will push the eye forward
  • painful eye movements and they may be reduced
  • as pressure increases, optic nerve may be involved which will affect vision (reduced visual acuity)
  • proptosis/exopthalmous
  • orbital veins drain to cavernous sinus, pterygoid venous plexus and facial veins
  • potential route for infection to spread intracranially (ex. Cavernous sinus thrombosis, meningitis)

See S9 anatomy slides 12-13

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10
Q

List the contents of the orbital cavity

A
  • eyeball
  • fat
  • associated extra-ocular muscles
  • nerves and blood vessels
  • lacrimal apparatus
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11
Q

Describe the lacrimal apparatus

A
  • structures involved in tear film production and drainage
  • consists of lacrimal gland, lacrimal ducts and lacrimal canaliculi
  • lacrimal gland secretes tears and is under parasympathetic control via facial nerve
  • tears enter conjunctival sac through lacrimal ducts and pass into the lacrimal lake
  • from here the fluid drains into lacrimal sac before going to nasal cavity via the nasolacrimal duct
  • Blinking (caused by palpebral orbicularis oculi) distributes tear film across front of eye, rinsing and lubricating conjunctivae and cornea

See S9 anatomy slide 15

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12
Q

What is epiphora?

A
  • obstruction to the drainage system in lacrimal apparatus

- overflow of tears over lower eyelid

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13
Q

How is the eye protected from corneal abrasions and ulcerations?

A
  • outer epithelial layer of cornea is constantly undergoing mitosis
  • so easily regenerates if damaged
  • injuries that penetrate deeper into cornea can lead to permanent scarring and possible visual impairment
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14
Q

Describe the anatomy of the eyeball

A
  • globe with 3 layers
  • Outer: sclera (white of eye), tough outer layer that is a continuation of dura around optic nerve and is continuous anteriorly as transparent CORNEA; provides attachment for extra-ocular muscles and gives shape to the eyeball
  • Middle: choroid, ciliary body and iris (vascular)
  • Inner: retina which has an inner photosensitive layer (neurosensory cell layer) lying on an outer pigmented epithelial cell layer

Eyeball is maintained in position by

  • suspensions ligament (sits underneath like a sling)
  • extra-ocular muscles
  • supported by lots of bridal fat

See S9 anatomy slide 16

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15
Q

Describe the two layers of the retina

A

Pigmented epithelial cell layer

  • contains melanin which helps to absorb scattered light passing into the eye
  • reduced reflection and allows us to focus images appropriately on to the retina

Neurosensory layer

  • area of retina that senses light
  • where photoreceptors (rods and cones) are found
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16
Q

What is the conjunctival membrane (aka conjunctivae) and what happens when it is damaged?

A
  • thin transparent layer of cells covering the sclera except the cornea
  • is highly vascular with blood vessels within membrane
  • produced mucous component of tear film

Inflammation

  • known as conjunctivitis
  • highly contagious
  • eye appears red
  • often viral aetiology
  • eye feels uncomfortable and “gritty” with tearing
  • treatment: good hygiene and eye drops

Haemorrhage

  • known as subconjunctival haemorrhage
  • causes “red eye”
  • when one of the vessels ruptures
  • common, not painful
  • slowly resolves, like a bruise

See S9 anatomy slide 17

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17
Q

What is the limbus?

A

-junction between conjunctivae and cornea

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18
Q

Describe how light is used by the eye

A
  • need transparent structures and medium
  • need to refract light to bring to a focal point
  • most of refraction occurs between air and cornea
  • several structures refract light (all transparent): cornea and it’s associated tear film, Lens, aqueous humour and vitreous Humour

Cones

  • located in the macula (slightly darker than rest of retina, lies lateral to optic disc) of the retina
  • fovea: very centre of macula which contains only cones
  • responsible for high visual acuity and colour viscous

Rods

  • active at low light levels, do not mediate colour vision
  • abundant in peripheral parts of retina
  • photoreceptors convert the light energy into electric impulses
  • action potentionasl generated in response to light via retinal ganglion cells (RGC)
  • RGC axons collect in area of optic disc forming the optic nerve
  • optic disc= blind spot since no photoreceptors are present
  • action potentials propagated along visual pathway to occipital lobe for interpretation

See S9 anatomy slide 19, 22-23

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19
Q

How does the shape of your eyeball affect your ability to focus light appropriately onto retina?

A
  • Myopia: short-sighted, eyeball too long so point of focus is too far in front of retina
  • Hypermetriopia: long-sighted, eyeball too short so point fo focus
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20
Q

What is the accommodation reflex?

A
  • focussing NEAR objects requires greater refraction of light
  • with very near objects, light rays are more divergent and greater refraction beyond the capabilities of the cornea is needed
  • so we must increase the refractive power of the eye through the accommodation reflex
  • consists of 3 components: automatic contraction of pupil, convergence of eyes, thickening of lens
  • pupillary constriction ensures light from the near object passes through centre of lens
  • convergence ensures that both retinae focus on the one object
  • contraction of ciliary muscles moves ciliar body closer to the lens and so the pull of the circular suspensory ligament loosens the lens which allows the lens to be more biconvex (rounder)
  • as we age, lens becomes stiffer and less able to change shape
  • known as PRESBYOPIA

See S9 anatomy slide 20-21

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21
Q

Explain what blurring of vision is

A

Pathology affecting

  • transparency of structures anterior to retina (ex. Opacity in lens such as cataract)
  • ability of structures to refract light (ex. Irregularity of corneal surface (astigmatism), ability of lens to change shape (presbyopia), or shape of eyeball
  • and the retina (including macula) or optic nerve (ex. Retinal detachment, age-related macular degeneration, optic neuritis)
  • will cause blurring of vision/decreased acuity

See S9 anatomy slide 24

22
Q

What is decreased visual acuity-refractive error?

A
  • errors of refraction have no effect on light travelling perpendicular to the cornea/lens (i.e. light doesn’t need to be refracted to be brought into focus on the macula)
  • blurring of vision due to a refractive error, acuity improves with pin-hole testing
  • if acuity doesn’t improve with pin-hole then it’s a problem with the retina

See S9 anatomy slide 25

23
Q

What are the 3 chambers of the eye?

A
  • Anterior :aqueous humour
  • Posterior: aqueous humour
  • Vitreous chamber: vitreous humour

See S9 anatomy slide 26

24
Q

Describe the vitreous chamber

A

-filled with transparent jelly-like vitreous humour

25
Q

Describe the anterior chamber

A
  • space between cornea and iris

- communicated with posterior chamber through pupil

26
Q

Describe the posterior chamber

A
  • space between the iris and lens

- ciliary body and processes are found here and secrete all the aqueous humour

27
Q

What is the importance of aqueous humour

A
  • helps support shape of eyeball by the pressure it exerts
  • provides nourishment to lens and cornea since they are avascular
  • drains through irido-corneal angle into the canal of Schliemann via the trabecular mesh work and back into venous circulation

See S9 anatomy slide 27

28
Q

What is glaucoma?

A
  • optic nerve damage secondary to raised intraocular pressure
  • is irreversible damage and death to optic nerve
  • causes impairment of vision or blindness
  • can lead to optic neuropathy
  • open-angle and closed-angle

See S9 anatomy slide 28

29
Q

What is open-angle glaucoma?

A
  • most common
  • caused by blockage WITHIN the trabecular mesh work which drains the aqueous humour
  • develops painlessly and insidiously over time
  • difficult to pick up as it may initially be asymptomatic
  • Diagnosis: look for raised Iop, cupping of optic disc, visual field loss (i.e. Periphery)
  • treatment: eye drops that reduce production of aqueous humour, anything to reduce IOP, or surgery
30
Q

What is close-angled glaucoma?

A
  • if iridologist-corneal angle is narrowed by the peripheral edge of the iris
  • less common
  • access to trabecular mesh work is blocked off leading to rapid rise in IOP
  • pt. Presents with sudden onset of painful red eye, blurred visio or halos around object (due to corneal oedema), fixed or sluggish semi-dilated often irregular oval-shaped pupil, nausea and vomiting
  • eye will feel hard and tender to palpate through upper eyelid
  • emergency because irreversible sight loss can occur within few hours
  • management: diuretics, muscarinic eye drops, strong analgesia as they help to open the angle to improve drainage
  • long-sighted middle aged or elderly people are at most risk

See S9 anatomy slide 29

31
Q

Describe the extrinsic muscles of the eye and orbit

A
  • muscles of the eyelid
  • extra ocular muscles that move the eyeball
  • innervated mainly by somatic motor nerves but has some sympathetic innervation
  • ex. Orbicularis oculi and LPS
32
Q

Describe the intrinsic muscles of the eye and orbit

A
  • muscles of the iris (dilator and constrictor of the pupil)
  • ciliary muscle controls thickness of the less
  • innervated by autonomic (visceral) nerves)
  • ex. Sphincter pupillae and dilator pupillae
33
Q

Which extraocular muscle is the only one that originates from the floor of the orbit?

A

Inferior oblique

34
Q

Where do all the rectus muscles originate from? (Except inferior oblique)

A

-back of orbit

35
Q

What is the purpose of the trochlea in terms of superior oblique muscle?

A

-trochlea is where superior oblique swings back 45 degrees to insert into top of eyeball

See S9 extra ocular slide 3-4

36
Q

Explain the primary resting gaze

A
  • maintained by equal and opposite pull of all extraocular muscles
  • even “at rest”, constancy of activity in all extra ocular muscles on the eyeball
  • each muscle has ANTAGONIST of its movement
  • during resting gaze their actions are balanced allowing for forward gaze
  • visual axis of both eyes are aligned
  • double vision occurs when the fusion doesnt occur
  • 2 images that reach cortex then “fused”, so seen as ONE
  • binocular vision allows for depth perception enabling “3D” vision (that’s why 2 eyes)

S9 extra ocular slide 5

37
Q

What do your extraocular muscles do when you change position of gaze?

A
  • exert greater pull through action of certain extraocular muscles, while antagonists relax
  • muscles moving BOTH eyes must be highly coordinated and move simulataneously
  • visual axes must remain aligned (conjugate gaze)
  • if visual axes are malaligned you get diplopia

See S9 extraocular slide 6-7

LR6SO4

38
Q

Describe the axis that extraocular muscles follow

A

Visual axis (axis of the eyeball): goes right through the middle of the eyeball
Axis of the orbit: axis which extraocular muscles run in line with
-therefore some muscles attach to globe at an OBLIQUE angle
-those attaching to superior and inferior surfaces
-confers some muscles several “actions” of movement on globe (not just simply “up” and “down”)

39
Q

What are the actions of lateral and medial rectus and what are they innervated by?

A
  • lateral rectus: abducts eye and is innervated by abducens (VI)
  • medial rectus: adducts eye and innervated by oculomotor (III)

See S9 extra ocular slide 10

40
Q

What is superior rectus muscle? (OIAN)

A
  • elevates eyeball but has other action since it inserts obliquely
  • arises from apex of orbit
  • inserts into superior anterolateral surface of globe
  • action (if starting from primary resting gaze): elevate, slightly adducts, slightly intorts

See S9 extraocular slide 11

41
Q

What is inferior rectus muscle? (OIAN)

A
  • depresses eyeball but has other actions
  • because inserted obliquely
  • originates from apex of orbit
  • inserts into anteroinferior surface of globe
  • action (if from primary resting gaze): depress, slightly adducts, slightly extorts

S9 extraocular slide 12

42
Q

What is the superior oblique muscle? (OIAN)

A
  • intorts eyeball but also has depressing action
  • arises from apex of orbit, passes through trochlea, inserts into superoposterior aspect of globe
  • action (if from primary resting gaze): intorts, depress, slightly abducts
  • down and out position

See S9 extraocular slide 13

43
Q

What is the inferior oblique muscle? (OIAN)

A
  • extort eyeball but also has elevation action
  • arises from anteromedial surface of floor of orbit; inserts into inferoposterior aspect of globe
  • action (if starting from primary resting gaze): extort, elevate, slightly abduct

See S9 extraocular slide 14-15

44
Q

How are the extraocular muscles affected in abnormalities of gaze?

A
  • constancy of activity in all extra ocular muscles on the eyeball
  • at resting gaze, equal and opposite pull
  • if a muscle is weakened, its “influence” is lost
  • other muscle actions no longer antagonized (balanced out)
  • resting position of eyeball may deviate= strabismus (squint) due to actions of remaining working muscles
  • difficulties with moving eye in certain directions of gaze
  • can be congenital or acquired (ex. Cranial nerve lesions)

See S9 extraocular slide 16-18

45
Q

How would you clinically examine eye movements?

A
  • need to isolate an action of EACH muscle to test them
  • some muscles have >1 action; some “share similar movements”
  • lateral and medial rectus are straight forward since they each only perform one action on the eye
  • BUT elevation and depression (in midline) involved two muscles each
  • do H test with fingers

See S9 extra ocular slide 19-20

46
Q

How can we isolate the elevation and depression actions of the extraocular muscles?

A
  • move starting position of eye
  • if medially adducting first: then test for elevation and depression, only superior and inferior oblique will be working
  • if laterally abducting first: then test for elevation and depression, only superior and inferior rectus working
  • elevation and depression cant be isolated from midline so have to start at a different position

See S9 extra ocular slide 21-24

47
Q

How do cranial nerve palsies occur in the eye?

A
  • abnormalities of eye movements and diplopia
  • may be obvious from initial inspection of the eye
  • and/or become more apparent on testing eye movements
  • CN III, IV, VI innervate muscles that move eyeball
  • can be affected by raised ICP (eg. Intracranial haemorrhage or tumour)
  • BUT ALL can also be affected by VASCULAR disease (microvascular complications) from DIABETES and HYPERTENSION
  • otherwise patients are asymptomatic and lesions will self-resolve within a few months
  • history will help give clearer indication for more concerning underlying cause
  • headache, vomiting could suggest raised ICP
  • recent head injury
  • presence of pupil involvement in CN III lesions

See S9 extra ocular slide 29

48
Q

Describe CN III nerve palsy in the eye

A
  • not all CN III nerve lesions will affect the pupil
  • MOST extra ocular eye muscles are innervated by CN III (except LR and SO)
  • innervated majority of eyelid (LPS) and sphincter pupillae muscle
  • vasculopathic (microvascular) lesions (ex. Diabetes/hypertension)
  • PUPIL is spared
  • compressive lesions (raised ICP, tumour, posterior communicating artery aneurysm)
  • parasympathetics run on periphery of CN III
  • so PUPIL involved

See S9 extraocular slide 26

49
Q

Describe CN IV nerve palsy in the eye

A
  • innervates SO ONLY
  • SO acts to intort, depress and abduct the eye
  • losing these actions so eyeball is held EXTORTED, slightly elevated and slightly adducted
  • compensate for the slight extortion of eyeball by tilting head slightly
  • abnormality in gaze can be very subtle and often missed
  • worsening diplopia (on downward vertical gaze) especially looking down and medially (ex. Walking down stairs, reading(
  • SO is main DEPRESSOR of eyeball when in ADDUCTION

See S9 extraocular slide 27

50
Q

Describe CN VI nerve palsy in the eye

A
  • innervated LR
  • unopposed pull of MR
  • unable to ABDUCT the eye on affected side
  • report diplopia, made worse on HORIZONTAL gaze

See S9 extra ocular slide 28-32