8: Physical and Chemical Injuries Flashcards
Common alteration of the buccal mucosa most often associated with pressure, frictional irritation, or sucking trauma from the facial surface of teeth, 13% of population?
linea alba
What is the clinical presentation of linea alba?
white line, bilateral at level of occlusal plane with no other associated problems
Linea alba treatment
None, no biopsy
What causes linea alba
hyperkeratosis
Lesion from chronic, habitual nibbling with a higher incidence in females presenting with thickend, shredded, white areas? Lip chewing? Tongue Chewing?
Morsicatio Bucarum, Morsicatio Labiorum, Morsicatio Linguarum
How would differentiat Morsicatio Buccarum from Luekoplakia from Lichen planus?
Morsicatio bucarum has ragged torn look Leukoplakia has homogenous look Lichen Planus has Striae of Wickam
How could you differentiate Morsicatio Linguarum from Oral Hairy Leukoplakia?
biopsy does not show EBV, candidiasis and no history of HIV
5 ways to get Ulcerations?
Physical, thermal, Electrical, TUGSE, Riga-Fede disease
What is TUGSE?
Traumatic Ulcerative Granuloma with Stromal Eosinophilia
With what is TUGSE associated?
Eosinophils = allergies or parasites
What is the only clinical differentiation between a TUGSE ulcer and an aphthous ulcer?
TUGSE is slower to heal (>7 days)
These are traumatic ulcerations of the ventral tongue in nursing babies and are a variation of TUGSE
Riga-Fede Disease
Represent 5% of all burn admissions
Electrical burns
2 types of electrical burns and which is most common oral
Contact and Arc. Arc is most common orally with saliva acting as conductor
Age and most common location for electric burns
<4 years old. Lips and commissures presenting charred yellow with little bleeding
3 stages of treatment of ulcers (in order)
1.) Remove obvious injury
2.) Treat symptoms: cellulose films, topical
antibiotics, Orabase)
3.) If not healed in 2 weeks, Biopsy
Are natal teeth associated with Riga-Fede disease extra teeth or are they the babies deciduous teeth simply erupted early
early erupted deciduous teeth.
4 sources of Chemical injuries
Aspirin, OTC med, Gasoline, Iatrogenic dental chemicals (formocreosol, etch, Hydrogen peroxide, Silver nitrate)
Why are OTC meds a source of chemical injuries
Have a lot of eugenol and phenol
How can one get a cotton roll burn (3)?
Cotton roll wicks out moisture and causes necrosis, holds caustic chemicals against the epithelium, or is removed and takes epithelium w/ it
All systemic anticancer therapies (antineoplastic therapies) cause what?
death of some normal cells
What are the 2 acute oral changes associated w/ cancer chemotherapy?
Mucocitis and hemorrhage
Where does oral mucocitis associated with cancer chemotherapy present?
Nonkeratinized surfaces (Buccal mucosa, ventrolateral tongue, soft palate, floor of mouth)
How does Oral Mucocitis associated with cancer chemotherapy and radiation treatment look clinically?
early develops white discoloration from lack of keratin desquamation followed by loss of that layer & replacement by atrophic mucosa that is edematous, erythmatous & friable. Finally is ulcerated and covered by yellowish fibrinopurulent membrane. Recap: white, red, yellow
Where does Radition therapy acute mucocitis present?
mucosal surface w/in direct portals of radiation
What are the 2 acute changes associated with cancer radiation therapy?
Acute mucocitis and dermatitis
What is the character of Radiation Therapy Dermatitis based on?
Varies w/ amount received. Can be erythema, edema, burning, purities all the way to necrosis and deep ulcerations.
What are the 5 Sequelae (a pathological condition resulting from disease [Wiki definition]) to Cancer Chemotherapy?
Increased susceptibility to infection, oral mucocitis, oral ulceration, increased risk of hemorrhage, or impaired healing
The introral hemorrhage that is a sequelae of cancer chemotherapy is secondary to what
thrombocytopenia caused by bone marrow suppression
This is a complication associated with head and neck radiation
Xerostomia
What is the course of radiation induced Xerostomia?
Changes after 1 week. Decrease in saliva after 6 weeks. Parotids dramatically/irreversibly affected. Decreased bactericidal activity of saliva cause increased caries
8 Sequelae of Radiation therapy
Hemorrhage, Mucocitis, Dermatitis, Xerostomia, Osteoradionecrosis, Loss of taste/ altered taste (Hypgeusia/dysguesia), Trismus, Developmental anomalies (e.g. microdonts, hypoglossia) which are dependent on age and treatment
What can cause the osteoradionecrosis
hypoxia, hypovascularity, hypocellularity
What increases the risk of developing osteoradionecrosis and what are the treatment limits?
Surgery. Tx pt either 21 days prior to radiation (to allow healing prior to therapy) or wait 1 year after radiation therapy is complete
What can dentist do pretherapy for cancer patient
remove current or potential oral foci of infection. Give OHI