7. Vision Flashcards
Age related changes to vision
- corneal yellowing and increased opacity
- changes in corneal curve
- increased lens flexibility and density, opaque
- decrease pupil size
- sclerosis and rigidity of iris
- atrophy of ciliary muscle
- shrinkage of gelatinous substance in the vitreous
- atrophy of photoreceptor cells
- thinning and sclerosis of retinal blood vessels
- degeneration of neurons in the visual cortex
Functional consequences of impaired vision
- need for 3-5 times more light than before
- effect on driving
- increased risk for unsafe mobility and falls
- increased difficulty performing ADLs
- anxiety, depression, lower levels of psychological well-being
copy paste of age related changes
Cornea- yellowing and increased opacity, changes in curvature
Arcus senilis- grey around iris
lens- increase in size and density
Iris- sclerosis and rigidity of the iris
Pupil- decrease in size = less light entering retina
Ciliary muscle- stiffening, atrophy of muscle
Vitreous body – shrinkage of gelatinous substance in the vitreous
Photoreceptor cells- (usually converts light into electrical signals to nerve cells) - atrophy (die)
retinal blood vessel – thinning and sclerosis of vessel
optic nerve- slower processing of visual information
Visual cortex- degeneration of neurons in visual cortex p. 330
Consequences of Age-Related Changes on Vision
- diminished acuity
- slower response to changes in light
- increased sensitivity to glare
- narrowing visual field
- lower depth perception
- altered colour perception
- distorted perception of flashing lights
- diminished processing of visual information
- dry eyes
4 Pathologic conditions affecting vision
- cataract
- leading, reversible cause of vision impairment - age-related macular degeneration
- leading cause of severe vision loss - glaucoma
- causes loss of peripheral vision leading to blindness if untreated - diabetic retinopathy
Cataract
- Lens becomes cloudy= light scatters= blurry vision
- Leading cause of avoidable blindness
- Caused by age-related changes or congenital - due to an illness, likerubella, that your mother had duringpregnancy.
- Risk factors include: exposure to sunlight, smoking, malnutrition, trauma, adverse med reactions, diabetes
Assessment: Symptoms include C: cloudy blurry A: Acquiring more light T: toned down colour H: Halos around objects Y: yellow tint to most things
Interventions:
- surgical removal of lens and installation of artificial lens
- teaching about benefits of surgery for people with information processing difficulties (dementia, depression)
- smoking cessation
- use sunglasses
Age-related Macular Degeneration (AMD)
2 types: wet, dry
2 types of central vision loss, peripheral vision stays intact
- wet - more severe, ends in blindness
- dry - less severe, progresses slowly
-no cure
-treatment is limited because of the slow progression
-focus on screening, early detection, prevention
Assessment:
-test eyes daily with Amsler grid to catch sudden changes to central visual field
-face test- cover one eye and look in mirror to notice any central blurring or missing parts
Interventions:
- no cure
- no treatment
- focus on health promotion:
- diet high in antioxidants like dark green veg
- eat fish for omega 3
- improve cardio health
- smoking cessation
- reduce HTN
- control weight
- avoid processed foods
Glaucoma
3 types
Eye disease with elevated intraocular pressure in which damage to the eye optic nevre can lead to loss of vision and blindness
- Chronic (open-angle)
- most common
- IOP
- cause: clogged trabecule
- early signs: poor vision in dim light, increased sensitivity to glare
- progression: headaches, tired eyes, poor peripheral vision, halos around lights
- annual eye exams to detect
- treated with meds or laser
- painless - Normal Pressure
- optic nerve is damaged yet NO IOP
- unknown cause
- blind spots due to damaged nerve fibers
- permanent blindness unless treated with drops or surgery - Acute (closed-angle)
- fluid cannot go anywhere so goes back to eyes and builds up and pushes on retina
- IOP increases quickly as drainage is blocked
- less common
- MEDICAL EMERGENCY
- symptoms: pain, blurred vision, headache, pupil dilation, high IOP, nausea, vomiting
- blindness unless treated with meds or surgery
Assessment:
- past medical hx
- age 65+ annual eye exam for screening
- pts with medication controlled glaucoma should have check-up q6months
- annual screening for Asian, African, Hispanic, and family hx of glaucoma who are age 40+
Interventions:
- medications to decrease IOP
- laser surgery
- use of colour contrast to identify objects, glare control
- semi or annual eye exams
Diabetic Retinopathy
- effects of elevated blood sugar caused by diabetes
- disease of microvasculature
- increased vessel permeability= blood and lipid leakage leads to macular edema and hard exudates
- Hard exudates(lipid)Hard exudatesare small white or yellowish-white deposits with sharp margins. Often, they appear waxy, shiny, or glistening. They are located in the outer layers of the retina, deep to the retinal vessels.
Assessment:
- early signs are microaneurysms, flame-shaped hemorrhages, hard exudates
- annual dilated funduscopic exam of the eye beginning 5 years after diagnosis of type 1 diabetes and at the time of type 2 diagnosis
Interventions:
- constant control of blood glucose, cholesterol, and BP
- laser photocoagulation treatments to halt disease progression (Photocoagulation = a laser is used to finely cauterize ocular blood vessels to attempt to bring about various therapeutic benefits.)
General Interventions:
- sunglasses
- regualr eye exams
- proper lighting
- good nutrition
- smoking cessation
- med compliance