4.1 Communicable diseases Flashcards

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1
Q

tuberculosis

A

bacteria- myobacterium

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2
Q

bacterial meningitis

A

bacteria

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3
Q

ring rot- plants

A

bacterium

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4
Q

HIV/AIDS

A

virus

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5
Q

influenza

A

virus

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6
Q

tobacco mosaic virus- plants

A

virus

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7
Q

black sigatoka- plants

A

fungus

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8
Q

blight- plants

A

protoctistan

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9
Q

ringworm

A

fungus

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10
Q

athlete’s foot

A

fungus

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11
Q

malaria

A

protoctistan

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12
Q

what is malaria caused by?

A

a person with malaria has gametes of plasmodium in blood
female anopheles mosquito sucks blood
plasmodium develops goes to salivary glands
bites an uninfected person
goes to liver, then blood (repeats)

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13
Q

physical plant defenses against pathogens

A
cellulose cell wall
lignin thickening
waxy cuticles
bark
stomatal closure
callose
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14
Q

primary defenses against pathogens in animals

A
the skin
blood clotting and skin repair
mucous membranes
inflammation
antibodies in tear fluid
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15
Q

How does antibody structure help its function?(7)

A

2 light chains and 2 heavy chains / 4 polypeptide
chains ;
variable region allows , binding / attachment , to
antigen ;
two variable regions allow binding of more than
one (of the same) antigen ;
variable region on different antibodies allows specificity
to different antigens ;
constant region allows , recognition by / attachment to / binding to , (named) phagocytes ;
hinge (region) allows flexibility ;
disulfide , bonds / bridges , hold , polypeptides / light
and heavy chains , together ;

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16
Q

What is neutralisation?

A

cover / block , binding site / antigen / receptor site (on
pathogen) ;
bind to toxins ;
prevent , binding / entry , to (host) cell ;

17
Q

What is agglutination? (2)

A

clump / bind together , (many) pathogens ;
(clump) too large to , enter (host) cell / cross
membranes ;
increase likelihood of being consumed by (named)
phagocyte

18
Q

state four means of transmission.

A

direct physical contact
faecal-oral transmission
droplet infectoin
spores

19
Q

what are opsonins?

A

proteins that bind to antigen on pathogen and allow phagocytes to bind

20
Q

how do phagocytes engulf pathogens? 5

A

recognises pathogens on antigen
cytoplasm extends around pathogen engulfing it
pathogen now contained in phagosome
a lysosome containing diestive enzymes fuses with phagosome
phaocyte presents pathogens antigens on its

21
Q

what are the 4 types of lymphocytes, and their functions?

A

killer- attack and kill host body cells displaying foreign antigens
helper - release cytokines stimulating B cels to develop and stimulate phagocytosis
regulator- shut down immune response
memory- long term immunnity

22
Q

what are secondary non specific defenses in the body? ((3)

A

opsonins (bind to one antigen on the pathoen and allow phagocytes to bind)
neutrophils
macrophages

23
Q

what are the 5 steps of the specific immune response?

A
infection of pathogen
presentation of antigens
clonal selection
clonal expansion
differentiation
24
Q

What is herd vaccination?

A

vaccinating almost all of population (80-85%)

25
Q

What is rin vaccination?

A

vaccinating people in immediate vicinity

26
Q

What is the difference between active and passive immunity?

A
active= immune system is activated and produces own antibodies
passive= when antibodies are passed on to individual through breast feeding or innjection
27
Q

what is the difference between natural and artificial immunity?

A
natural= immunity achieved through normal life processes
artificial= immunity achiived as result of medical intervention
28
Q

What is the role of callose?

A

large polysaccharide that blocks sieve tubes

29
Q

describe the steps of phagocytosis. (5)

A
pathogen recognised
pathogen enulfed
trapped in phagosome
lysosomes fuse with phagosome
digestion
30
Q

What is the difference in role of B and T lymphocytes?

A

B lymphocytes manufacture antibodies

T lymphocytes release cytokines

31
Q

What is meant by the term “vaccination”? (2)

A

artificial introduction of antigenic material

which stimulates the immune response

32
Q

distinguish between the primary defenses and secondary defenses in mammels. (2)

A

primary defences prevent pathogen from entering the body

secondary defences attack the pathogen once its in the body

33
Q

mast cells release histamine. What does histamine do?

A

cause vasodilation

capillaries more permeable to white blood cells