(3) Lecture 18: Sport Related Concussions Flashcards

1
Q

Sport Related Concussion

A

a traumatic brain injury induced by biomechanical forces

may be linear or rotational

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2
Q

Linear injury

A
  1. Injury at impact site
  2. brain strikes skull on OPPOSITE side (contre-coup)
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3
Q

Rotational injury

A
  1. Brain strikes skull on other side
  2. Brain rotates - stretches or tears structures/vessels as it shears on itself

CSF doesn’t help w/ rotational/shear injuries

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4
Q

Common symptoms

A
  • headaches, fatigue, and dizziness are common in youth
  • difficulty concentrating
  • “fogginess”
  • dizziness
  • light sensitivity
  • visual blurring or double vision

NONE of these are always present

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5
Q

Clinical Domains of SRC

A
  1. Symptoms → Somatic (headache), Cognitive (fog) & Emotional (mood swings)
  2. Physical signs → LOC, Amnesia, Neurological deficits
  3. Balance impairment → gait unsteadiness
  4. Behavioural changes → irritability
  5. Cognitive impairment → slowed reaction times
  6. Sleep/Wake disturbances → somnolence (feeling really tired)
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6
Q

When should a SRC be suspected?

A

If symptoms or signs in any ONE or more of the clinical domains are present

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7
Q

“I didn’t see him hit his head”

A

SRC can be caused either by
- direct blow to head, face, neck, OR
- blow elsewhere on body w/ an impulsive force TRANSMITTED to head (fwd-back movement)

Therefore, they don’t have to get hit in the head

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8
Q

“He didn’t even get hit hard”

A
  • NO agreed-on biomechanical threshold for injury
  • force does NOT factor in on symptom severity

Therefore, they don’t have to be hit hard to be concussed

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9
Q

“Needs better equipment”

A
  • helmets were made to prevent catastrophic head injuries like skull fractures, not low impact/rotational forces
  • evidence for mouthguard preventing SRC is mixed

Therefore, better equipment does not necessarily prevent SRC

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10
Q

“Concussions are getting worse”

A
  • No such thing as a minor, severe, Grade 1,2 or 3
  • time missed is getting longer b/c we are getting smarter with how to deal with them (not b/c SRCs ae getting worse but we know to sit out longer)

Therefore, concussions are not getting worse, we’re getting smarter

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11
Q

Acute medical management of SRCs

A
  • disposition of player must be determined in a timely manner
  • standard orientation Q’s (time, place, person) are unreliable when used in isolation
  • diagnosis should combine subjective symptom reports + clinical exam
  • a combo of sideline tests should be used (NOT in isolation) - combo of symptom eval, postural control on firm surface, neurocognitive screening = best
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12
Q

Sideline tests

A
  • modified balance error scoring system (mBESS)
  • vestibular/ocular motor screening (VOMS)
  • neurocognitive testing (SAC)
  • post-concussion symptom scale (PCSS)

all part of SCAT-5 EXCEPT VOMS

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13
Q

Test for postural stability

A

Modified balance error scoring system (mBESS)

part of SCAT-5

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14
Q

Test for oculomotor function

A

Vestibular/ocular motor screening (VOMS)

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15
Q

Test for neurocognitive functions

A

Standard assessment of concussion (SAC)

part of SCAT-5

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16
Q

Test for SRC symptoms

A

Post-concussion symptom scale (PCSS)

part of SCAT-5

17
Q

SCAT 5

A

most well-established and developed instrument for sideline SRC assessment

  • use the SCRIPT - be consistent
  • SAC immediate and delayed word recall lists includes option to use 10 words to minimize ceiling effect
  • rapid neuro screening
  • SCAT5 utility appears to decrease significantly 3-5 days after injury
18
Q

Evaluation of SRC

A
  • remember to RE-EVALUATE
  • serial monitoring for deterioration is essential over initial few hours after injury

Red Flags = immediate hospital trip
- Person complains of neck pain
- Deteriorating conscious state
- Increasing confusion or irritability
- Severe or increasing headache
- Repeated vomiting
- Unusual behaviour change
- Seizure or convulsion
- Double vision
- Weakness or tingling/burning in arms or legs

19
Q

Return to Play

A

expected duration of symptoms in children is up to 4 weeks

20
Q

Children under 13 Return to Play

A
  • evidence to suggest that the developing brain is MORE vulnerable to injury
  • NEVER RETURN TO PLAY ON SAME DAY
  • should not return until clinically completely symptom-free

WHEN IN DOUBT, SIT THEM OUT

21
Q

Neurometabolic Changes and Concussion

A
  • at day of injury, glucose need increases and blood flow decreases = mismatch
  • 1st 2 weeks – brain does NOT operate like it should = doesn’t get enough blood flow or glucose
22
Q

Second Impact Syndrome

A
  • occurs up to 14 days post-injury
  • occurs in athletes w/ prior concussion, who return to play before resolution of symptoms
  • catastrophic increase in INTRACRANIAL PRESSURE following relatively minor second impact
  • most often occurs in athletes < 21 years old
23
Q

Safe and Effective RTP People

A

Athlete
- everything is based on their subjective reports (must be honest and forthcoming)

Parent: often liaison btwn medical team and sport team

Medical: voice of reason (critical eye - communicate RTP stages)

Coach: plans practice (team culture)

MUST work together to idealize environment

24
Q

Return to Play Stages

A
  1. Symptom-limited activity
    - goal: gradual reintroduction of work/school
  2. Light aerobic exercises: walking, stat. bike at slow pace
    - NO resistance training
    - goal: increase HR
  3. Sport-specific exercise: no head impact activities
    - goal: add movement
  4. Non-contact training drill: harder training drills
    - may start progressive resistance training
    - goal: exercise, coordination + thinking
  5. Full contact practice: participate in normal training
    - goal: restore confidence + assess functional skills by coaching staff
  6. Return to sport: normal game play

player MUST HAVE 24 hrs SYMPTOM FREE BTWN STAGES
- if symptoms return, they go back to previous stage after 24hrs symptom free