3- Assessment & Research Flashcards

1
Q

Psychological Assessment and Reliability/Validity

A

Systematic gathering and evaluation of info related to sme’s psychological sx and problems => Integrate knowledge of assessment into broader context

Purposes:

  • Describe current functioning
  • Dx
  • Identify therapeutic needs
  • Monitor tx over time

Reliability:

  • Test retest (be mindful of what you measure ex: contract construct)
  • Inter-rater

Validity:

  • Face validity
  • Content validity
  • Construct validity

*Without reliability, validity is irrelevant

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2
Q

Approaches to Assessment

A
  • Clinical: uses clinician’s exp and personal judment
  • Actuarial: use of objective, empirical data (mostly in research)
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3
Q

Potential Biases in Assessment (3)

A

Theoretical orientation of the clinician

  • Shapes types of assessment tools used
  • Shapes interpretations of same beh
  • Shapes treatment recommendations

Underemphasis on broader socialcultural context

  • DSM places disorder within the individual
  • What about the person’s environment?

Lack of cultural sensitivity

  • Imposing Western perspectives on other cultures
  • Beh mean different things in different cultures ex: eye contact (in Japan), belief in evil spirits
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4
Q

Clinical Interviews and Content

A
  • Unstructured
  • Semi-Structured => can skip a few sections if no sx ex: SCID, MSE (examination used in hospitals, etc)
  • Structured ex: DIS

=> Diagnostic Interview Content

  • Identifying features
  • Reason for referral
  • History of Present Illness (HPI)*
  • Psychiatric history
  • Medical history
  • Habits (Substance use)
  • Family history
  • Social and developmental history
  • Review of symptoms
  • Mental status exam
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5
Q

Self-Report Rating Scales

A

Indicate presence/absence of trait/beh AND severity

ex: Beck Depression Inventory (BDI), Beck Anxiety Inventory (BAI)

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6
Q

Psychological Tests

A

Two main kinds used in clinical psychology:

  • Intelligence Tests (ex: WISC/WAIS)
  • Personality Tests: Projective or Objective
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7
Q

Projective Personality Tests (2)

A

Rooted in psychoanalytic principles => Person presented with ambiguous stimulus will project their unconscious motives, needs, drives, feelings, and defenses

  • Rorschach Inkblot Test: 10 inkblots, lacking in reliability/validity, efforts to improve psychometrics (ex: performance assessment system) *got posted on wiki
  • Thematic Apperception Test: project psychological needs onto characters, pro: might be able to tap into less socially desirable deb vs con: psychometrics

*Generally lack reliability/validity but continued use among clinicians who accept underlying psychodynamic theory

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8
Q

Objective Personality Tests

A

Standardized procedures => Established norms and Statistically validated methods of interpretation ex: MMPI, MCMI

=> MMPI:

  • 567 questions (true/false/cannot say)
  • Contrasted gr method to ascertain validity (compare item resp in disordered gr vs control gr)

Has clinical scales (ex: hypochondriasis, hysteria, paranoia, etc), Validity scales (Lie and Infrequency Scales), DON’T PROVIDE A DX BUT HELPUL TO GUIDE ONE

=> MCMI: 195 questions (true/false), validated on clinical samples, Complementary to MMPI, but more focused on personality dx (aligned with DSM-IV axis II dx) with 15 personality scales (ex: histrionic, schizoid, dependent) and 10 clinical syndrome scales (ex: dysthymia, anxiety, somatoform, bipolar)

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9
Q

Biological Assessment

A

=> Brain Imaging:

Electroencephalogram (EEG)
- Electrodes on scalp measure brain’s electrical activity

Computed Tomography (CT)
- Produces detailed cross-section of brain

Magnetic Resonance Imaging (MRI)
- Structural: brain volume
- Functional: connectivity*

Positron Emission Tomography (PET)
- Requires injection of radioactive tracer

=> Neuropsychological Testing:

  • Cognitive/Executive/Visual/Motor functioning
  • Attention/concentration/Memory
  • Motivation
  • Language/Somatosensory/olfaction
  • Neurodevelopmental dx ex: ADHD, Intellectual Disability, Learning Disability
  • Neurocognitive dx ex: due to Alzheimer’s or TBI

*Can rule out organic impairment in other disorders (ex: depression, anxiety)

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10
Q

Purpose of Clinical Research

A
  • Describe clinical presentations
  • Understand epidemiology
  • Identify etiology
  • Prediction of beh
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11
Q

Epidemiology

A
  • Prevalence: % of people in a population with a dx at a particular point in time (ex: past month, year, lifetime)
  • Incidence: % of people who develop a disorder for the 1st time during a specific period

Risk Factors:

  • For epidemiologists, this is a correlate (most often demographic variables) associated with different dx
  • Psychologists use this term to mean a predictor, or cause.
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12
Q

Case Studies

A

Case studies: Detailed account of single patient ex: Anna O case study by Freud

Problems:
- Biased reporting
- Generalizability

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13
Q

Experimental Methods

A

Experimental vs. control group => Manipulate IV and measure effect on DV *Key design used to assess tx outcomes

Things to consider:

  • Random assignment
  • Pre-test vs. Post-test
  • Placebo
  • Blinding
  • Statistical significance: means it is extremely unlikely that the obtained results could have occurred by chance (p<.05)
  • Clinical significance: refers to the treatment’s practical utility => Consider effect size, Cost of tx, Particular outcome

Pros and Cons of Experiments

  • Allows us to determine causality (useful for determining if tx is beneficial)
  • Potential ethical concerns: Withholding effective tx and Etiological questions that can’t be addressed
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14
Q

Correl/Cross-Sectional/Longitudinal Designs

A

Correlational: Psychopathology aims to establish causality BUT, core problem with research is use of correlational designs (Due to the nature of our etiological questions)

*Correlation does not equal causality!

Instead, try to:

  • Rule out third variables
  • Establish temporal precedence

Cross-Sectional: Many in clinical psychology, Makes it hard to establish temporal precedence

=> Often “control for” third variables:
- Control groups (often “healthy controls”)
- Include control variables in your analyses

Longitudinal: Can establish some temporal precedence by looking across time

Different types:
- Retrospective
- Follow-up
- High Risk

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15
Q

Longitudinal Studies (3)

A

Retrospective: Collect a sample of people with a dx => Try to determine what preceded it through:

  • Self-report
  • Existing archival data

Follow-Up: Typically studies follow only people with the disorder over time (Due to high resources required)

  • See what happens to an “already-ill-sample”
  • Downside: makes it difficult to derive etiological explanations

High Risk: Variant of follow-up => Identify people who are likely to develop a dx (Follow them over time)

  • Offspring of people with a dx (genetic) => On the basis of a biological abnormality, Beh variable

Cons:

  • Genetic: Need to find people who have the dx and also have children (hard to recruit and underrep)
  • Biological: Associations not well-proven
  • Behaviors: May be a risk factor, or may be early manifestation of the disease
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16
Q

Sampling Issues: Patients vs Community

A

Patient populations are not representation of people with the dx in the community:

  • Clinical populations tend to be more severe, have more comorbidities, more likely to be female, chronic

=> General population, get a sense of the disorder “in the wild”

Control groups: Healthy controls, Psychiatric controls, Match on potential confounds

17
Q

Genetic Epidemiology, Gene Environment Correlations (rGE) and Epigenetics

A

Genetic Epidemio: Nature vs. Nurture question => born with the dx or do they develop it through environmental influences *Genetic = inherited traits

Epigenetics:

  • Environment influences genes! => DNA is fixed, but regulated (Regulation and expression of genes)
  • Some genes ”turn on” at certain developmental periods
  • Under certain environmental circumstances

rGE:

  • Passive => Biological parents provide both genes and env
  • Active => People with certain genes select certain environments ex: “Niche picking”
  • Evocative (reactive) =>People with certain genes will engage in beh that elicit certain responses from others

*Caution in interpretation genetic contributions!

Mode of Transmission:

  • Single-gene transmission
  • Polygenic transmission*
  • Mixed transmission
18
Q

Family/Adoption/Twins Studies

A

Family: First, does disorder run in families?

  • Identify proband
  • Look for concordance
  • Assess family members: Informant Report (Family History Study) & Interview (Family Study)

=> Many dx do run in families

  • Subthreshold/symptoms
  • Coaggregation
  • If concordance rates increase with genetic similarity, this suggests genetic role BUT does not prove it *Shared environment is confounded

Adoption: Parent as a proband or Adoptee as a proband (to see if genes or env)

Issues:

  • Adoption rare event (small samples)
  • Selective placement (matching?)

Twin Studies: Monozygotic (Mz) twins (Share 100% genes aka identical) vs Dizogotic twins (Dz) (Share 50% genes)

=> Greater concordance among Mz (vs Dz) suggests genetic basis

Issues:

  • Rare, small samples (~1% of population = twins)
  • Role of environment => Mz environment is + similar vs Dz, Dz twins show higher concordance rates than non-twin siblings, Same-sex Dz twins show higher concordance rates than opposite-sex Dz twins
  • Not deterministic
  • Outcome often determined by G x E interaction => Notion that adverse effects of genes on mental health only expressed under certain environmental conditions