2.9 Neurological, Sensory-Motor Flashcards

1
Q

Describe the following reflexes and their significance
REFLEXS CORNEAL
CN V & VII

A

When the cornea is touched there should be a brainstem reflex leading to blinking or bilateral eyes and tearing

Absence means something is affecting the nerves such as a tumor, brain stem CVA or brain death

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2
Q

Describe the following reflexes and their significance

OCULOCEPHALIC “DOLL’S EYES”

A

Positive reflex: when the head is turned the eyes should turn with opposite

Negative reflex: when the head is turned the eyes become fixed straight ahead

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3
Q

Describe the following reflexes and their significance

OCULOVESTIBULAR “COLD CALORICS”

A

When ice cold water is used to irrigate the EAR. The brainstem reflex should trigger the eyes to look toward the direction of the irrigated ear

Absence of the reflex indicate brain death

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4
Q

Describe the following reflexes and their significance
GAG
CN IX & X

A

There should be contraction of posterior pharynx muscles when stimulated

Absence can mean over sedation or brain death

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5
Q

Describe the following reflexes and their significance

BABINSKI

A

Planter response to movement of a hard object up the bottom of the food. Normal response is curling of all toes inward (negative Babinski)

Dorsiflexion or fanning of the toes indicates presence of Babinski (abnormal) and can indicate spinal cord or brain injury. (positive Babinski)

This is a primitive response in infants and can also occur with ETOH intoxication or seizure activity

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6
Q

Neuro assessment abnormal findings and their significance

RESPIRATORY CONSIDERATIONS

A

Damage to pons/medulla leads to inability to regulate breathing patterns based on oxygen and carbon dioxide. As a result, you have irregular patterns of breathing depending on the area of injury

Excessive yawning or sighing: first sign of deterioration
Cheyne-stokes: altered deep and rapid breathing with periods of apnea
Hyperventilation: 40 breaths/min or higher
Apneustic: long inhalation and long exhalation
Ataxic/Apneic: uncoordinated or irregular breathing, pt no longer responding to carbon dioxide

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7
Q

What is the Glasgow Coma Scale

A

Measure a person’s level of consciousness after a brain injury. The GCS assesses a person based on their ability to perform eye movements, speak, and move their body
3 PARTS

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8
Q

Components of the Glasgow Coma Scale

EYE OPENING

A
Eye Opening:
Spontaneously 4
To sound 3
To pain/pressure 2
No response 1
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9
Q

Components of the Glasgow Coma Scale

VERBAL RESPONSES

A
Verbal responses:
Oriented 5
Confused 4
Single words 3
Incomprehensible sounds 2
No response 1
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10
Q

Components of the Glasgow Coma Scale

BEST MOTOR RESPONSE

A
Best motor response:
Obeys commands 6
Localizes pain 5
Flexion-withdrawal 4
Abnormal flexion (decorticate) 3
Abnormal extension (decerebrate) 2
No response (flaccid) 1
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11
Q

What does a GCS of 3 mean?

A

Could mean patient is dead
OR
Severe neurological injury

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12
Q

Describe the following head injury

BLUNT

A

No break in skin:
Acceleration (hit in head with bat)
Deceleration (fall)
Acceleration/decelerations (combination of two), Rotation, usually not good outcome, (brain rotating within skull, MVA, shaking baby)

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13
Q

Describe the following head injury

PENETRATING

A

Break in skin: gunshot, knives, metal object, rocks/objects of nature.
NI:
Never pull out object, secure and held in place
Control bleeding
High infection risk

Domestic: CPS, APS

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14
Q

Describe the following head injury

COUP-CONTRECOOUP

A

Acceleration and Deceleration head injury

ie: MVA with the head being forced forward and then back with anterior and posterior injury. Think “whiplash”

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15
Q

Pathophysiology and assessment finding of the following hematomas
EPIDURAL

A

Focal (localized) injury
Hematoma/Hemorrhage

Epidural:
Blood in space between the skull and dura mater, typically from an arterial bleed. Common with skull fractures

Findings:
Loss of consciousness then regain consciousness
ICP
Emergent surgical

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16
Q

Pathophysiology and assessment finding of the following hematomas
SUBDURAL

A

Focal (localized) injury
Hematoma/Hemorrhage

Subdural:
Blood in space between dura and arachnoid typically from a venous bleeding

Findings:
Acute: s/s during time of injury or few days later
Pupil changes
HA
Resp changes
Chronic: s/s can be weeks to months
Fall is common
Mimic dementia
Slow changes to complete loss of consciousness
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17
Q

Pathophysiology and assessment finding of the following hematomas
INTRACEREBRAL

A

Focal (localized) injury
Hematoma/Hemorrhage

Intracerebral:
Blood deep in brain tissue, most common with contusions, penetrating trauma, depressed skull fractures

Findings:
HA
Vomiting
Seizures
Reduced level of consciousness
Hemiparesis
Surgical need
Drain placed
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18
Q

Identify the complication of a traumatic brain injury

HERNIATION

A

Herniation Syndrome:
Shifting of the brain tissue into another part of the brain due to blood or edema.
Shifting shears/tears the vasculature causing decreased brain perfusion and ultimately brainstem compression and herniation

Supratentorial (3 types):
Cingulate: highest up in brain, increased ICP
Central: compression of midbrain, rapid decline of level of consciousness or coma
Uncal/lateral: pressing brain centrally, CNIII trapped, pupil dilation, decreased level of consciousness, respiratory arrest

Infratentorial:
Upward or downward displacement through foramen magnum altering vital functioning. May have altered respirations, fixed pupils, posturing, eventually resp or cardiac arrest

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19
Q

Identify the complication of a traumatic brain injury

BRAIN DEATH

A
Non reversable loss of brain function
Must maintain normothermia
Absent of all reflexes
No depressant drug/ETOH poisoning
Institutional policy may include EEG, brain flow study, apnea test
Notify Gift of Life
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20
Q

Identify the complication of a traumatic brain injury

SIADH

A

Posterior pituitary can alter release of ADH

SIADH: syndrome of inappropriate antidiuretic hormone)
TOO much ADH causing retention of water
Urine: low volume, very concentrated, high specific gravity
Serum: low serum osmolality

Treatment:
Restrict fluid
Slowly replace Na+

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21
Q

Identify the complication of a traumatic brain injury

DIABETES INSIPIDUS

A

Posterior pituitary can alter release of ADH

Diabetes Insipidus:
Insufficient ADH causing diuresis
Urine: increased urine output, low specific gravity
Serum: increased serum osmolality

Treatment:
Fluid replacement
Reduce Na+
Replace ADH with vasopressin

22
Q

Identify the complication of a traumatic brain injury

HYPERTHERMIA

A

Impaired function of the hypothalamus altered temperature regulations (as long as infection has been ruled out). Increased temperature means increased cerebral metabolic rate and increased ICP. Antipyretics will not help. Use of ice packs and cooling blankets.

23
Q

Identify conditions that predispose a patient to increased intracranial pressure (IICP)

A
Hydrocephalus
Bleeding in the brain
Swelling of the brain
Aneurysms
Brain/head injury
High blood pressure
Stroke
Infections: meningitis, encephalitis
24
Q

Identify the signs and symptoms of IICP

A

Early s/s:
Decreased level of consciousness
HA
N/V

Late s/s:
Pupil changes
Posturing
Cushing’s Triad

25
Q

Explain why a sudden rise in IICP is such a catastrophic situation

A

.

26
Q

Describe type of intracranial monitoring

EPIDURAL PROBE

A

Probe is placed between skull and dura mater
No drain
Can monitor ICP numbers

27
Q

Describe type of intracranial monitoring

SUBARACHNOID SCREW

A

Fiber optic catheter placement allows the sensor to record from inside the subdural space
No drain
Can monitor ICP numbers

28
Q

Describe type of intracranial monitoring

INTRAVENTRICULAR CATHETER

A

IVC is used to connect the ventricles of the brain to an external ventricular drainage device (EVD) system. typically inserted into the right lateral ventricle as not to interfere with language

29
Q

Describe purpose and priority nursing interventions for patient with a ventriculostomy

A

IVC is used to connect the ventricles of the brain to an external ventricular drainage device (EVD) system. typically inserted into the right lateral ventricle as not to interfere with language
NI:
Dependent drainage of CSF
Closely monitor output
Risk for infection, keep system sterile
Must level transducer at the tragus of the ear (per physicians order)
Monitor ICP and CPP (cerebral profusion pressure) readings

30
Q

Describe nursing care and interventions that prevents IICP in the critically ill patient

A
Maintain dressings, drains, special precautions for bone flap removal
Close monitoring of vitals and ICP
Reduce infection risk (VAP, antibiotics)
Frequent neuro checks
HOB up min 30 degrees
Low stimulation
Seizure precautions
Family teaching
Pain control
31
Q

Describe nursing implications and rationale for use of medication for head injury patient
DIURETICS:
MANNITOL
FUROSEMIDE

A

Diuretics: in attempt to lower ICP

Mannitol: hyperosmotic
Furosemide: loop diuretic (enhances Mannitol)

Nursing:
Strict I/O
Monitor for fluid overload
Monitor for dehydration

32
Q
Describe nursing implications and rationale for use of medication for head injury patient
ANTICONVULSANTS:
PHENYTOIN
DIAZEPAM
LEVETIRACETAM
LORAZEPAM
A

Anticonvulsants: to reduce the likelihood of seizures

Phenytoin: anticonvulsant
Levetiracetam: anticonvulsant

Diazepam: benzo, antianxiety, anticonvulsant, sedative/hypnotic, muscle relaxant
Lorazepam: benzo, antianxiety, sedative/hypnotic, analgesic adjunct

Nursing:
Monitor for SI
Rash
Vitals: resp, BP

33
Q
Describe nursing implications and rationale for use of medication for head injury patient
B/P MEDS:
LABETALOL
HYDRALAZINE
NOREPINEPHRINE
A

BP meds to maintain MAP (70-110 norm) within parameters

Labetalol: beta blocker, antianginal, antihypertension

Hydralazine: vasodilator, antihypertensive

Norepinephrine: vasopressor, produces vasoconstriction

Nursing:
Monitor BP
I/O
Monitor for fluid overload

34
Q
Describe nursing implications and rationale for use of medication for head injury patient
SEDATIVE/HYPNOTICS:
PROPOFOL
MIDAZOLAM
DEXMEDETOMIDINE
A

Sedative/hypnotics: to reduce stimulation, muscle contraction and overall cerebral O2 demand

Propofol: anesthetic, short 1/2 life, may decrease BP, MUST be mechanically intubated

Midazolam: benzo, antianxiety, anticonvulsant, sedative/hypnotic, CNS depression, amnesia

Dexmedetomidine: sedative/hypnotic, ICU sedation, may decrease BP and HR

Nursing:
Monitor HR and BP
SI

35
Q

Describe nursing implications and rationale for use of medication for head injury patient
STEROIDS

A

Steroids: to reduce cerebral edema
**NOT indicated in TBI, only with tumors

Dexamethasone: anti-inflammatory

Nursing:
Check blood sugar
I/O
Check edema

36
Q

Describe nursing implications and rationale for use of medication for head injury patient
HORMONES
VASOPRESSIN

A

Hormones: for treatment of diabetes and Syndrome of Inappropriate Antidiuretic Hormone Secretion (SIADH)

Vasopressin: replacement hormone, antidiuretic, vasopressor, reduce urine output and increase urine osmolality

Nursing:
I/O
Monitor BP, HR

37
Q

Describe nursing implications and rationale for use of medication for head injury patient
GI MEDS:
FAMOTIDINE
PANTOPRAZOLE

A

GI prophylaxis: to reduce likelihood of ulcers due to the reduce gut motility and stress on the body

Famotidine: antiulcer, histamine antagonist

Pantoprazole: antiulcer, proton pump inhibitor

Nursing:
Monitor for blood in stool, emesis, gastric aspiration

38
Q

Describe nursing implications and rationale for use of medication for head injury patient
ANTIPSYCHOTIC:
PHENOTHIAZINE
CHLORPROMAZINE

A

Antipsychotic: with alternate use for hiccups or shivering. Alters dopamine in CSN

Chlorpromazine: phenothiazines, antiemetic, antipsychotic

Nursing:
Monitor fluid intake
Bowel function
BP

39
Q

Describe nursing implications and rationale for use of medication for head injury patient
NEUROMUSCULAR BLOCKING AGENT:
VECURONIUM

A

Neuromuscular blocking agent: to provide paralysis in the event of uncontrolled seizures or need for induced hypothermia

Vecuronium: IV fusion

Nursing:
Monitor HR, BP
Peripheral nerve stimulator may be used to determine level of blockade induced. Determines level of paralyze of patient

Four impulse are delivered to sites of ulnar, facial or tibial nerves:
0:4 too much paralytic
2-3:4 adequate paralysis
4:4 paralysis is insufficient

40
Q

Identify priority assessments for barbiturate coma and neuromuscular blocking agents in pt with IICP

A

.

41
Q

Describe the med/surg management of patients with cerebral aneurysm

A

Out pouching of cerebral artery, most common location is the circle of Willis
If identified before rupture surgical interventions include clipping or coiling

Prevent and treat of ruptured aneurysm:
Vasospasm: use of Ca+ channel blocker
Rebleeding: treat IICP

42
Q

Describe the post op nursing care of a craniotomy patient

A

Cranial cavity is surgically opened, tumor/blood clot is excised via the bone flap removal and ultimate replacement of the bone

Post op:

43
Q

Identify nutritional modifications that are indicated for patients with head injuries

A
Patients may have swallow inability
Tube feedings might be necessary
Can increase metabolism
Increase potassium intake
Reduce carbs (too many carbs can cause CO2 buildup)
NO nasal tubes, ONLY oral tubes
44
Q

Describe post op care of the carotid endarterectomy patient

A

Surgical intervention to remove atherosclerotic plaque to prevent ischemic stroke (at the carotid artery bifurcation)
Vitals: every hour
HR: cardiac monitor
Resp: monitor airway
Pulse ox: O2
Dressing: to neck
JP drain: check
Cranial nerves: may have abnormality after surgery
HOB: 30 degrees, support head with any movement

45
Q

Describe pre and post op care for patient with corneal transplants

A

Lamellar: superficial replacement of cornea

  • *Penetrating: full thickness cornea is removed and replaced by donor
  • very fine sutures remain in place for 1 year (cornea is avascular and slow healing)
  • low risk of corneal rejection d/t avascular nature

NC:
Pain meds
Position on unaffected side with HOB 30 degrees
Administer steroid drops to reduce inflammation
Assess eye patch/dressing for drainage
Teach pt to avoid; coughing, sneezing, vomiting, straining
Sudden sharp pain in eye may indicate hemorrhage
Vision can take up to 12 mo to return

46
Q

Describe pre and post op care of patients with enucleation

A

Removal of eye d/t malignancy, infection, glaucoma, intractable pain/trauma

Outpatient
Round implant placed to maintain shape
Pressure dressing 24+ hours
Pt can be up after surgery

NC:
Observe for signs of hemorrhage
Teach pt about s/s of infection and admin of drops
Provide psychological support
Irrigation of socket may be required

Long term eye prothesis is placed within 1-2 mo

47
Q

Describe the effects a head injury has on reproductive health

A

Patient can experience:
Sexual dysfunction
Changes in hormone levels
Infertility

48
Q

What is the Cushing’s Triad?

A

Increased ICP
Increased BP
Decreased HR
Irregular respirations

49
Q

Progression of IICP (cascade)

A
Cranial insult/injury=
Tissue edema=
Increase of ICP=
Compression of blood vessels=
Decreased cerebral blood flow=
Decreased O2 with death of brain cells=
Edema around ischemic tissue=
Increased ICP with compression of brainstem/respiratory centers=
Increase CO2 accumulation=
Vasodilation=
Increased ICP as result of increased blood volume=
Death
50
Q

8 (…ate) Interventions for ICP and TBI

A
  1. Elevate
    - HOB min 30 degrees
    - avoid hip and neck flexion
    - The silence: reduce stimulation, provide block care
  2. Oxygenate
    - deliver O2 is appropriate
    - blood transfusion: monitor Hgb for anemia (acidosis is NOT good = vasodilation = cerebral edema
    - limit suctioning: coughing/vomiting can increase ICP
  3. Intubate
    - GCS of <8, notify provider
    - protect airway
    - optimal ventilation in critical patients
  4. Hyperventilate
    - normal pCO2 is 35-45 mmHg
    - pCO2 = a potent vasodilator
    - hypercapnia (high pCO2) causes: vasodilation = cerebral edema = increased ICP
    - hypocapnia (low pCO2) causes: vasoconstriction = increase pH = decreased ICP = ischemia (lack of O2)
  5. Medicate
    - BP med to maintain MAP within parameters
    - anticonvulsants
    - GI prophylaxis
    - antipsychotic
    - diuretics
    - hormones
    - steroids
  6. Sedate
    - sedatives/hypnotics to reduce stimulation, muscle contraction and overall cerebral O2 demand
    - sedation does NOT remove their pain
  7. Refrigerate
    - hyperthermia
    - hypothermia
    - goal normothermia
  8. Evacuate
    - surgical intervention
    - ventriculostomy
    - nursing care post op