2.2 Cardiovascular, Cardiac Monitoring Flashcards

1
Q

Identify and explain the significance of the P wave

A

Usually upright and uniform
Followed by a QRS complex
Represents atrial depolarization

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2
Q

Identify, measure and explain the significance of the QRS complex

A

Measured from the start of the Q to the end of S wave
Represents time from Bundle of His impulse through the ventricles
Varies in shape
Not all patients have a Q or S
Normal: <0.12 seconds

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3
Q

Identify and explain the significance of the T wave

A

Normally upright and uniform
Rounded and smooth
Inverted T waves may indicate myocardial ischemia
Peaked T waves may indicate hyperkalemia or myocardial injury
Represents ventricular repolarization
No associated muscle activity

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4
Q

How do you measure PR interval? Represents? What is the ranges?

A

PR Interval
Measure from beginning of P wave to beginning of QRS complex
Represents time from SA Node impulse through the AV Node and Bundle of His
Normal: 0.12-0.20 seconds

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5
Q

Describe parameters and identify regular sinus rhythm

A

R-R intervals are constant
P-P intervals are constant
Rate is 60-100 (counting the R wave in a 6 sec strip)
P waves are uniform with 1 P wave to 1 QRS complex
PR interval is between 0.12-0.20 seconds
QRS complex is <0.12 seconds

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6
Q

Describe nursing care for patient on telemetry

A

Verify order
Explain procedure and why monitoring is needed
Explain patient responsibilities
Prepare skin: alcohol wipe, shave skin if necessary
Correct lead placement
Connect to monitor
Set alarms specific to the patient

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7
Q

What teaching should the RN to patient and family on telemetry?

A
Keep leads on at all times
No showering
Do not adjust monitoring system
Call if lead falls off
Call if chest pain or change in heart rhythm
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8
Q

Identify and explain significance of Ventricular tachycardia dysrhythmias?

A
Ventricular tachycardia
LIFE THREATENING!
Regularity: usually regular (saw tooth)
Rate: ventricular rate 150-250 beats
P waves: none
PR interval: none
QRS complex: wide, >0.12 seconds but uniform

Most often caused by coronary artery disease, MI, cardiomyopathy, valve disease, cardiac surgery
May be pulseless or may be perfusing and even stable
Assess patient
Treat with antiarrhythmic meds if stable
Cardioversion if unstable

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9
Q

Identify and explain significance of Ventricular fibrillation dysrhythmias?

A
Ventricular fibrillation
LIFE THREATENING!
Regularity: chaotic baseline
Rate: no identifiable waves to measure
P waves: non identifiable
PR interval: none
QRS complex: none
CODE BLUE, can not produce a pulse
GOAL IS DEFIBRILLATION
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10
Q

identify and explain significance of Ventricular standstill (asystole) dysrhythmias?

A
No electrical activity
Complete arrest
No P, PR interval, QRS
CODE BLUE
IMMEDIATE CPR
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11
Q

Explain the formation, significance and treatment of sinus tachycardia dysrhythmias?

A
Regular and constant R-R
Rate >120
P waves uniform and 1 P to 1 QRS complex
PR interval 0.12-0.20 seconds and constant
QRS is <0.12

Normal sympathetic response to exercise or stress
NOT normal to persist at rest
May be caused by shock, bleeding, fever, pain, anemia, HF, hypoxia, hyperthyroidism, PE
May be caused by: caffeine, atropine, epinephrine, dopamine

Assess pt and treat underlying cause

**tachycardia at rest is an ominous sign

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12
Q

Explain the formation, significance and treatment of sinus bradycardia dysrhythmias?

A

Regular and constant R-R waves
Rate is <60
P waves are uniform, 1 P wave to 1 QRS complex
PR interval is 0.12-0.20 seconds and constant
QRS complex is <0.12 seconds

May be normal for young pts and athletes
May be caused by:
Vagal stimulation (parasympathetic)
Myocardial Ischemia
Elevated intracranial pressure
Hypothyroid (myxedema)
Medications that may cause this: digitalis, beta blockers, calcium channel blockers etc

Assess pt and treat only if symptomatic: syncope, lowered BP, chest pain, light headedness

Treatment: atropine, pacemaker

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13
Q

Explain the formation, significance and treatment of premature atrial contractions (PAC’s) dysrhythmias?

A

Regular except premature atrial contractions (PAC’s)
Rate 60-100
P wave is premature, abnormal shape at PAC only
PR interval of ectopic will measure differently than the rest of the beats
QRS is <0.12 seconds

Irritable area in the atria fires before the SA node
Common, typically asymptomatic and benign
May be caused by caffeine, nicotine, HF, pulmonary disease, myocardial ischemia
Frequent PAC’s can lead to atrial tachycardias

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14
Q

Explain the formation, significance and treatment of atrial fibrilation?

A

Regularity: grossly irregular
Rate: controlled ventricular rate if <100. Rapid ventricular response if >100
P wave: No P waves, only fibrillatory waves
PR interval: unable to measure
QRS complex <0.12 seconds

Chaotic firing of impulses from multiple points in the atria
No atrial contraction, only fibrillation
May be chronic and paroxysmal (come and go)
Risk for thrombus, requires anticoagulation

Multiple causes: cardiac surgery, almost any cardiac disease, pulmonary disease, hypoxia, hyperthyroid

Assess pt. Treatment is aimed at rate control
Meds: amiodarone, calcium channel blockers, beta blockers, digitalis

*consider cardioversion, after anticoagulation

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15
Q

Explain the formation, significance and treatment of bundle branch blocks?

A
Regularity: regular
Rate: 60-100
P waves: normal, uniform, 1 P followed by 1 QRS complex
PR interval: <0.20 second and constant
QRS complex >0.12 seconds

Delay in conduction through left or right bundle branches
Bundle of His and bundle branches are special fibers that carry impulses quickly
Assess patient, rarely needs treatment
Becomes a significant problem with both left and right bundles are involved.

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16
Q

Describe treatment and rationale for cardioversions?

A
Cardioversion:
Elective or emergent
With pulse (Afib, Aflutter, SVT, VT)
Cardiac output
Conscious patient
Synchronized
Sedation if possible
Energy level lower
17
Q

Describe treatment and rationale for defibrillation?

A
Defibrillation
Emergent
Pulseless (Vtach or Vfib)
No cardiac output
Unconscious patient
Not synchronized
No sedation
Energy level high
18
Q

Describe treatment and rationale for implantable cardioverter-defibrillators?

A

Implanted cardioverter/defibrillator (ICD)

Delivers a chock for VF/VT: synchronized or not as indicated
Procedure and postop care same as pacemaker
Not all ICDs are also pacemakers
Painful when they fire
Patient is admitted for treatment when ICD firing for medication adjustments. Transplant candidate?

19
Q

Describe treatment and rationale for pacemakers?

A

Pacemaker Indications:
Bradycardia
Sick Sinus Syndrome (tachy-brady)
Severe heart failure (increase cardiac output)

Many types:
Atrial
Ventricular (generates inverted, wide QRS, skips the bundle branches)
Atrioventricular Sequential (dual chamber)

Operate “on demand”: when heart rate falls below the set rate of pacemaker

20
Q

Identify EKG changes that accompany hypokalemia?

A

Conduction depends on sodium-potassium ion pump
Hypokalemia:
Frequent PACs and PVCs
Increased PR intervals and U wave
Risk for afib/flutter or SVT
Risk for prolonged QT or Torsades de Pointes

21
Q

Identify EKG changes that accompany hyperkalemia?

A

Conduction depends on sodium-potassium ion pump
Hyperkalemia:
Peaked T waves and increased PR interval =
Widening QRS and ventricular arrythmias = arrest

22
Q

Identify EKG changes that occur with digitalis (digoxin) therapy?

A

Digitalis (digoxin) effect appears as a down sloping ST segment depression

23
Q

Explain the cardiac cycle?

A
Sinoatrial (SA) node
Atrioventricular (AV) junction
-AV node
-Bundle of His
Left bundle branches (2 divisions): left posterior and anterior fascicle
Right bundle branch
Purkinje fibers
24
Q

Outline BLS procedures?

A

Secure the scene
Initial assessment:
Conscious? Breathing? Color?
Not responsive? Yell for help or dial 911

25
Q

What is the 5 step approach to interpretation of 6 second strip?

A
Regularity
Rate
P waves
PR Intervals
QRS complex
26
Q

Describe treatment and rationale for various dysrhythmias

BRADYCARDIA

A

Only treated if symptomatic
-signs of hypoperfusion and low cardiac output

Atropine: 1st line, Inhibits acetylcholine

Dopamine or epinephrine drip: 2nd line

27
Q

Describe treatment and rationale for various dysrhythmias

SVT

A

Attempt vagal maneuver first

  • increase parasympathetic stimulation
  • cough, hold breath, bear down
  • carotid massage by provider ONLY

Adenosine

  • goal to pause conduction, chemical cardioversion
  • short half life, rapid IV push with rapid 20ml flush

Beat blockers
Calcium channel blockers
Amiodarone
-slow rate

Cardioversion if unstable

28
Q

What are the anti-arrhythmic medications?

A

Anti-arrhythmic medications

Sodium channel blockers

Beta blockers

Potassium channel blockers

Calcium channel blockers
-diltiazem and verapamil only affect heart rate

29
Q

Describe treatment and rationale for various dysrhythmias

ATRIAL FIB/FLUTTER

A
Amiodaron
Metoprolol: beta blocker
Diltiazem: calcium channel blocker
Verapamil: calcium channel blocker
Digoxin
Sotalol: potassium channel blocker
Dofetilide: potassium channel blocker
Apixaban: anticoagulant
Warfarin: anticoagulant
Heparin/Enoxaparin: anticoagulant
30
Q

Describe treatment and rationale for various dysrhythmias

VENTRICULAR DYSRHYTHMIAS

A
Lidocaine
Beta blockers
Amiodarone
Magnesium sulfate: for Torsades de Pointes
Epinephrine/Adrenaline
31
Q

Conduction components

A

Sinoatrial (SA) Node: 60-100 beats/min
Primary pacemaker
Fastest intrinsic rate

Atrioventricular junction (AV): 40-60 beats/min
AV Node and Bundle of His (Bundle of His leads to left and right bundle branches)
Delay the SA impulse for filling: secondary pacemaker (gate keeper)

Ventricle: 20-40 beats/min
Purkinje fibers initiated conduction in the ventricles
Tertiary pacemaker (backup to the backup)