20: Orthopaedic Hip Conditions Flashcards

1
Q

Describe the blood supply to the femoral head.

A
  • The profunda femoris artery is the largest branch of the femoral artery.
  • Shortly after its origin, it gives off medial (MFCA) and lateral (LFCA) circumflex arteries.
  • The MFCA is the major contributor to the femoral head. It gives off two branches.
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2
Q

what is the clinical significance of an intracapsular fracture on femur neck vs an extracapsular fracture?

A
  • intracapsular fracture: blood supply disrupted
  • extracapsular fracture: blood supply maintained
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3
Q

what is trochanteric brusitis?

A
  • The trochanteric bursa is a fluid-filled sac sandwiched between hip abductors and IT (iliotibial) band.
  • Bursitis is inflammation of this bursa causing swelling.
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4
Q

list causes of trochanteric bursitis

A
  • trauma
  • overuse: athletes- often runners, repetitive movements

abnormal movements:
- distant problem: e.g. scoliosis
- local problem: muscle wasting following surgery, total hip replacement, osteoarthritis

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5
Q

trochanteric bursitis clinical presentation and examination cues

A
  • pain: point tenderness, lateral hip
  • LOOK: may have scars from previous surgery, may have muscle wasting: gluteals.
  • FEEL: tenderness at greater tuberosity
  • MOVE: worst pain in active abduction
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6
Q

trochanteric bursitis investigations

A
  • history and exam
  • x-ray: may be normal, can show OA, THR, spine abnormalities
  • MRI: shows soft tissues and fluid
  • US: can be therapeutic as well as dagnostic . guided injection
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7
Q

trochanteric bursitis treatment

A
  • NSAIDs
  • relative rest/activity modification
  • phsyio
  • corticosteroid injection
  • surgery: buresctomy, rarely required
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8
Q

what is avascular necrosis?

A

death of bone due to loss of blood supply

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9
Q

avascular necrosis risk factors

A
  • trauma
  • idiopathic
  • hypercoaguable states
  • steroids
  • sickle cell disease
  • lymphoma
  • leukaemia
  • Caisson’s disease
  • alcoholism
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10
Q

what is the final common pathway in avascular necrosis?

A

intravascular coagulation

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11
Q

avascular necrosis symptoms and signs

A

symptoms:
- insidious onset of groin pain
- pain with stairs, walking uphill and impact activities
- limp

Examination signs:
- can be largely normal
- may replicate early arthritis: reduced ROM, stiff joint

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12
Q

avascular necrosis investigations

A
  • history and exam
  • x-ray
  • MRI: 99% sensitive and specific, will identify earliest changes
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13
Q

avascular necrosis treatment

non-operative and operative

A

Non-operative:
- reduce weight-bearing (crutches)
- NSAIDs
- Bisphosphonates in early AVN (controversial)
- anticoagulants
- physio

Surgical:
- restore blood supply: core decompression +/- vascularised graft
- move the lesion away from the weight-bearing area > rotational osteotomy
- total hip replacement

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14
Q

Describe Femoroacetabular Impingement (FAI)

A
  • results in impingement of femoral neck against anterior edge of acetabulum
  • broadly divided into two categories: Cam lesion, Pincer
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15
Q

describe FAI - Cam lesion

A
  • femoral based impingement
  • excess bone leading to decreased head to neck ratio and aspherical head of femur.
  • abutment of lesion on edge of acetabulum
  • usually in young athletic males
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16
Q

Describe an FAI - Pincer

A

- acetabulum-based impingement
- usually in active females
- abnormal acetabulum leading to: anterosuperior acetabular rim overhange and acetabular protrusion.
- Abutment of lesion on edge of acetabulum

17
Q

Femoroacetabular Impingement (FAI) associated injuries

A
  • labral degeneration and tears
  • cartilage damage and flap tears
  • secondary hip osteoarthritis
18
Q

Femoroacetabular Impingement (FAI) presentation

A
  • groin pain: worse with flexion
  • mechanical symptoms: block to movement, pain with certain manoeuvres e.g. getting out of chair, squatting, lunging
19
Q

Femoroacetabular Impingement (FAI) exam clues

A
  • reduced flexion and internal rotation
  • positive FADIR test: flexion, adduction, internal rotation
20
Q

Femoroacetabular Impingement (FAI) treatment

A

Non-operative:
- activity modification
- NSAIDs
- physio

Operative:
- Arthroscopy: shave down defect, deal with labral tears, resect artic cartilage flaps
- Open surgery: resection, periacetabular oesteotomy, hip arthroplasty: resurfacing or replacement.

21
Q

list some causes of a labral tear

A
  • FAI
  • trauma
  • OA
  • dysplasia
  • collagen diseases e.g. Ehlers-Danlos
22
Q

labral tear presentation and exam clues

A

Presentation:
- groin or hip pain
- snapping sensation
- jamming or locking

Exam:
- can be normal
- positive FABER test: flexion, abduction, external rotation

23
Q

labral tear investigations

A
  • history and exam
  • x-ray: OA, dysplasia
  • MRI arthrogram: 92% sensitive
  • diagnostic injection with local anaesthetic
24
Q

labral tear treatment

A

non-operative:
- activity modification
- NSAIDs
- physio
- injection of steroids

Operative:
- arthroscopy: repair, resection