[2] Lecture 9: Cardiac Arrhythmias Flashcards

1
Q

HR > 100bpm
And
Causes:

A

Tachycardia

Increased body temp.
Stimulation by sympathetic nerves
Toxic conditions of the heart

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2
Q

Endogenously mediated tachycardia:[exercise]

A

HR increase
CO increase
Filling time reduced but SV doesn’t decline

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3
Q

Sympathetic stimulation when exercising maintains SV how?

A

Increase contractility
Systolic interval reduced
Increase venous return

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4
Q

What are effects of pathologically mediated tachycardia?

A

HR increases

CO decreases

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5
Q

Why do the effects of pathological mediated tachycardia occur?

A

MAP decreases and activates sympathetic NS after the fact and is unable to compensate

No muscle pump to increase venous return

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6
Q

What is bradycardia and what causes it?

A
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7
Q

Respiratory type of sinus arrhythmia

A

Medullary resp. Center into vasomotor center during inspiratory /expiration cycles….increase/ decrease HR through sympathetic and vagus nerves to the heart

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8
Q

Sudden cessation of P waves- standstill of atria..ventricles pick up AV node rhythm. QRS pace is slower but not otherwise altered.

A

Sinoatrial block

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9
Q

Ischemia of AV node or AV bundles through coronary insufficiency
Compression of AV bundles by scar tissue
Inflammation of AV node/bundle
Extreme stimulation of the heart by the vagus nerve

A

Atrioventricular block

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10
Q

Extended PR interval:

Longer w/ slower HR
Shorter w/ faster HR

> .20s PR

A

1st degree AV block

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11
Q

Extended PR that results in dropped beats of the ventricle

A

2nd degree AV degree

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12
Q

No relation between the rate of the P waves and the rate of QRS-T complexes

A

Complete Atrioventricular block

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13
Q

Resumption of the ventricular beat may be due to parts of the purkinje system acting as ectopic pacemaker

A

Ventricular escape

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14
Q

Periodic fainting spells

A

Stokes-Adams syndrome

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15
Q

An alteration in the amplitude of P waves, QRS complexes, or T waves

Referred to as electrical alternans

A

Partial intraventricular block

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16
Q

These heart beats occur before they should

Most result from ectopic focus

A

Premature contractions

17
Q

What causes ectopic foci:

A

Local ischemic areas
Calcified plaques
Irritation of the conduction system or nodes

18
Q

HR becomes rapid suddenly in bursts that last sec’s, min, hr’s, or longer
Then, ends suddenly

A

Paroxysmal tachycardia

19
Q

How can paroxysmal tachycardia resolve?

A

Pacemaker of heart instantly shifts back to the sinus node.

20
Q

The twitching of individual muscle fibers in the atria or ventricles and also in recently denervated skeletal muscle fibers

Occurs as a result of circus movements

A

Fibrillation

21
Q

3 different conditions that cause the impulse of heart to travel around the circle:

A

1-pathway around the circle is too long.
2-length of the pathway remains constant but the velocity of the conduction slows down.
3-refractory period of the muscle might become greatly shortened.

22
Q

A contraction or excitation wave traveling continuously in circular fashion around a ring of uncle or through the wall of the heart

A

Circus movements

23
Q

What condition generally causes circus movements d.t circle being too long

A

Enlarged/dilated heart

by the time the impulse returns to its starting point, the heart muscle will be in the refractory period.

24
Q

When the length of the pathway remains normal, but the conduction velocity is slowed down in heart. What 3 things could cause this?

A

1-blockage of purkinje system
2- ischemia*
3-high potassium levels

25
Q

What would cause the refractory period of the cardiac muscle to be shortened?

A

Rx: epinephrine
Or
Electrical stimulation

26
Q

The atrial muscle fibers are separated from ventricular muscle fibers by the:

A

cardiac fibrous skeleton

27
Q

Ventricular and atrial fibrillation can occur separate from each other

A

A fib

28
Q

Causes of A fib [2]:

A

Enlargement of atria-d/t valve lesions

Inadequate emptying of the ventricles causing blood to back up into the atria