15: Gait Abnormalities- Mahoney Flashcards

1
Q

4 pathways that need to be intact for normal gait

A
  • intact reflex arc (intact sensory n, functional synapse in spinal cord, intact motor n. fiber, NMJ, and competent muscle)(reflex arc is not dependent on CNS)
  • corticospinal/pyramidal tract (fine discrete voluntary movement)
  • extrapyramidal tract (manintain m. tone and control gross automatic movements)
  • cerebellar system ( by receiving both sensory and motor input, coordinates muscular activity, maintains equilibrium and helps control posture)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

hyperreflexia =

A

UMN Lesion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

normal supraspinal inhibition of antagonistic m. lost =

A

clonus

sign of UMN lesion

ex: drosiflex ankle, see oscillating motion back

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

________ weakness of arm extensors and leg flexors with UMN lesion

A

spastic

leg flexor = flexors of hip, knee and ankle dorsiflexors

= decorticate movement

therefore…. SPASTCIITY of arm flexors and leg extensors

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

hyporeflexia =

A

LMN lesion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

_______ weakness with LMN lesion

A

flaccid

observe m. atrophy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

fine movements of m. seen under skin due to sensitization to Ach

A

fasciculations

associated with LMN lesion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

unilateral UMNL –> _______ gait

A

spastic gait

aka hemiparetic/hemiplegic or STROKE gait

from a CVA, brian injury, brain abscess

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

_____ plegia =

A

spasticity

spasticity = increased muscle tone due to UMNL

due to exaggeration of stretch reflec

rate sensitive or velocity dependent (slow- normal, rapid - increased tone results)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

describe “stroke gait”

A

leg is extended and internally rotated because leg flexors weakened

leg swings laterally and forward to clear ground (circumduction)

contralateral hip may tilt downwards to prevent toes from catching floor as leg advanced forward

ipsilateral arm flexed at elbow, internally rotated and adducted; wrist flexed, fingers flexed(due to weakness of arm extensors)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q
CVA patterns
cerebral hemisphere =
brain stem =
spinal cord - cervical =
spinal cord - below cervical =
A

hemiplegia
quadriplegia
quadriplegia
paraplegia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

paraparetic/spastic diplegic or CP gait or “scissors gait”

A

bilateral UMN lesion

due to CP, CVA, MS, spinal cord disease

not CP before the age of 2 b/c brain not fully myelinated

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

type of motor impairment with scissors gait

A

spastic hemiplegia- lesions of contralateral cerebral cortex

spastic diplegia - bilateral cerebral cortex

spastic quadriplegia

atheotid CP

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

describe scissors gait

A
  • legs extended and thighs tightly adducted
  • legs circumducted
  • legs slightly flexed at hips and knees (crouching)
  • arms mildly flexed
  • mimicked by running in kene deep water
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

cerebellar ataxic aka DRUNK giat

A

cerebellar leison

broad based, spped and length of stride varies irregularly from step to step

posture is erect, feet are separated

difficulty walking tandem

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

cerebellar ataxic gait is normal in..

A

children less than 2 yo

NO positive romberg sign

difficult standing with feet together even with eyes open

17
Q

describe sensory ataxic gait

A
  • resembles drunk gait
  • problem with propioceptors or peripheral n.
  • positive Romberg test
  • commonly seen in DIABETICS with loss of positiion sense. need to look at floor to tell them where their foot is
18
Q

describe vestibular gait

A
  • pathology located in inner ear
  • falling to affected side whether standing or walking
  • asymmetric nystagmus
  • normal proprioception and m. strength exclude sensory ataxia and hemiparesis
19
Q

describe steppage gait

A
  • DROPFOOT or neuropathic gait
  • unilateral = L5 radiculopathy, sciatic neuropathy, common peroneal n. neuropathy
  • bilateral = distal plyneuropathy (diabetes*), lumbosacral polyradiculopathy

weakness of ankle dorsiflexion, leg lifted higher than normal during swing phase to prevent toes from catching on floor

20
Q

describe waddling gait

A

aka Trendelenberg or gluteus medis limp or duck walk or myopathic gait

  • proximal LL weakness due to myopahty, NMJ disease, proximal symmetric spinal m. atrophy
  • may see with hip DJD
21
Q

trendelenger gait: when m. too weak to keep the pelvis levele when the ______ foot is picked up, the pelvis will drop down on the ________ side, producing pelvic rocking

A

unaffected
unaffected

trunk tilts toward the affected side to lift hip on unaffected side and provide extra distance b/w foot and floor

pelvis rotated forward to assist with forward motion of unaffected side

usually bilateral so looks waddlingly

22
Q

describe parkinsonian gait pathology

A
  • form of extrapyramidal disease
  • leion of substantia nigra causing decreased dopamine levels
  • HYPOkinetic gait
23
Q

what does a parkinsonian gait look like

A
  • forward rigid stoop with head and neck bent forward, with modest flexion at hips and knees
  • arms flexed at elbows and adducted at shoulders, with resting pronation-supination tremor
  • trouble arising from chair
  • center of gravity tends to remain in back of legs, so once standing there is tendency to fall backwards RETROPULSE
  • gait initiated with short, shuffling steps whcih is exacerbated when turning PEDESTAL TURNS must stop before turning
  • FESTINATION once center of gravity gets in front of legs, body tries to catch up with center of gravity with increasing speed
24
Q

pathophys of choreoatheotic gait

A
  • WORMlike gait
  • extrapyramidal process
  • wildly ataxic gait/HYPERkinetic gait-gait interrupted by abrupt lg. amplitude involuntary movements
  • similar movements seen in arms, neck, face
  • balance not affected
25
Q

describe antalgic gait “limping”

A

pt favors (does not put normal weight on painful extremity which usually results in limited knee flexion and less prominent heel strike and toe off, shortened stance phase and smaller steps

seen following surgeries (injury)

26
Q

toe-walkers aka Equinas Gait

A
  • usually secondary to congenitally tight gastroc-soleus or sudden growth spurt
  • always consider neurological disorder like CP, myelomeningocele, spastic hemiplegia (especially if from birth)