13. Interpreting Abnormal ECGs Flashcards
What is sinus rhythm?
If every QRS complex is preceded by a P wave
What are atrioventricular conduction blocks?
Delay/failure of conduction of impulses from atria to ventricles via AVN and Bundle of His
What are the 3 types of atrioventricular conduction blocks?
First degree heart block
Second degree heart block: Mobitz type 1 and 2
Third degree heart block
What are the causes of heart block?
Degeneration of electrical conducting system with age - sclerosis and fibrosis
Acute myocardial ischaemia
Medications
Valvular heart disease
What happens in first degree AV block?
Conduction is slowed without skipped beats
All normal P waves followed by QRS complexes but PR interval is longer than normal (>0.2 seconds)
What happens in Mobitz type 1 second degree AV block?
Successively longs PR intervals until one QRS is dropped then cycle starts again
Electrical signal is not conducted through to ventricles
What happens in Mobitz type 2 second degree AV block?
PR intervals do not lengthen - sudden dropped QRS complex without prior PR changes
Atrial rhythm (p waves) is regular
Ventricular rhythm is irregular
High risk progression to complete heart block
What happens in third degree AV block?
Atria and ventricles depolarising independently - complete failure of AV conduction
Ventricular pacemaker takes over (20-40bpm)
Typically too slow to maintain blood pressure
Usually wide QRS complex
Urgent pacemaker required
What is a bundle branch block?
Delayed conduction within the bundle branches (right or left)
P wave and PR intervals normal
Wide QRS complex because ventricular depolarisation takes longer
Where can abnormal rhythms arise from?
Aria (superventricular arrhythmia) - sinus node, atrium or AV node
Ventricles (ventricular arrhythmias)
What is the difference in ECGs between supraventricular and ventricular arrhythmias?
Supra - normal or narrow QRS complex
Ventricular - wide and bizarre QRS complexes
Describe atrial fibrillation
Arises from multiple atrial foci
Rapid chaotic impulses
No P waves, just wavy baseline, irregular R-R intervals
Impulses reach AVN at rapid irregular rate
Not all conducted by when conducted ventricles depolarised normally so normal QRS
What are the haemodynamic effects of atrial fibrillation?
Loss of atrial contraction leads to increased blood stasis
Stasis most evident in left atrium
Flow velocity markedly reduced concomitantly with impaired contractility of left atrial appendage
Leads to small clots in LA, risk for ischaemic stroke
What is premature ventricular ectopic beats (PVCs)?
Ectopic focus in ventricle muscles
Impulse does not spread via fast His-purkinje system, so much slower depolarisation of ventricles do wider QRS
Premature because occurs earlier than would be expected for next sinus impulse
Can lead to ventricular tachycardia
What is ventricular tachycardia?
Run of more than 3 consecutive PVCs
Persistent VTACH is dangerous rhythm requiring treatment
High risk progression to ventricular fibrillation