11.11.2019 - version 2 Flashcards
The African swine fever virus can be inactivated by irradiation
F
inactivated by 2% NaOH, 0.3% formalin, hypochlorite for 30 min, or iodine
The ASF causes haemadsorption in cell cultures
T
The ASF infects the swine per os
T
The ASF virus replicates in lymphocytes
F
replicates in macrophages, monocytes, endothel, hepatocytes, tubular epithelium, and granulocytes, but NOT lymphocytes
In ASF cases abortion is never observed
F
all the 3 strains can cause abortion
In acute ASF cases the leading pathological finding are the haemorrhages
T
Vaccines are available to be used in the control of ASF
F no vaccine (no antibody production)
ASF is shed in the saliva of the infected animals
T
saliva and nasal discharge. Can start to shed 48 hours prior to the appearance of CS
The FMD virus is very sensitive to the environmental conditions
F non-enveloped and good resistance, esp in shadows, wet and cool sites of pastures. dirty stall = week Manure = 40 days frozen meat = months milk powder = years
The FMD virus can retain its infectivity in dried milk for months
T
All serotypes of FMD can be detected world-wide
F
7 serotypes: A and O found wordwide, C is rare
Swine shed FMD virus for a shorter time comparing to cattle
T
Swine: a few weeks, but amount is over 1000x more than cattle
Cattle: 0.5-3 years
Hedgehog is susceptible to FMD
T ruminants (incl wild species), pig, guinea pig, hedgehog, dog and man
The primary replication site for FMD is the tongue mucosa
F
primary replication: pharyngeal and laryngeal mucosa
After immune response the FMD vesicles can recover fully
T
For laboratory tests FMD samples should be submitted in buffered transport
medium
T
Talfan disease is a disease of pigs
T
The clinical signs of Talfan disease are more sever than that of Teschen disease
F
Talfan disease can be seen in animals below 4 months of age
T
below 4 months of age (usually btw 3-8 weeks)
Flaccid paralysis is a typical clinical sign of Talfan disease
T
transient flaccid paralysis of hindlimbs
Pigs are infected with swine vesicular disease virus per os
T
per os or through skin abrasions
Swine vesicular disease virus can cause vesicles on the snout of pigs
T
Haemorrhages can be seen post mortem in the case of swine vesicular disease
F
no gross pathological changes as they don’t die from the disease
The mortality of swine vesicular disease is very high, it can be 50-60%
F
usually asymptomatic, and in case of any CS, the recovery is fast
Duck hepatitis A virus is shed in the faeces
T
shed in feces for several weeks
The resistance of duck hepatitis A is high
T
high resistance - can survive in bedding for 10 weeks
Germinative infection can happen in the case of duck hepatitis
F
Clinical signs of the duck hepatitis can be seen in all age groups
F
affects ducklings up to 6 weeks of age
The primary source of VES infection is sea-origin feed
T
primary source of infection: raw meat of sea animals, fish
Feline calicivirus infection can cause the limping of kittens
T
can cause “limping syndrome”, which may be immunocomplex mediated, and has been observed after immunization with live, attenuated vaccines
RHD virus can be propagated in cell culture
F
has to test vaccines in live animals
The orthohepevirus A causes clinical symptoms only in human
T
Is the causative agent of hepatitis E.
CS of animals: absent (maybe reproductive issues)
CS of humans: asymptomatic/moderate/fatal