11. Basic ECG Flashcards

1
Q

Cardiac conduction pathway

A

SA node
internodal fascicles
bachmann’s bundle
AV node
Bundle of His
Right Bundle branch
Left bundle branch
purkinje fibers

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2
Q

where does the electrical current slow down in the heart

A

AV node

allows for atria to fully contract before the ventricles contract
maximizes ventricular filling

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3
Q

pacemaker cells

A

initiate heart beat
set heart rate

SA node
AV node

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4
Q

primary pacemaker of heart

A

SA node

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5
Q

SA node beats per min

A

60-100 bpm

sinus beats

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6
Q

AV node beats per min

A

40-60 bpm

junctional beats

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7
Q

ventricular muscle beats per min

A

30-40 bpm

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8
Q

p wave

A

atrial contraction

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9
Q

QRS

A

ventricular contraction

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10
Q

premature atrial contractions

A

heartbeats initiated by atrial myocardium

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11
Q

retrograde current is seen in

A

junctional beats initiated by AV node

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12
Q

retrograde current on ecg

A

inverted p wave

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13
Q

upright p wave

A

heartbeat originated from SA node or atria

ategrade conduction

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14
Q

inverted p wave

A

heartbeat originated from AV node

retrograde conduction

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15
Q

wide QRS

A

slow ventricular depolarization

heartbeat initiated by ventricular myocardium

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16
Q

1 large box

A

0.2 sec

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17
Q

1 small box

A

0.04 sec

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18
Q

1 second

A

5 large boxes

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19
Q

1 min

A

300 large boxes

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20
Q

RR interval

A

heart rate

300/#lg boxes

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21
Q

intervals include

A

a wave

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22
Q

normal p wave

A

3 small boxes
<0.12s

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23
Q

normal QRS

A

<0.12s
1.5-3 sm boxes

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24
Q

T wave

A

ventricular repolarization

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25
normal T wave
<5mm height
26
U wave indicates
hypokalemia
27
J point
point where S wave returns to baseline
28
Delta wave
upward slurring Q wave wollff parkinson white syndome
29
J wave
hypothermia bump on S wave
30
PR interval
beginning of P to start of Q
31
normal PR interval
0.12-0.2s 3-5 sm boxes
32
long PR interval
indicates delayed conduction in AV node
33
QT interval
begining of Q wave to end of T wave
34
what prolongs QT interval
zofran phenergan subarachnoid hemorrhage
35
PR segment
end of P wave to beginning of Q wave
36
ST segment
J point to start of T wave
37
premature beat
heartbeats happens before expected "fast" PAC PVC PJC
38
escape beat
heartbeat that comes oafter long pause "slow" ventricular junctional
39
systole
heart contraction heart is not perfusing organs are perfused
40
diastole
heart relaxation heart is perfused organs are not perfused
41
slower heart rate
increases coronary perfusion
42
faster heart rate
worse coronary perfusion
43
ventricular filling
amount of blood that fills the ventricles prior to ventricular contraction preload
44
decrease ventricular filling
decreased SV decreases CO
45
active ventricular filling
during atrial contraction incr preload normal SV normal CO
46
passive ventricular filling
when atria dont contract decr preload decr SV decr CO
47
heart conditions that reduce ventricular filling
1. heartbeat w/o atrial contraction (no p wave) 2. premature heartbeat 3. rapid heart rate
48
do premature heartbeats produce a pulse
no
49
ECG leads detect
electrical difference (voltage) between 2 limbs
50
3 lead ECG limitation
not as sensitive for detecting myocardial ischemia occuring in left ventricle
51
lead 1
white to black (-): RA (+): LA
52
lead 2
white to red (-): RA (+): LL
53
lead 3
black to red (-): LA (+): LL
54
what additional leads are in 5 lead ECG?
green brown (V5)
55
green lead
neutral grounding lead
56
brown lead
V5 precordial lead helps detect left ventricular ischemia
57
lead locations
white - right green - below green black - left brown - below black red - below brown
58
how to get best ECG connection
cleanse site of application w/alcohol exfoliate skin layer avoid placing over hair
59
irregular sinus
HR fluctuating w/inspiration and expiration inspiration: faster expiration: slower
60
why is inspriation HR faster
decrease intrathoracic pressure increases preload increases HR
61
why is expiration HR slower
increase intrathoracic pressure decreases preload decreases HR
62
sinus tachy
P wave HR>100 bpm
63
sinus tachy etilogies
hypovolemia hypotension pain/light anesthesia
64
sinus tachycardia causes
incr cardiac O2 demand decr cardiac O2 supply hypovolemia
65
sinus tachy treatment
give fluids deepen anesthetic beta blocker
66
sinus brady
p wave HR <60bpm
67
sinus brady is good for what pts
healthy pts who exercise pts w/CAD
68
lower HR
incr O2 supply decr O2 demand
69
bradycardia is bad in what pt population
kids
70
what heart rate is always a cause for concern
<=30bpm
71
bradycardia treatment
robinul atropine epinephrine cardiac pacing (unresponsive to meds)
72
temporary transcutaneous pacing
SA node pacing pads via defibrillator
73
pacemaker
implantable device to act as artificial SA node
74
ectopy
any heartbeat that orginates outside the SA node - AV node - atrial myocardiaum - ventricular myocardium
75
PAC
premature beat upright p wave normal/narrow QRS
76
when are PACs concerning
when they start occuring frequently
77
PJC
premature beat missing/inverted p wave normal QRS retrograde
78
when are PJCs concerning
when they start occuring frequently
79
PVC
premature beat no p wave wide/bizarre/different QRS
80
what can trigger PVC
ischemia pH imbalance electrolyte abnormalities caffeine stress abnormal electrical pathways
81
do PVCs produce a pulse
no
82
what is the pulse rate for bigeminal PVCs
half of what the ECG says the pulse rate is
83
bigeminal PVC
every other beat
84
unifocal PVC
same shape = same origin
85
multifocal PVC
different shape = different origin
86
what is more concerning: unifocal or multifocal PVC?
multifocal
87
couplet PVC
2 in a row
88
salvo PVC
3 in a row
89
PVC treatment
antiarrythmics - lidocaine (100mg) - amiodarone robinul (speed HR up)
90
junctional escape beat
escape beat inverted/missing p wave normal QRS
91
junctional escape beat physiology
SA node failed AV node starts heartbeat SA node starts working again
92
when should we be concerned w/junctional escape beats
if they occur frequently treat w/robinul/atropine/pacing
93
junctional escape beat treatment
robinul/atropine pacing
94
ventricular escape beat
long pause wide QRS no p wave
95
ventricular escape beat physiology
SA and AV node fail ventricular myocardium initiates beat SA node works again
96
atrial flutter
sawtooth p waves (250-350/min) wide p waves
97
atrial flutter physiology
decr ventricular filling decr CO incr O2 demand
98
when are we concerned about atrial flutter
would not do an elective case until pt has been evaluated by a cardiologist
99
type 1 atrial flutter
<350bpm
100
type 2 atrial flutter
>350bpm
101
atrial flutter treatment
amiodarone, sotalol, digoxin synchronized cardioversion (for hypotensive pts)
102
shocking heart
treats unstable fast rhythms
103
pacing heart
treats unstable slow rhythms
104
Afib
no p waves narrow QRS may or may not have fibrillation waves irregularly irregular rhythm
105
Afib physilogy
atria quivering w/500 atrial impulses/min
106
Afib risks
clot formation in LA (blood pooling) CO decr hypotension
107
Afib CO decreases by
25-50%
108
what type of afib is more concerning: chronic or acute?
acute afib is more concerning suffer significant drop in CO
109
controlled afib
ventricular rate <100bpm
110
uncontrolled afib
ventricular rate >100bpm\ rapid ventricular response
111
afib treatment
medications synchronized cardioversion
112
how long should a pt be anticoagulated prior to cardioversion
3 weeks prior 4 week post
113
junctional rhythm
inverted/absent P normal QRS
114
normal junction
40-60bpm
115
accelerated junctional
60-100 bpm
116
junctional tachycardia
>100bpm
117
junctional rhythm concerns
slower HR reduced ventricular filling
118
junctional rhythm treatment
robinul to incr HR
119
SVT
HR >150bpm normal QRS may or may not have P waves
120
SVT concerns
decr ventricular filling decr CO
121
SVT treatment
vagal maneuvers adenosine (slows SA/AV) synchronized csrdioversion antiarrythmics
122
ventricular escape rhythm (idoventricular)
no p wave wide WRS slow HR <60bpm
123
ventricular escape physiology
SA and AV node failed ventricular myocardium is beating
124
ventricular escape concerns
no active ventricular filling HR is low low CO
125
ventricular escape treatment
cardiac pacing epinephrine avoid lidocaine (suppresses ventricular beat)
126
accelerates idivoentricular rhythm
60-100bm
127
Vtach
>100bpm no p waves wide QRS (same shape)
128
polymorphic Vtach
torsades de pointes twisting
129
Vtach physiology
ventricular myocardium initiates heart beat at rapid rate high O2 consumption minimal ventricular filling
130
does vtach have a pulse?
potentially
131
Vtach treatment
electrical cardioversion
132
monomorphic vtach treatment
amiodarone lidocaine
133
polymorphic vtach
magnesium
134
amiodarone does what to QT interval
prolongs it
135
Vfib
no real p waves or QRS complexes shorter deflections
136
Vfib physiology
quivering ventricles rapid rate high O2 consumption no pulse no CO
137
Vfib treatment
defibrillation CPR
138
agonal rhythm
slow complex rhythm immediately preceding asystole
139
do agonal rhythms produce cardiac output
nope
140
asystole
cardiac arrest CPR epinephrine do not defibrilate
141
pulseless electrical activity (PEA)
pt has no pulse ECG shows electrical activity
142
PEA is most likely seen with what rhythms
sinus bradycardish ventricularish AV blockish slowish type
143
cause of PEA
heart does not contract insufficient cardiac output to generate pulse and supply blood to organs
144
PEA treatment
CPR epinephrine NO defibrillation
145
1st Deg AV block
long PR interval >200msec (1 lg box)
146
2d Deg AV block
dropped QRS complex
147
2d Deg AV Type 1
dropped QRS increasingly long PR intervals
148
2d Deg AV Type 2
dropped QRS unchanging PR intervals
149
where is the block in 2d deg type 2
below the AV node in bundle of his or bundle branches
150
3d deg AV block
wandering P waves slow ventricular rate
151
3d deg AV block physiology
AV node completely blocekd ventricles must initiate heart beat atria and ventricals are not synchronized decreased CO
152
treatment for complete heart block
cardiac pacing epinephrine AVOID lidocaine
153
pacemaker atrial lead
sense when atria is contracting
154
pacemaker ventricular lead
paces 120-200msec after each atrial beat
155
Ischemia/Infarction ECG indications
ST segment changes abnormal T waves abnormal Q waves
156
ST depression indicates
ischemia
157
ST elevation indicates
infarction
158
myocardial ischemia treatment: increase O2 supply
1. 100% FiO2 2. decrease HR --give beta blockers 3. mx normal BP ---avoid hypotension 4. give NTG 5. give aspirin
159
myocardial ischemia treatment: decrease O2 demand
1. decr HR --give beta blockers 2. avoid pain/anxiety/tachycardia --give narcs/sedatives 3. avoid high afterload --avoid HTN