10-30 L3 Cranial nerves Flashcards

1
Q

Olfactory hallucinations

A

damage to the primary cortical olfactory area in the temporal lobe (masses or seizures)

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2
Q

Anosmia/hyposmia

A

Fractures of the floor of the anterior cranial fossa involving the cribriform plate
-CSF Rhinorrhea
tumors, abscesses, meniniomas: compression of olfactory bulb or tract

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3
Q

Anomalous trichromatism

  • protanomaly
  • deuteranomaly
  • tritanomaly
A

Anomalous trichromatism: defective color vision

  • protanomaly: R cones abnormal
  • deuteranomaly: G cones abnormal
  • Tritanomaly: B cones abnormal
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4
Q

Dichromatopsia

  • protanopia
  • deuteranopia
  • tritanopia
A

dichromatopisa: defect in matching colors, b/c instead of 3 wavelengths you have 2.
- protanopia: R cones absent
- deuteranopia: G cones absent
- Tritanopia: B cones absent

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5
Q

What’s the most common form of color blindness?

A

Anomalous trichromatism: deuteranomaly

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6
Q

What is the path of the visual field from the upper half

A

Upper visual field–>lower retina–> loop of meyer–>lingual gyrus

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7
Q

Damage to the visual system can be caused by?

A
defects in the development (anophthalmia, cyclopia)
vascular problems (diabetes, occlusion)
trauma to the face or head
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8
Q

Anophthalmia

A

abscess of one or both eyes

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9
Q

Cyclopia

A

failure of the embryonic prosencephalon to properly divide the eye in to two cavities.

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10
Q

Papilledema

A

increased intracranial pressure

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11
Q

Glaucoma

A

excessive intraocular pressure

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12
Q

Cataract

A

loss of transparency of the lens

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13
Q

Monocular blindness

A

damage to the optic nerve results in the loss of input from the ipsilateral eye only
-the pt. will complain of blindness in that eye.

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14
Q

What happens to the visual field if the chiasm is compromised (pituitary tumor).

A

if the medial aspect of the chiasm is compromised decussating axons are affected leading to loss of visual input from the nasal hemiretinas in both eyes
(bitemporal hemianopsia)

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15
Q

What happens to the visual field if the lateral aspect of the chiasm is damaged? (e.g. lesion due to an aneurysm)

A

Input from the temporal retinal half of the ipsilateral eye is lost, this results in loss of the ipsilateral nasal visual field.

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16
Q

What happens to the visual field if a lesion of Meyer’s loop (anterior part of the temporal

A

results in a homonymous superior quadratic anopsia.

17
Q

What happens to the visual field when damage to the optic tracts, lateral geniculate nucleus, optic radiation, or visual cortex?

A

results in loss of input from the contralateral visual fields of both eyes.

18
Q

Any cortical lesion is going to have what distinctive feature?

A

Macular sparing

19
Q

Cochlear or spiral ganglion consists of cells located where?

A

In the spiral configuration at the periphery of the modiolus.

20
Q

Where does high and low freq occur in the cochlea?

A

high freq. (base of the cochlea)

low freq. (apex of the cochlea)

21
Q

Where will a lesion resulting in deafness occur?

A

a lesion up to and including the cochlear nuclei will result in deafness

22
Q

What happens when an interruption of the lateral lemniscus occurs?

A

decreased ability to localize sound

23
Q

Name the different types of conductive hearing loss (4-DODO)

A

Damage to the tympanic membrane
Occlusion of the auditory canal (cerumen impaction)
Disruption of the operation of the ossicular chain
Otitis media

24
Q

Cerumen impaction

A

wax in the ear

25
Q

Otitis media

A

an inflammation of the middle ear cleft

26
Q

Disruption of the operation of the ossicular chain (FOD)

A
  • Fluid accumulation (Otitis media)
  • Otosclerosis
  • Disarticulation
27
Q

Name the different types of sensorineural hearing loss (2-DP)

A
  • Disorders affecting the cochlea

- Problems in the transmission of information along the auditory pathways.

28
Q

Disorders affecting the cochlea (4- HOMP)

A
  • Hydrops
  • Ototoxis agents
  • Meniere’s disease (fluid build up in the modiluosu)
  • Presbycusis (getting old)
29
Q

Problems in transmission of information along the auditory pathways

A

lesions to cranial n VIII

30
Q

Review slide

What are the 3 tracks leaving the vestibular nuclei (counter rotation of the eye).

A
  • abducens nucleus (6) opposite
  • oculomotor nucleus (3) same
  • abducens nucleus same side to go to (oculomotor nucleus opp, and LR same)
31
Q

Damage to the vestibular apparatus causes what?

A
  • dizziness, falling to the side of the lesion, abnormal eye movements
  • Benign paroxysmal vertigo (BPV)
32
Q

Acoustic neuroma (schwannoma)

Facial n component

A

weakness of muscles of facial expression
lost of taste (anterior 2/3 of the tongue)
Lack of stimulation of salivary, mucosal and lacrimal glands on affected side (7th n)

33
Q

Acoustic neuroma (schwannoma)

Vestibular component

A

dizziness, nausea, disorder of balance

34
Q

Acoustic neuroma (schwannoma)

cochlear component

A

tinnitus, ipsilateral deafness

35
Q

Meinere’s disease (SAUL)

A
  • Severe vertigo, vomiting, ataxia
  • Auditory and vestibular symptoms
  • Unilateral deafness and tinnitus
  • Lesion affecting the production of the endolymph
36
Q

Motion sickness (Kinetosis)

A
  • occurs when there is discrepancy between vestibular and visual inputs
  • Symptoms include dizziness, vomiting, sweating
37
Q

Acute alcohol intoxication

A

Alcohol infiltrates the cupulae

results in illusions of movement in response to certain orientations of the head.

38
Q

Which of the CN are SA?

A

CN II, III, VII (2/3 anterior taste), VIII (balance, hearing), IX (taste around the epiglottis)