1 cardio lecture Flashcards

1
Q

Angina

A
  • the main manifestation of the coronary artery disease (CAD)
  • Typical angina (definite):
    1) Substernal chest discomfort with a characteristic quality and duration that is2) provoked by exertion or emotional stress and 3) relieved by rest or NTG.
  • Atypical angina (probable): meets 2 of the above characteristics.
  • Noncardiac chest pain: meets one or none of the typical anginal characteristics. (AHA 2009
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2
Q

CAD

A

discrepancy between the amount of oxygen supplied to the cardiac tissue and the needs;

  • other cause of angina: inflam.involving the coronary art.(syphilis, vasculitis); angina“with normal coronary arteries“
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3
Q

Typical angina

A
  • S:Precordial area
  • R:left uper limb cubital side to last 2 fingers
  • C:Constriction ppressure squeezing
  • T:no more than 15 minutes
  • E:Extra demand on the heart exercise, an emotional upset, exposure to cold, a heavy meal
  • rest nitroglycerine
  • S:Aanxiety dyspnea
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4
Q

Atypical angina

A
  • S:Interscapular, upper part of the body, epigastrium; sometimes: just in areas of radiation
  • R: ​jaw Posterior limbs posterior Thorax
  • C: weakness discomfort
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5
Q

[*] Prinzmetal angina (variant angina):

A
  • Coronarian spasm – hyperactivity of sympathetic nervous system
  • During sleeping (REM phase- rapid eye movement)
  • Severe pain, anxiety, dyspnea
  • Same hour of the night (or day)
  • Elevation of the ST segment (diff.dg. with acute myocardial infarction)- occurs only during the crisis !!!
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6
Q

Non-ischaemic pain:
Pericarditis

A

Pericarditis

  • Substernal pain, like a “knife”(dry p.) or pressure (p.effusion) or absent
  • Left border of the sternum or epigastrium radiating to the neck and arms
  • increases: pressure over the thorax, respiratory movements
  • decreases: leaning forward – antalgic position in dry pericarditis and against dyspnea in pericardial effusion !
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7
Q

Pain due to aortic causes

A
  • Aortitis (syphilis)
  • Aortic aneurysm
  • Acute dissection of aorta:
    –suddenly and maximal intensity onset;
    –anterior thoracic pain is associated with anterior arch or aortic root dissection
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8
Q

Forms of dyspnea in cardiac patients:

A

–Exertional d. – progressive: the intensity of the effort leading to dyspnea declines progressively

–Dyspnea at rest with orthopnea

–Paroxysmal d.: ACUTE LVF- 3 types:

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9
Q

–Paroxysmal dyspnea

A

Nocturnal paroxysmal dyspnea​

  • a sudden awakening of the patient, after a couple hours of sleep, with a feeling of severe anxiety, breathlessness, and suffocation- orthopnea
  • the manifestations are relieved spontaneously
  • Episodes of this may be so frightening that the patient may be afraid to resume sleeping

Cardiac Asthma

  • Bronchial obstruction is associated

Acute pulmonary Edema

  • Transsudate al alveoli level
  • Severe dyspnea and cough- frothy pink sputum
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10
Q

Exertional d. – progressive: the intensity of the effort leading to dyspnea declines progressively

A
  • the degree of activity necessary to induce this symptom: difference between healthy-cardiac subjects
  • CHRONIC LVF (CAD, hypertensive cardiopathy, valvular heart diseases etc)
  • The severity of d. decreases when RIGHT cardiac failure occurs !!! – pulmonary stasis decreases when peripheral (sistemic) stasis develops
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11
Q

–Dyspnea at rest with orthopnea

A

[*] LV advances
[*] is relieved with elevation of the head - “how many pillows do you need?”
[*] develops when the patient is awake, within min.after the recumbency
[*] Cough- nonproductive, during recumbency- an “orthopnea equivalent.”

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12
Q

Palpitations:

A
  • An unpleasent sensation in which a person is aware of an irregular, hard, or rapid heartbeat.
  • Due to hyperexcitability of the central nervous system or abnormal cardiac function (rhythm, the force of contraction)
  • A very common symptom
  • Physiological cond. (coffee, smoking, effort, stress)
  • Cardiac diseases- arrhythmias (any cardiopathy)
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13
Q

Palitations

The correlation with a cardiopathy is supposed when:

A
  • Palpitations- persistent and frequent
  • Associatedwith chest pain,dyspnea
  • history of a cardiac disease
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14
Q

Orthopnea

A

Acute LVF

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15
Q

Immobile

A

during crisis off angina

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16
Q

Agitation, no specific position to relieve the pain

A

AMI

17
Q

Squatting

A

congenital cardiopathy (redistribution of the blood to the brain)

18
Q

Leaning forward-

A

pericarditis

19
Q

corvisart face

A
20
Q

cyanosis

A

–Central: congenital cardiopathy (R L shunt)
–Periph.: RCF
–Mixed: cor pulmonale

21
Q

Palor

A

Low cardiac output

  • CF
  • Arrythmias
  • acute myocardial infarction
  • crisis of angina

​Cafe au lait : pallor+ cyanosis –> infectios endocarditis

22
Q

Jaundice

A

Chronic liver stasis

RCF

Chronic Pericarditis

23
Q

clubbing finger

A
  • congenital cardiopathies
  • endocarditis
  • cor pulmonale
24
Q

Meynet nodules

A

reuhmatic fever

(on the back of the wrist, the outside elbow, and the front of the knees), erytema marginatum (well-demarcated edge and clearing central portion)

25
Q

signs for infectious endocarditis

A

[*] Osler nodes:
–Septic embolisation- fingers, toes
[*] Janeway lesions:
–Haemorrage the thenar and hypothenar eminences
[*] splinter haemorrhages on fingernail

26
Q

Fever (37-38 Celsius):

A

infective endocaditis

27
Q

Systolic epigastric pulsations:

A
  • right ventricle dilatation= Harzer sign,
  • abd.aortic aneurysm,
  • tricuspid regurgitation
  • thin people: N.!- abdominal aorta
  • -Emphysema: low position of the diaphragm allows the transmission of the normal activity of the RV
  • -Hepatomegaly-stasis: the normal abd.aortic pulsations are transmitted to the enlarged liver
28
Q

apex impulse

A

= the point of maximum outward movement of the left ventricle during systole

  • corresponds to the onset of ventricular ejection or systole
  • one third of the systole
  • *-IVth – VI th intercostal space** on the midclavicular line
  • Dorsal, lateral decubitus (! A correction of 2cm.to the right)
  • Area of 2 cm2
  • Location, amplitude, size
29
Q

Apex impulses

Displacement

A

With normal position of trachea:
–Left ventricular enlargement - the apex is displaced downwards and laterally.
–Right ventricular enlargement - the apex will displaced laterally.
–Cardiomegaly
–Pectus excavatum (unusual location)
–Situs inversus/ dextrocardia

With shifted trachea
–Away from the hemithorax in case of: pleural effusion, pneumothorax
–Towards the hemithorax: atelectasis, fibrosis (Pressure differrence)

30
Q

increased intenity of apex impulses

A

–LV hypertrophy,
–Pressure overload: aortic stenosis, systemic hypertension- forceful sustained (a dome-shaped impulse)
–Volume overload: aortic, mitral regurgitation- produces a less sustained impulse- hyperdynamic apex impulse

31
Q

decreased intenity of apex impulse

A

–Dextrocardia
–apex behind a Rib
–Pericarditis (left)
–Obesity and thick chest wall
–Pericardial effusion
–Emphysema

32
Q

Double apex beat

A

[*] atrial contraction (atrial hypertrophy) or late outward movement of a ventricular aneurysm

33
Q

Tapping apex beat-

A

the first sound is loud and may be felt

34
Q
  1. Thrill
A

= a palpable murmur- a vibration produced by the physical energy of a loud sound

-“le fremissement cataire” = purring of a cat

  • diastolic thrill, Vth left intercostal space, increased in left lateral decubitus- mitral stenosis
  • Systolic thrill, 2nd right intercostal space- aortic stenosis
35
Q

Pericardial rub

A
  • pericarditis (dry)
  • During diastole and systole
  • Increased when leaning forward
36
Q

murmur grading

A

I/VI Barely audible

II/VI Audible after a few seconds of auscultation, low intensity

III/VI Immediately audible, moderate intensity

IV/VI Loud intensity without a precordial thrill

V/VI Loud intensity with a precordial thrill

VI/VI Loudest intensity, precordial thrill, audible with stethoscope slightly away from thoracic wall

37
Q

murmur

A

–Described according to their timing in the cardiac cycle; duration
–Location
–Radiation
–quality- tonal effect (rumbling)
–Pattern-shape
–Frequency (pitch)- relates to the velocity of the blood; high pitched murmurs occur when the blood flow from a high pressure chamber to a lower
–Grade (degree of intensity)– I-VI
–Increased- decreased intensity