Wound Care Flashcards
Standard Precautions
They are “standard” because every patient should be viewed as infected.
Transmission based precautions
- 2nd tier, KNOWN infection
9 elements of standard precautions
- Hand hygiene
- PPE
- Safe handling and disposal of sharps
- Cleaning and decontamination
- Safe handling of waste
- Safe handling of linens
- Respiratory and cough hygiene and etiquette
- Aseptic non-touch technique
- Safe handling of blood and body spillage
Hand hygiene
Single most important
Before and after
15-30 seconds
Turn off tap and open door with paper towel
Nail brush
PPE
If contact with blood or fluids is a possibility
Gloves, gowns, face shields
Safe handling and disposal of sharps
Disposal containers
Cleaning and decontamination
Clean after use, single use, shared items
- visibly dirty to sertilize
Safe handling of waste
Wear appropriate PPE
Don’t smoke or eat
Wash hands
Dispose as appropriate
Safe handling of linens
Always wear gloves
Roll up
Do not carry against body
Designated container (not shared with clean)
Respiratory and cough hygiene and etiquette
Cover your cough, tissue-disposal
Mask
Separation and ventilation as appropriate
Aseptic non-touch technique
Sterile technique
Safe handling of blood and body spillage
PPE as appropriate
Approved cleaner
Red bag
Who is responsible for safety precautions?
Your employer
- Responsible for providing all needed equipment
- Training you
- Developing policies
- Keeping up with infections
* impact to funding
- Treating you if you become infected
You
- Following policy
Transmission-based precautions
When you know a client has a highly transmissible disease (contact, droplet, or airborne)
- depends on disorder
- gloves, gowns, mask as appropriate
Contact precautions
Clean hands before entering and when leaving the room
Put on gloves and gown before room entry. Discard before room exit.
Use dedicated or disposable equipment. Clean and disinfect reusable equipment before use on another person.
Droplet precautions
Clean hands before entering and when leaving the room.
Make sure their eyes, nose, and mouth are fully covered before room entry and remove before exiting.
Airborne precautions
Clean handing before entering and when leaving the room.
Put on a fit-tested N-95 or higher level respirator before room entry and remove after exiting and closing the door.
Door to room must remain closed.
Causes of wounds
Surgery
- incisions
Trauma
- lacerations
- abrasions
- fractures
- burns
- punctures
O2 loss
- pressure sores
*decubitus (can be helped by repositioning to remove pressure)
* bedsore
- gangrene
- infections
Venous failure
- blood stops flowing
- spontaneous
Stages of wound healing
Inflammation
- vascular and cellular responses
- cannot occur in dead tissue
Proliferation
- healing begins
- as early as 48 hours after injury
Maturation
- strengthening and reorganizing of new tissue
- 6 months to 2 years after an injury
- scar tissue is never more than 80% the tensile strength of normal tissue
What happens in the inflammatory stage?
Body immediately responds to control body loss, prevent infection and fluid loss, signals cells necessary for repair.
- transudate
- Localized blood vessels constrict for several minutes-prevent blood loss.
- Platelets aggregate at the site of injury to create a clot.
After 30 minutes
- Excudate is formed
- Histamine and prostoglandins are released, causing redness, warmth, and swelling.
At the cellular level
- margination (call are attracted to walls)
- phagocytosis (bacteria and debris)
- macrophages (secrete enzymes and growth factors)
- mast cells (secrete inflammatory mediators)
Scab begins to form
Cardinal signs of inflammation
Swelling - tumor
Redness - rubor (erythemal)
Warmth - calor
Pain - dolor
Decreased function - functio laesa
Problem with inflammation
Lack of inflammatory response - AIDS and HIV
Chronic inflammation/excessive for injury
How to mediate inflammation
Elevation
Ice
MEM (manual edema mobilization/retrograde massage)
AROM
Compression
Debridement
Meds
Proliferation
Once the cells necessary for repair and regeneration reach the site of injury, the proliferation phase begins.
Can be as quick as 48 hours.
4 phases:
1. Angiogenesis
- The formation of new blood vessels (buds)
- Supply nutrients to the wound
- Tiny red dots
2. Granulation
- Debris and bacteria is removed
- Granular tissue provides temporary lattice - later replaced by scar tissue
- Fibroblasts begin to lay down extracellular matrix
3. Wound contraction
- Myofibroblasts pull the wound margins together
- Degree of contraction is based on shape, depth, and size
4. Epithelialization
- Epithelial cells multiply to fill the wound
Problems during proliferation
Hypogranulation
Hypertrophic granulation
General failure
- shear injury
- infection
- maceration: rotting skin
Too wet, too dry
Over cleaning
Maturation and remodleing
Granular tissue laid down in the proliferative phase must be strengthened and reorganized.
- This is called scarring
- This process can take up to 2 years
- Most change occurs in the first 6 months
- A scar that is pink is still remodeling
- A flesh color is finished
Opportunity to affect the scar
Hypertrophy and contracture
Occurs with any wound
- hope to see it soften and flatten after a few weeks
- redness is normal - vascularity decreases by 6 months to 2 years
A scar that is thickening is referred to by many as hypertrophic.
Excessive scaring - raised and outside the boarders of the wound is keloid, some don’t differentiate between hypertrophy and keloid
Reasons for hypertrophy or keloid
Genetics
Race
Depth of burn or injury
Infection
Length of time to heal
What do we do during the maturation and remodeling phase?
Desensitization
- C fibers which carry pain can be overridden by stimulating the large A fibers.
* pressure, rubbing, vibrations, TENS, motion, and function
* typically 10 minute sessions at level of irritation 4-5x a day
Scar management
- pressure
* wraps
*elastomers
*Jobst
- massage
- modalities
*iontophoresis
Surgery
- extremes
- problems
Stages of pressure sores
Stage I: a reddened area on the skin that, when pressed, is “nonblanchable.” This indicates that a pressure ulcer is starting to develop
Stage II: the skin blisters or forms an open sore; the area around the sore may be red and irritated
Stage III: the skin breakdown now looks like a crater where there is damage to the tissue below the skin
Stage IV: the pressure ulcer has become so deep that there is damage to the muscle and bone, and sometimes tendons and joints
Stage V: full thickness tissue loss in which the base of the ulcer is covered by slough and/or eschar in the wound bed
Factors that will affect healing
Circulation
Sensation
Mechanical stress
Age
Nutrition
Comorbidities
Medication
Behaviors
- alcohol use
- smoking
Superficial first degree burn
Sunburn, brief flash burn, brief chemical or heat burn
Effects epidermis only
Painful, no blisters, only redness
Healing: 3-7 days
No scarring likely
Superficial partial thickness second degree burn
Severe sunburn, contact with stove, prolonged exposure to chemical or heat
Affects epidermis and dermis
Redness, wet, blisters, painful
Healing: 2 weeks
Minimal chance of scarring, usually only if prolonged or infected
Must pop blisters if:
- the fluid isn’t clear
- it’s on a joint
- it significantly impacts function
Deep partial thickness second degree burn
Prolonged exposure to heat or strong chemicals
Epidermis and most of dermis, but enough to heal
Redness, painful, large blisters, often pop up to to shear size
Typically heal in 2-3 weeks
Can convert to full thickness if infection occurs
Likelihood of scarring increases, lack of use, contractures and deformities
Full thickness third degree burn
Extreme heat, prolonged heat
Damage through epidermis and dermis, no viable skin under
Not painful due to damaged nerve endings, but surround tissue (2nd and 1st degree burns) will be painful
Pale in color, nonblanching, dry, black in some cases
Requires surgery (grafting) if over 3x3 (silver dollar)
Scarring very likely
Grafting most likely