Why do we gain weight?- Lecture 3 Flashcards

1
Q

NHS (2016)

Obesity rates

A

62% of adults in UK are overweight or obese

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2
Q

Most at risk

A

Dieters
Students
Low SES indiv.
Shift workers

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3
Q

Dieters and weight gain

-physiological explanation

A

-dieting is period of starvation so increases fat storage and food cravings
-Reductions in leptin (hormone produced by fat that inhibits hunger) and insulin
-Increase of ghrelin (associated with increased hunger)
Increased food salience and desire to binge eat

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4
Q

Dieters and weight gain

-psychological explanation

A
  • disconnect with internal signals of hunger and fullness
  • stop listening to internal bodily signals likely to rely more on external cues
  • relying on external cues in an obesogenic environment can lead to excess food intake
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5
Q

Shift workers and weight gain evidence…

A

Sun et al. (2017)- meta analysis- shift workers 29% more likely to be obese

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6
Q

Shift workers and weight gain

-Physiological explanation

A

FAT ABSORPTION

  • Disruption of circadian rhythm may influence digestion
  • Protein NFIL3 in gut during day is disrupted and can malfunction
  • when mice sleep in day less NFIL3 produced so gain weight

METABOLISM

  • circadian rhythm also influence metabolic rate
  • shift patterns have slower metabolisms (around 8% less) – (Buxton et al., 2018)
  • Insulin levels also drop- lead to cravings for sweet energy dense products
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7
Q

Shift workers and weight gain

-Psychological explanation

A

Tend to increase snack food consumption (Gifkin et al., 2018)- often only food available

  • Increased evidence of depression & stress (Driesen et al., 2010)
  • Greater levels of emotional eating and other bad coping strategies- e.g. smoking, alcohol abuse, reduced exercise
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8
Q

Low SES and weight gain

A

In past- social disadvantage used to be associated with undernourishment- swapped around in 1970s (Ban et al., 2018)

TYPE OF FOOD AVAILABLE

  • Freedman & Bell (2009)- due to food outlets in certain areas- density of fast food outlets is higher in deprived areas
  • evidence linking food outlets to obesity is correlational and weak (Cobb et al., 2015)
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9
Q

Wansink, Painter & North (2005)

A

Self Filling Bowls- People use the bowl as cue for how much to eat
-73% more eaten in one condition but felt equally
-Fullness determined by how much people ‘believed’ they had eaten
Findings have been replicated (Brunstrom et al., 2012)
EXTERNAL CUES POWERFUL

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10
Q

Low SES and the Insurance Hypothesis

Physiological Explanation

A

When we think availability of food is not certain- most species seek to store resources (Nettle, 2017)
->humans respond in similar manner - Nettle et al. (2018); Sim et al. (2018)

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11
Q

Nettle (2017) (Insurance Hypothesis)

A

suggests that because females can become infertile when undernourished, they are more likely to store food during times of uncertainty

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12
Q

Insurance Hypothesis Evidence:
Nettle (2018) study
Sim et al (2018) study

A

-food insecure individuals ate more in a taste test
And moderated by childhood experience of food insecurity

-manipulated how deprived people felt by asking them to imagine scenarios where they were mistreated= Relative deprivation predicted intake

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13
Q

Limitations of Insurance Hypothesis Evidence…

A
  • Correlational
  • Not tested in actual food insecure populations (e.g. food bank visitors)
  • Rationers didn’t have higher rates of obesity
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14
Q

Social disadvantage and coping mechanisms

A

Hemmingsson (2014) model posits that psychological and emotional distress is a fundamental link between socioeconomic disadvantage and weight gain- how we cope with these stresses such as emotional eating.
-BUT only theoretical model…

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15
Q

Spinosa et al (2018)

A

Tested out Hemmingssons model ->Psychological distress and subsequent emotional eating represent a serial pathway that links lower SES with obesity

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16
Q

Freshman 15

A

In first year of Uni- av. weight gain of 15 pounds (1 stone)
Due to:
1.Change in eating habits- meals higher in calories, uncertainty about nutrition
2.Late night eating- less governed body clock
3.Stress/emotional eating- lots of work
4.Drinking- social norms at uni
5.Snack eating- whats available

17
Q

What makes an Obesogenic Environment?

A

Variety
Distraction
Portion Size

18
Q

Variety

Obesogenic Environment

A

Variety stimulates appetite
Even different forms of the same flavour
Sweet and savoury most dramatic as at either ends of flavour
Even in ‘fake’ variety (Kahn & Wansink, 2004) i.e. smarties in diff colours

19
Q

Variety

Obesogenic Environment

A

Variety stimulates appetite
Even different forms of the same flavour
Sweet and savoury most dramatic as at either ends of flavour
Even in ‘fake’ variety (Kahn & Wansink, 2004) i.e. smarties in diff colours
Surrounded by highly palatable foods that are cheaper than nutririonally rich foods

20
Q

Why does variety make us eat more?

A

Sensory specific satiety- become satiated when eating the same food
New sensory properties of food leads to increased appetite
Bodies are designed for scarcity but operate in times of plenty

21
Q

Psychological explanation behind variety effects

A

Using heuristics

Decide how filling a food is by weight (Spence et al, 2014) or by volume (Keenan et al., 2015)

22
Q

Distraction

Obesogenic environment

A

Performing multiple tasks (phone, watching tv) whilst eating, increases consumption (Oldham- Cooper et al., 2011).
IMPORTANCE OF MEMORY
Bottom-less bowls (Wansink, painter & North, 2005)
Could be that we require memory to code how much we’ve eaten

23
Q

Brunstrom et al. (2012)

A

Amnesic patients find difficult to register fullness as their episodic memory is impaired- Participants who thought they had consumed the larger 500-ml portion reported significantly less hunger- perceived hunger over actual

24
Q

Portion size

Obesogenic environment

A

Applies to all types of foods and drinks, meals and affects almost everyone- bar very young
children

25
Q

Why we have large portion sized

A

Average sizes have increased in

26
Q

Why we have large portion sized

A

Average sizes have increased in restaurants, super sized meals and drinks
Especially if taught to clear our plates as children- no waste

27
Q

Wansink & Wansink (2010)

A

Portion sizes have increased over the years- evident in images of the last supper- in art throughout history
BUT- not very stringent, art is interpretation and therefore may have been manipulated by artist (SUBJECTIVE)

28
Q

Culture and portion size

A

Cross-culturally significant?- Portion size effect seen in tribes who have not been subjected to western influence
BUT-French and North Americans both have high fat and sugar diets, but Americans have much higher rates of Obesity

29
Q

Brunstrom et al. (2012)

A

The Samburu Tribe in Kenya- when given more, they consume more.

30
Q

The French Paradox

Wansink et al. (2007)

A
French= eat till no longer hungry
Americans= eat till full
31
Q

Robinson and Kersbergen (2018) DOGS

A

Dogs given larger portion sizes also eat more= portion size effect is ingrained and innate, no matter what species

32
Q

Ways of staying a healthy weight…

A
Avoid:
night shifts
diets
intake of fatty foods
variety
Large portions
Distractions whilst eating
>>>Much like a Mediterranean lifestyle
33
Q

Mediterranean lifestyle

A

Physiological explanation= foods consumed are lower in fat (veg)
Psychological = Normal sized portions, low levels of variety, distraction, stress and dieting
=== shows how a combination of healthy physiological and psychological factors helps maintain a healthy lifestyle– minimising risk factors for obesity

34
Q

Bilman, Van Kleef and Triip (2017)

A

Many many external cues that can override internal cues of hunger
Different stages of consumption that can be affected by external cues
-Consumers in affluent societies are seriously affected by cues in the direct environment in which food decisions take place- as food is often readily available
–Big link to external cues in our obesogenic environment and development of Obesity