Weeks 7-13 Flashcards

1
Q

What is prevention?

A

A type of intervention aiming to reduce the likelihood or impact of a disease.

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2
Q

Difference in universal or selective prevention scope.

A

Universal: deal with risk regardless of anyone’s vulnerability (vaccine programs).
Selective: target groups at elevated risk (AIDS prevention).

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3
Q

Difference in primary and secondary prevention.

A

Primary: prevent disease before it occurs.
Secondary: reduce the impact of an already occurred disease.

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4
Q

Explain promotion.

A

A type of intervention aiming to improve health and wellness to the highest point.

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5
Q

Explain the idea of intervention using education. What is wrong with this intervention?

A

The idea that knowledge is power - if someone knows the risk of a behaviour, they will change accordingly. Critiques - we know that having knowledge does not make behaviour change.

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6
Q

Explain the health belief model.

A

Factors that impact someone taking action are: the perceived susceptibility of catching illness, and the perceived severity of the illness, how motivated someone is, and the perceived benefits and barriers to the action. An action is impacted by someones self-efficacy (capability to complete action), and cues to action.

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7
Q

Explain the theory of reasoned action and the theory of planned behaviour.

A

TRA: attitudes toward a health behaviour and the subjective norms of society influence someone’s intention to complete a behaviour. If both are positive, intention will develop and behaviour occurs.
TPB: follows the same pattern but acknowledges that intention is not always enough to change behaviour. Perceived behaviour control influences whether someone allows for intention to overtake behaviour or not.

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8
Q

Explain the transtheoretical model.

A

Five stages that make change - can move between them all. Precontemplation: individual unaware of harm and doesn’t want to make change. Contemplation: plans to make change in next 6 months. Preparation: prepared to make change soon and wants to help well-being. Action: recently changed their behaviour and attitudes and want to maintain it. Maintenance: maintaining behaviours for more than 6 months.

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9
Q

What are the 4 strategies Rothman et al. (2015) believe can be used to create healthier habits?

A
  1. Motivating action through thinking of others.
  2. Aid translation of intention into action by making if-then plans.
  3. Disrupt existing habits by changing environments and making policies that make it harder to follow unhealthy habits.
  4. Develop routines that create new habits by piggybacking habits.
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10
Q

Explain Conner’s study about using Rothman’s strategy of motivating action through others regarding dangerous alcohol use.

A

Tracked alcohol consumption throughout the year when being given social or health messages. Social messages were more effective than health messages, especially when tailored to that population.

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11
Q

Explain the three principles to initiate change.

A

Three principles are:
Capability - must have the physical and psychological ability to complete behaviour.
Motivation - have the mechanisms to activate or inhibit behaviour.
Opportunity - to complete it.

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12
Q

What three factors are a part of informing people about behavioural decisions?

A

Framing effects: framing the message impacts its perception.
Order of information: topics at the front generally chosen first.
Comparison: comparing one action to overall health can influence behaviour.

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13
Q

What four factors are part of incentivizing people about behavioural decisions?

A

Behavioural incentives: showing incentive to use a behaviour.
Social norms: setting norms in the environment about what others are doing.
Social comparisons: how my behaviour compares to others.
Prosocial motives: motivated to give to others.

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14
Q

What are the four factors that are part of guiding behavioural decisions?

A

Cues and prompts.
Defaults: make one action a default.
Recommendations: health professionals enrolling patients into treatment plans.
Self-control devices.

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15
Q

Define nudging.

A

Structuring the environment to promote positive behaviour with minimal resistance from people.

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16
Q

What are the strengths and weaknesses of nudging?

A

Strengths: low cost, easy, apply on a large scale.
Weaknesses: limited effect size, publication bias, may only work on some people, may backfire.

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17
Q

What is psychoneuroimmunology?

A

A biomedical concept that considers how the brain, behaviour, and immune system interact.

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18
Q

Define a pathogen. What is a pathogen-specific response?

A

Pathogen: microorganism that causes disease.
Pathogen-specific response: when the body recognises a pathogen as dangerous, and then initiates a response to remove it.

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19
Q

What are the two branches of the immune system and how do they differ in time and location?

A

Innate immune response: not adaptive to time, not specific, quick.
Adaptive immune response: specific to certain issues in the body, slow.

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20
Q

What does the innate immune response consist of?

A

External barriers like skin, and internal defences such as inflammation, fever, and chemical signals.

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21
Q

Explain the purpose of inflammation.

A

The body swells and becomes hot and painful. Inflammation occurs when there is a harmful stimuli in the body, and it tries to cure it.

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22
Q

Difference between acute and chronic inflammation. Why is acute good and chronic bad?

A

Acute inflammation: arises from tissue or organ damage. Fast onset and lasts a few days.
Chronic inflammation: arises from complex factors. Slow onset and can last for months or years.
Acute is good because it signals our immune cells where the infection is and it helps to repair tissue. Chronic is bad because the body is on alert which can damage tissue.

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23
Q

Explain the purpose of a c-reactive protein and what the levels mean.

A

CRP is produced in the liver in response to acute inflammation. Want low levels as this indicates lack of sickness.

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24
Q

Explain the link between inflammation and depression. Can anti-inflammatories reduce depression?

A

Proinflammatory cytokines are elevated in depression. A study found that anti-inflammatory agents can reduce depression, especially in conjunction with anti-depressants, when compared to a placebo.

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25
Q

How do diet, inflammation, and depression all link?

A

Moderate adherence to a Mediterranean diet is shown to reduce depression, and reduced inflammation is also linked to depression. Healthy diet is linked with reduced depression partially due to decreased inflammation.

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26
Q

Explain the link between acute stress and immune response.

A

Acute stress enhances immune response - especially innate responses. More cells are in circulation sending inflammatory agents to the site of inflammation. After this, immunosuppression occurs where the cortisol reduces the immune system back to baseline.

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27
Q

Explain the link between chronic stress and immune response.

A

When stress is chronic, there is increased cortisol. This increased cortisol suppresses the immune system below baseline, inhibiting immune functioning. This makes people more susceptible to biological illness.

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28
Q

Explain the Kiecolt-Glaser and Loving (2005) study about hostile marriages and wound healing.

A

All couples completed questionnaires to assess their hostility. All given blisters, and then assessed for their healing. Those in hostile marriages healed at 60% of the rate of non-hostile couples. The non-hostile people healed faster each day, and faster overall.

29
Q

Explain the link between emotional style and cold perception by Cohen (2003).

A

All assessed for their personality style, and then provided with a cold. Symptoms assessed. Positive styles showed the same level of objective and subjective symptoms. Negative styles showed the same objective symptoms, but had higher subjective symptoms. They more likely to interpret physical sensations as something negative.

30
Q

What are telomeres and what do they represent?

A

Telomeres are caps on the end of DNA strands. Each time DNA replicates, part of the telomere is lost. Telomeres are a biomarker of biological aging.

31
Q

What is telomerase and what is it’s function?

A

A type of enzyme that can replenish telomere caps on certain cells.

32
Q

Explain the link between inflammation, stress and telomeres.

A

Chronic inflammation increases telomere shortening. Chronic stress also increases telomere shortening due to the activation of cortisol releasing free radicals that damage cells.

33
Q

Explain the results of the Wong et al. (2014) study about inflammatory markers and telomere length.

A

C-reactive protein (a measure of inflammation) significantly predicts reductions in telomere length over time. Higher rates of inflammation at the baseline show shorter telomere length over time. The shorter length is a biomarker for cellular aging.

34
Q

Explain the results of the Epel et al. (2004) study looking at the telomere length of mothers with ill children and stress.

A

Mothers with sick children showed reduced telomere length and increased stress. Showed a link between the two - increased perceived stress and shorter telomere length. Stressed mothers also have less telomerase.

35
Q

Explain the link between early life stress and telomeres.

A

Children who experienced violence in their home had reduced telomeres over time. Their telomeres were also shorter at the baseline, as well as at the end.

36
Q

Explain the link between telomeres and intrauterine stress.

A

Exposure to stress in the womb is associated with shorter telomere length in adulthood by 3.5 years.

37
Q

What are the three psychological factors that influence susceptibility of catching COVID19?

A
  1. Psychological factors can influence biological susceptibility to the virus through the immune system.
  2. Psychological factors can influence behavioural susceptibility to the virus.
  3. Psychological factors can influence acceptability of public policy.
38
Q

Explain what biological factors increase susceptibility to COVID.

A

The big 4 health behaviours, poor sleep, obesity, chronic stress, lower positive emotional styles, poor social support, SES, psychiatric conditions.

39
Q

What three factors did Cohen (2020) state increase susceptibility to respiratory illness?

A

Risky behaviors, psychological stress, social relationships.

40
Q

Explain some of the psychological risk factors that influence the likelihood of catching COVID.

A

Germ aversion, pathogen disgust sensitivity, certain personality traits, political orientation, racial and socioeconomic disparities.

41
Q

Explain the findings from Shook et al. (2020) regarding psychological characteristics that people use to avoid getting sick.

A

People who scored higher on germ aversion and pathogen disgust sensitivity were more concerned about COVID and more likely to engage in preventative behaviours. People with high neuroticism had high concern for illness, but were not likely to engage in preventative behaviours. Conscientiousness showed high concern and engaging in preventative behaviours.

42
Q

Explain the differences between acute and chronic pain.

A

Acute pain: pain with rapid onset that goes quickly. Associated with SAM activation.
Chronic pain: paid with a slower onset, lasts over 6 months, can often predict onset of depression. Associated with HPA activation.

43
Q

Explain nocioceptive pain.

A

Pain from injury or damage to the body and is due to nocioceptors - pain receptors in the body that initiate a pain sequence.

44
Q

Explain neuropathic pain.

A

Malfunctioning nerves send pain signals or the misfiring of somatosensory system in the brain.

45
Q

What ways can pain be measured?

A

Behavioural observation, self-report visual analogue, questionnaires, facial scales, ecological momentary assessment.

46
Q

Explain the difference between experienced and remembered pain.

A

Remembered pain tends to be higher in intensity than experienced pain, as it is biased when reporting pain from over a long period.

47
Q

What are peak and end effects of pain?

A

The tendency to prioritize high intensity pain episodes and more recent pain episodes when summarizing pain.

48
Q

What is duration neglect?

A

The tendency to ignore pain-free periods of time when summarizing pain.

49
Q

Explain bottom-up effects when referring to pain.

A

Nocioceptors at the site of injury are activated and carry nerve impulses from the site of injury up to the brain through the spinal cord. Gate in the spinal cord is usually open and pain will go to the brain.

50
Q

What can be done to reduce or remove the pain of a bottom-up effect?

A

Activation of A-delta fibers can close the gate at the spinal cord and can prevent pain signals from A-delta fibers (sharp pain) and C fibers (dull pain) to reach the brain. Touching or rubbing the pain activates A-delta fibers, reducing pain.

51
Q

Explain top-down effects when it comes to pain.

A

Descending analgesic nerves (brain to spinal cord) with receptors that match exogenous opiates and endorphins. When these are ingested or activated, they inhibit ascending pain signals by closing the gate from the spinal cord to the brain through activating inhibitory interneurons.

52
Q

How is the anterior cingulate cortex (ACC) involved in pain?

A

It is involved with how distressed we feel in response to pain. Involved in how we feel toward a sensory component.

53
Q

What are the common links between emotional and physical pain?

A

Anterior cingulate cortex can cause increased pain-evoked activity and anterior insula.

54
Q

What are the common links between social and physical pain?

A

Social isolation and rejection increases pain-evoked activity in the ACC regions similar to physical pain.

55
Q

What four psychological factors increase pain?

A
  1. Pain catastrophizing: tendency to magnify the threat value of a pain stimulus.
  2. Negative emotion: increases pain-evoked activity in the ACC.
  3. Isolation and rejection: pain evoked activity in the ACC.
  4. Greater brain connectivity and dysfunction of pain inhibition pathways: increases chances of developing a chronic pain condition.
56
Q

What four psychological factors decrease pain?

A
  1. Distraction: reduces the transmission of pain signals ascending from the spinal cord.
  2. Reduction in emotional or social pain: mindfulness decreases activity in the ACC.
  3. Self-induced analgesia: increases endorphin activity and activated descending analgesic pathways.
  4. Psychological treatments: can reduce the experience of pain.
57
Q

Define complementary and alternative medicine.

A

Treatments to cure illness and improve health and well-being that are outside of the standard westernized medical approach.

58
Q

What evidence is there for acupuncture? What happened to it?

A

Cho et al. (1998) found that there was correlations between acupuncture spots on the foot and the brain - when the point of the foot was acupunctured, brain showed activation of the visual area.
The evidence was later retracted.

59
Q

What was the findings from Linde et al. (2005) regarding non-specific effects of acupuncture on pain?

A

Sham and acupuncture both showed reduced headaches compared to controls. Shows that acupuncture process may work, but the needling process to specific points of the body is not needed as needling anywhere seems to work.

60
Q

What is the difference between acupuncture and sham?

A

Acupuncture places needles into specific parts of the body that ‘correlate’ to specific parts of the brain. Sham needles do not go into the skin, and they do not go into specific parts of the body.

61
Q

What three mechanisms are possible for how placebos influence pain?

A
  1. Placebos may change the sensation coming up the spinal cord. Placebos may dampen the sensory signals coming from the brain stem.
  2. Placebos may change our interpretation of the sensory signals. Placebos dampen the interpretation within the prefrontal cortex.
  3. Placebos may change the way we report our pain. Placebos change our verbal reporting in the Broca’s area.
62
Q

How do placebo analgesics dampen sensory signals coming from the periphery?

A

Activate endogenous opioids, which activate descending analgesic nerves, which inhibit ascending pain signals by closing the gate from the spinal cord to the brain.

63
Q

Explain the purpose of Naloxone and how it works.

A

Naloxone is an opioid antagonist that reverses overdose.

64
Q

What have studies about Naloxone shown regarding placebo effects and pain?

A

Naloxone also blocks placebo effects as well as opioids. Placebos have blocked the pain, but since Naloxone blocks the placebo, pain increases again when Naloxone is present.

65
Q

How are placebos similar to our emotional responses to movies?

A

Placebos mirror some of the common everyday processes like movies by activating corresponding psychobiological mechanisms in the brain, even though we are aware they are not real.

66
Q

Explain the six caveats/complexities.

A
  1. Expectancies are important - stronger effect if expectations are clear and if drug is expensive.
  2. Visual input is important - drug works better if someone see’s it being administered.
  3. Most studies find placebo non-responders and responders - some researchers only analyze the responders rather than booth. Individual differences in responding is not known.
  4. Placebos are not without negative consequences - nocebo effect of making you feel worse instead of better.
  5. Placebo is a misonomer (there are multiple mediated placebo effects) - expectancies, beliefs, instructions can all influence pathways.
  6. Placebos could be more broadly defined as stimulating an active therapeutic process occurring within a psychosocial context - idea that circumstances, people etc are part of the body’s approach to healing itself.
67
Q

What are the three stages that information goes through when it is released? At what stage does the bias tend to occur?

A

Original article, university press release, news publications.
University press release is the main source of exaggeration.

68
Q

Explain the results about the Sumner et al. (2014) study about media bias within the different media sources.

A

Studied what part of media released exaggerated advice, made unfair causal claims, or made inaccurate human inferences. Results showed that university press is the main source of exaggeration. These exaggerated press releases caused increase likelihood of bad advice, causal claims, or human inference.