Week 7 Trunk and Spine Flashcards

1
Q

*Epedemiology of Back pain in children and adolescents

A
  • Population-based studies have demonstrated that low back pain is common in children and adolescents.
  • Factors that do not seem to contribute
    to back pain in school age children include:
    – Heavy back pack
    – Computer use
    – Physical activity
  • Low back pain is more common in school-age children:
    – high levels of psychosocial difficulties,
    – conduct problems, or
    – other somatic disorders.
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2
Q

*Describe various causes of Acute/chronic LBP in Children and adolescents

A

ACUTE =
- herniated discs,
- vertebral fractures - mc from MVA; falling from height
- muscle strains - physical activity, no radiating pain
- A slipped apophysis similar Sx to a herniated disc,
with pain radiating down the legs.

CHRONIC =
- developmental problems =
Scheuermann's disease
inflammatory spondyloarthropathies,
- psychological problems.
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3
Q

*Discuss the management of a child/adolescent with scoliosis dirrerenciating between idiopathic structural soliosis and non-structural scoliosis

A
™ Considerations:
– Skeletal maturity
– Growth potential
– Curve magnitude (>40 degree Cobb’s angle)
– Co-existing conditions (pathology, epilepsy)
– CP status
– Psychological factors
– Risk v benefits

IDIOPATHIC STRUCTURAL SCOLIOSIS
>25 degrees will be monitored by ortho team.
– 25-40 conservative Rx (bracing and exercise)
– >40 is surgical candidate. (especially if progressive)

IDIOPATHIC NON-STRUCTURAL SCOLIOSIS
- Correct any LLD / other underlying problem
- Promote physical activity (general fitness)
™- Specific stretch and strength activity if impairments
found
- Monitor child’s growth and any progress of curve
™- Usually disappear when underlying problem treated

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4
Q

*Describe the various types of scoliosis

A

™ Classification
– By age
– = Infantile onset

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5
Q

*Define Scheuerman’s Disease

A

Growth disorder of the spine with narrowing of the intervertebral discs, wedge vertebrae formation, collapse of endplates and kyphosis in the affected area.
=> TS, LS or thoracolumbar

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6
Q

*Discuss Etiology of Congenital malformations of the spine

A

-MC acquired during pregnancy.
™- FHx only around 1% of cases - usually associated
with multiple anomalies.
™- MC causative factor is assumed to be toxic damage during the pregnancy.
- FOLATE deficiency appears to play a role in the development of MYELOMENINGOCELE .

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7
Q

*Define spinal injuries

A

Fractures of the vertebral bodies and/or vertebral
arches, ligament injuries and/or dislocations of the
axial skeleton with or without neurological lesions.

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8
Q

Spinal Growth

A

Ossification
- Begins late fetal period and continues after birth
- Primary centres of ossification – spinous, transverse
and articular processes.
- Secondary centres – vertebral body, spinous &
transverse processes, ribs

0-3 = RAPID growth
™ 3- onset of puberty = relatively LINEAR growth
™ During adolescent growth spurts = RAPID growth

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9
Q

When do you refer a confirmed scoliosis patient?

A

All types and all degrees above 10 (really 20deg) should be referred
to ortho team for review and ongoing monitoring.

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10
Q

*Scheuerman’s Disease Etiology

A

™ MECHANICAL factors = adolecent athletes tall girls >6 foot
™ ENDOGENOUS factors = more frequent in families - not conclusively proven
™ POSTURE = increased kyphosis not conclusive
™ PSYCHOLOGICAL factors
™ OSTEOPOROSIS = decreased bone density “chicken or egg”

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11
Q

*Scheuerman’s Disease clinical features

A
™ An adolescent presenting with SEVERE
back pain with NO Hx OF TRAUMA
one should always consider the
possibility of lumbar or thoracolumbar
Scheuermann’s disease.
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12
Q

*Scheuerman’s Disease prognosis

A

= Fixed, thoracic kyphoses of LESS than 50° do
not represent a problem in adulthood, back pain is no more common or intense than in normal individuals.
= Cosmetically visual deformity is psychological burden (particularly for women).

= Fixed, thoracic kyphoses of MORE than 50°, back pain is no more frequent, but is likely to be MORE INTENSE than in normal individuals.
= Lung function may be impaired in very severe kyphoses.
= Kyphosis of more than 70° can also be progressive in adulthood.

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13
Q

*Scheuerman’s Disease management

A

Tx Options =
BRACING: Kyphosis >50 °, Patient has growth potential;
- Provide stabilisation + may reduce in younger pop.

SURGERY: Kyphosis >70 °; Mainly for cosmetic reasons

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14
Q

What are the radiologic features + associated diseases of Scheuermann’s Disease?

A
  • wedge vertebrae
  • narrowed IVD space
  • schmorl’s nodules
  • apophyseal ring herniation

Associated Diseases
– = Scoliosis
– = Spondylosis

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15
Q

*spinal injuries Etiology

A

d

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16
Q

*spinal injuries clinical features

A

d

17
Q

*spinal injuries prognosis

A

d

18
Q

*spinal injuries management

A

d

19
Q

*Congenital malformations of the spine - classification

A

NEURAL TUBE defects,
– defects of SEGMENTATION
– defects of FORMATION

20
Q

*Congenital malformations of the spine - NEURAL TUBE defects + example of MC

A
  • neural tube fails to completely close during the fourth week of embryonic development.
  • Structures overlying these midline abnormalities are severely affected and may be unable to form.
21
Q

*Congenital malformations of the spine - – defects of FORMATION

A

• absence of a structural element of a vertebra.
• Any region of the vertebral ring may be
affected:anterior, anterolateral, posterior, posterolateral
and lateral.
• Hemivertebrae
• Wedge vertebrae

22
Q

Congenital malformations of the spine - Associated Deformity

A
- Fusion of the ribs
™- Spinal dysraphism = Around 20–30% of patients with congenital malformations show intraspinal anomalies.
™- Pulmonary dysfunction
-™ Renal Malformations
™- Heart defects (7%)
™- Sprengel deformity (6%),
™- Cleft lip and palate (4%)
™- Shortening of the extremities (4%),
-™ Clubfoot (13%) and
-™ A talus verticalis (1% )of cases.
23
Q

*Congenital malformations of the spine - clinical features, prognosis,

A

h

24
Q

*Congenital malformations of the spine -– defects of SEGMENTATION

A

BLOCK VERTEBRAE = bilateral failure of segmentation
UNILATERAL BAR = unilateral failure of segmentation
UNILATERAL BAR & HEMIVERTEBRA = unilateral failure of segmentation

25
Q

h

A

Type determined by the pattern of involvement
of the vertebral arch, spinal cord, meninges and
overlying dermis.
™ The spinal cord defect may present at any
vertebral level with the most common being the
lumbosacral region, which is associated with the
final component of neural tube closure.