Week 7 finished Flashcards

1
Q

Simon is a 34 year old marketing coordinator for an accounting firm. He usually keeps fit by entering several triathlons and fun runs each year; he tells you he would typically average around 30km of running and 40km cycling each week.
Simon has just returned to Melbourne after 14 months working in Beijing where he was so busy with work that the only real exercise he got was the odd game of golf. He decided the only way he would get back into shape was to sign up for a half marathon; he’s training for this when he comes to see you.
Simon presents to you complaining of bilateral shin pain, which began shortly after his return to running. The pain initially came on after a run and he continued to train with the pain, in the hope it would resolve spontaneously. He’s consistently had the pain during and after running for about 4 weeks now and has decided it’s time to seek help.

A

xx

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2
Q
  1. What are the attachments and functions of the two menisci? Which is more commonly injured and why?
A

Two (med. & lat.) wedge-shaped, fibrocartilaginous discs, are attached to the tibial condyles

FUNCTIONS:
- Serve to increase the curvature of the tibial condyles, increasing joint congruency.
- assists in weight distribution, friction reduction, shock absorption and stability.
Horns and periphery are well innervated with nociceptive and proprioceptive fibres, indicating a role in providing information about joint position and motion and tissue deformation.

ATTACHMENTS:

  • Medial meniscus = semi-circle
  • Lateral meniscus = 4/5 of a circle
  • The open ends are termed horns and are attached to their respective intercondylar tubercles.
  • Some fibres of the anterior cruciate ligament (ACL) join the horns.
  • The anterior horns are joined by the transverse ligament.
  • Coronary ligaments anchor the periphery of each meniscus to the plateau.
  • Each meniscus is attached to the patella by capsular thickenings called patellomeniscal ligs.
  • The medial meniscus is also attached to the medial collateral ligament and the semimembranosus muscle.
  • The lateral meniscus is also attached to the posterior cruciate ligament (PCL) and popliteus muscle.

INJURIES:
Medial compartment of the knee carries vast majority of weight. Twisting force, particularly to a flexed knee may result in meniscal tear. Media meniscus is torn much more frequently due to less mobility (attachment to medial collateral, semimembranosus, ACL and more numerous coronary ligaments) of the meniscus itself but increase ROM and weight bearing of the medial compartment.

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3
Q
  1. Which parts of the menisci have vascular and neural supply?
A

Horns and periphery are well innervated with nociceptive and proprioceptive fibres.

Only the outer third of the meniscus is vascularised so often there is little effusion.

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4
Q
  1. What are plicae and bursae?
A

Plicae = folds of synovial tissue which occur to various extents in individuals.

Bursae = There are a great number of bursae associated
with the knee complex. These act to reduce friction between the many tendons and ligaments and bony structures.
- The four bursae, which communicate with the synovial cavity:
- Suprapatellar
- Popliteal
- Anserine
- Gastrocnemius.

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5
Q
  1. What is a popliteal cyst?
A

Popliteal or Baker’s cysts = fluid filled sacs in the popliteal region and usually result from chronic knee effusion.
The cyst may be a herniation of gastroc or semimem bursa or the synovial membrane.

  • Largely painless, they may cause restricted movement and consequent ache in adults.
  • Often resolve spontaneously.

NOT TO BE CONFUSED WITH A POPLITEAL ANEURYSM
o Will be painful
o Will be pulsitile and have a bruit.
o Oedema and pain in popliteal fossa
o May stretch tibial nerve referring to medial calf, ankle and foot

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6
Q
  1. List the four major ligaments of the knee and the motions resisted by each
A

MEDIAL COLLATERAL LIGAMENT:
Attaches to the medial aspect of the medial femoral condyle and inserts into the medial aspect of the proximal tibia (below the plateau). Blends with the joint capsule and attaches to the medial meniscus.
- Resists valgus stress, especially in the extended knee. (However, it may actually be more significant in the flexed position, when other ligaments are more lax)
- Checks lateral rotation & Taut in extension

LATERAL COLLATERAL LIGAMENT:
Strong cord extending, posteriorly, from the lateral femoral condyle to the head of the fibula
- Resists varus stress
- Resists lateral rotation combined with posterior displacement of the tibia
- Taut in extension

ANTERIOR CRUCIATE LIGAMENT
Attaches to the anterior tibia and extends, posteriorly and superiorly, to attach to the posterior part of the inner aspect of the lateral femoral condyle.
- Resists anterior displacement of the tibia on the femoral condyles.
- Checks both internal and external rotation.
- Medial rotation as ACL is tensed as it winds around the PCL and lateral rotation as it is stretched over the lateral femoral condyle.

POSTERIOR CRUCIATE LIGAMENT
Attaches to the posterior tibia and runs superiorly and anteriorly to attach to the inner aspect of the medial femoral condyle.
- Shorter and less oblique than the ACL
- Resists posterior displacement of the tibia on the femur.
- Resists varus and valgus stress
- Tension in the PCL in extension may be instrumental in creating the external rotation of the tibia, critical to the locking mechanism.
- Limits internal rotation

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7
Q
  1. Describe the locking mechanism of the knee and its role. What is the action of popliteus?
A

o Longer medial condyle continues to roll and slide posteriorly (after lateral has stopped), creates involuntary rotation.
- Open kinematic chain: External rotation of tibia
- Close kinematic chain: Internal rotation of femur
o This rotation brings the knee joint into the close-packed position, in which the intercondylar
tubercles are lodged in the intercondylar notch, the menisci are tightly wedged between femoral and tibial condyles and the ligaments are taut.
o This mechanism allows the patient to maintain an erect posture, with an extended knee, for long periods, with little muscular activity

To initiate flexion, this mechanism must be unlocked by internal rotation of the tibia on the femur by the poplitus muscle, or passive knee flexion.

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8
Q
  1. Why is the knee prone to dysfunction caused by loose bodies in the joint? What conditions predispose to loose bodies?
A

Because the knee locks in full extension with very little space between joint surfaces, the knee is very
susceptible to pain caused by loose bodies:
o Osteochondritis dessicans (avascular necrosis of joint surface – 1-3 loose bodies)
o Osteochondral fracture
o Osteoarthritis
o Synovial chondromatsis (snow storm knee)

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9
Q
  1. List the functions of the patella.
A
  • Anatomical pulley
  • Increases arm lever of the quads by deflecting the action line away from the axis of the joint.
  • Reduces friction between the quads tendon and femoral and tibial condyles.
  • Centralises the pull of the quads.
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10
Q
  1. Define genu valgus and varus. How do these influence patella dislocation?
A

Genu Valgum: Knock Knees
a medial tibiofemoral angle that is:
- >195 degrees
- or a Q angle of >20 degrees.

It causes increased compressive forces on the lateral condyles and increases tensile force on the medial ligaments. It also creates an increased lateral pull on the patella.

Genu Varum: Bow Legs
A medial tibiofemoral angle that is:
- < 180 degrees.
Rarely measured using Q angle.

Creates increased compressive forces on the medial condyles and tensile stress on the lateral ligaments.

Patella more likely to dislocate laterally, usually due to genu valgum, hence more common in females (females have a greated Q angle)

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11
Q
  1. List the borders and contents of the popliteal fossa.
A
  • Superior lateral border: Biceps femoris insertion
  • Superior medial border: Semimembranosus and semitendinosus
  • Inferior lateral border: Lateral head of gastrocs
  • Inferior medial border: Medial head of gastrocs
  • Roof: Skin and fasica
  • Floor: Popliteal surface of the femur, oblique popliteal ligament and the popliteal fascia.

Contents:

  • Popliteal vessels (artery, vein, lymph)
  • Fat
  • Tibial and common peroneal nerves
  • Small saphenous vein
  • Popliteal lymph nodes
  • Popliteal bursa
  • End branch of the posterior femoral cutaneous nerve
  • Articular branch of the obturator nerve.
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12
Q
  1. List the muscles and major vessels and nerves present in each compartment of the leg.
A
POSTERIOR COMPARTMENT:
      NERVES:
- Tibial Nerve
     ARTERIES:
- Post Tibial Artery &amp; Veins
- Peroneal Artery &amp; Veins
     MUSCLES:
Superficial layer:
- Soleus
- Gastrocnemius
- Plantaris

Deep layer:

  • Popliteus
  • Tibialis posterior
  • Flexor digitorum longus
  • Flexor hallucis longus
LATERAL COMPARTMENT:
     NERVES: 
- Superficial Peroneal Nerve
     ARTERIES:
- Peroneal Artery
     MUSCLES:
- Peroneus longus
- Peroneus brevis
- Peroneus tertius
ANTERIOR COMPARTMENT:
     NERVES:
- Deep Peroneal Nerve
     ARTERIES:
- Ant Tibial Artery &amp; Veins
     MUSCLES:
- Tibialis anterior
- Extensor hallucis longus
- Extensor digitorum longus.
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13
Q
  1. Where can each component of the peroneal nerve be compressed or injured?
A

Common peroneal nerve:
o Superficial and therefore most commonly injured
o Supplies mm in lateral and anterior compartments = foot drop
o Severed or stretched in fractures of fib head or by direct trauma to the side of the knee/fib head.

Superficial peroneal nerve:
o Stretch in ankle sprains (causing pain, paraesthesia and numbness in the lateral leg and dorsum of the foot)

Deep fibular nerve:
o Entrapment may follow overuse or injury of the
anterior compartment resulting in anterior compartment pain extending to the dorsum of the foot and between 1st and 2nd toes.
o Tight fitting ski boots often compress this nerve under the extensor retinaculum, causing pain and numbness in the dorsum of the foot and between the 1st and 2nd toes.

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14
Q
  1. List the superficial veins of the lower extremity and give a brief description of their location.
A

GREAT SAPHENOUS VEIN:
Originates from where the dorsal vein of the big toe (the Hallux) merges with the dorsal venous arch of the foot. It passes anterior to the medial malleolus. Ascends on the medial aspect of the leg and at the knee, it runs over the posterior border of the medial epicondyle of the femur bone.
Then courses anteriorly to lie on the anterior surface of the thigh before entering an opening in the fascia lata called the saphenous opening. It forms an arch, the saphenous arch dives into the femoral triangle to drain into the femoral vein.

SMALL SAPHENOUS VEIN:
Begins where the dorsal vein from the fifth digit (smallest toe) merges with the dorsal venous arch of the foot.

From its origin, it courses around the lateral aspect of the foot (inferior and posterior to the lateral malleolus) and runs along the posterior aspect of the leg (with the sural nerve), where it passes between the heads of the gastrocnemius muscle.

Usually it drains into the popliteal vein, at or above the level of the knee joint.

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15
Q

Clinical application
Simon decided to seek treatment because the shin pain has become obvious when walking and after even a light jog the pain is lingers for some time and may continue into the night. He also tells you that his first few steps in the morning are excruciating.
On examination, you note medial shin tenderness, particularly distally. Both feet are relatively pronated. Active resisted inversion and plantar-flexion reproduces his pain.

A

XX

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16
Q
  1. What is the likely cause of Simon’s pain?
A

Shin splints
o Tib ant and post
o Tendonitis then progresses to myositis
o That progresses to perositis
o Can progress to stress fracture (greater stress at periosteum, with already tight mm)
If do not take a break, osteoclastic activity is greater than osteoblastic activity.

Compartment syndrome
o Can be part of shin splints

Interosseous membrane

Muscular sprain (tib ant, tib post, peroneals)

Other random possible causes:
Fascial tears
Tibiofibular joint effusion
Bone bruise, stress f#, periostitis, periosteal contusion (Osgood Schlatter)
Compression of deep peroneal nerve
Referred pain from proximal structures
17
Q
  1. What factors are likely to have contributed to this condition?
A

Altered biomechanics of foot & ankle (eg. pronated feet)
o Tib ant eccentric contraction, has to go for longer.

High impact activity (dancing and running)

Deconditioning following rest after shoulder injury

Not wearing supportive shoes

Lack of resting (busy rehearsals)

18
Q
  1. Describe the anatomy of tibialis anterior and posterior and how each contributes to the arches of the feet. What is the role of each muscle in the gait cycle?
A

TIBIALIS ANTERIOR:
o O: Lateral tibia, interosseous membrane
o I: Medial cuneiform, base of 1st met
o Action: DF and inverts foot, supports medial arch via distal attachments
o Innervated by deep peroneal nerve (L4-5)

o Gait: Eccentric action upon heel strike & lowering of foot (PF) prevents foot-slap & decelerates foot pronation
o Also DF ankle during swing phase to clear toes from ground

TIBIALIS POSTERIOR:
o O: Posterior interosseous membrane, tibia, fibula
o I: Navicular tuberosity, cuneiform, metatarsals
o Action: PF and inverts foot; extensive distal attachments also support medial
longtitudinal arch
o Innervated by tibial nerve

o Gait: Potent supinator of foot, decelerates pronation of foot upon heel contact to early stance phase and supinates foot during mid stance to toe off

19
Q
  1. How could tenosynovitis of the muscles of the deep posterior compartment affect the posterior tibial nerve?
A

Tibial nerve:
In the popliteal fossa it gives off branches which supply popliteus, gastrocs, soleus, plantaris.

In then dives beneath the arch of soleus and travels in the deep posterior compartment of the leg, supplying tibialis posterior, flexor hallucis longus, flexor digitorum longus.

TENOSYNOVITIS:
The deep posterior compartment muscle tendons (T, D, H) run beneath the flexor retinaculum, inferior to the medial malleolus. They run with the posterior tibial artery and vein. Inflammation of the tendon sheathes within this space could cause swelling and compression of the posterior tibial nerve as it enters the plantar aspect of the foot.

FROM ANTERIOR TO POSTERIOR IN THE FLEXOR RETINACULUM:
TOM DICK AND VERY NAUGHT HARRY
Tibialis posterior, flexor digitorum longus, posterior tibial artery, posterior tibial vein, posterior tibial nerve, flexor hallucis longus.

20
Q
  1. How does foot posture and mechanics affect the action of tibialis posterior and anterior?
A

Excessive pronation puts both tib post and tib ant on stretch, so therefore there is not an optimal length/tension relationship.

With over-pronation, these muscles need to eccentrically contract longer.

21
Q
  1. What are shin splints?
A

AKA medial tibial stress syndrome

Shin splints are progressive pathology developing from tendonitis to myositis of tibialis anterior or posterior muscles. Without adequate rest, it can progress to periostitis (& possibly stress fracture)

Ssx:
o Tenderness or pain just lateral or medial to the shin bone
o With tendonitis, pain comes on with movement but gets better after ‘warm-up’
o Pain returns after activity

22
Q
  1. What is compartment syndrome?
A

The septa forming boundaries of the leg compartments are strong, so trauma to muscles may produce
haemorrhage, oedema & inflammation of these muscles. With arterial bleeding, the pressure may become high enough to compress structures in the compartment(s) concerned. Structures distal to the compressed areas may become ischemic & permanently injured (eg. loss of motor function). Anterior compartment more likely affected.

ACUTE: Involves loss of microcirculation resulting in ischemia and necrosis of muscles
o SURGICAL EMERGENCY!
o If someone comes in with red, hot, swollen compartment type syndrome with neuro ssx.

CHRONIC: More common, muscular hypertrophy and fluid stasis available compartment space, compressing on microcirculation resulting in reversible ischemic changes

Early Ssx:
o Swollen, palpably tense compartment
o Parasthesia, diminished distal pulses
o Better with rest

23
Q
  1. How can these conditions (shin splints and compartment syndrome) lead to stress fracture?
A

Stress fractures more commonly occur in tibia than fibula.
Occur when bone is under stress = bone remodelling involving osteoclastic activity with interrupted
osteoblastic lying down of new bone (by continued stress)

Stress f# most commonly occur between middle to distal 3rd of tibia on the medial side. Manifests as bone tenderness

  • Need bone scan to pick up stress f# in early stage
  • Later see radiopaque line on X-ray.
  • RX : Requires rest, modify foot biomechanics, good supportive shoes, minimized weight-bearing.
24
Q
  1. How are stress fractures differentiated from shin splints and compartment syndrome?
A

Bone scan and MRI or X-ray later

Compartment syndrome:
o Parethesia (1st ID cleft)
o Pain (Anterior tibia)
o Paresis (foot drop)
o Pallor
o Pulselessness (DP, post tib)

Stress fractures
o Night pain
o Deep localized pain

25
Q
  1. What structures or conditions could cause swelling in the upper posterior leg?
A
  • Baker’s cyst
  • Popliteal bursitis or gastroc bursitis
  • Joint effusion from acute injury (meniscal or ligamentous tear)
  • Popliteal aneurysm (pulsatile, bruit)
  • Gastroc tear, popliteal muscle spasm
  • Compartment syndrome
  • PCL injury
  • DVT
  • Abscess or infection
  • Superficial thrombophlebitis
26
Q
  1. What conditions should be considered where there is anterior swelling about the knee?
A
  • Bursitis
  • ACL rupture of injury
  • Meniscal injury
  • Fat pad inflammation
  • Fracture of patella.
  • Synovial effusion
  • Pes anserine tendonitis or bursitis
  • Osgood Schlatters patella tendinopathy
  • Quad injury