Week 11 finished Flashcards

1
Q

What is the extent of the pleural cavity?

A
  • Superior: The pleural cavities extend above rib 1 into the root of the neck superiorly. Here the pleura is reinforced by Sibson’s Fascia.
  • Inferior: The pleural cavities end at a level just above the costal margin. *At these locations it is susceptible to injury during abdominal surgery.
  • Medial: The are bordered medially by the mediastinum.
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2
Q

How is the thoracic cavity divided?

A

-Divided into 3 cavities: right and left pleural (or pulmonary) and mediastinum.

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3
Q

What is the lung pleura?

A

Each lung is invested by and enclosed in a serous pleural sac that consists of two continuous membranes: the visceral pleura, (which invests all the surfaces of the lungs forming a shiny outer surface) and the parietal pleura (which lines the pulmonary cavities).

-The pleural cavity (potential space between these two layers) contains a capillary layer of serous pleural fluid, allows the two layers to slide smoothly over each other. during respiration.

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4
Q

Anatomy of the pleura layers?

A

The visceral pleura (pulmonary pleura)- is adherent to the lung including in the horizontal and oblique fissures. It is shiny and slippery for movement against the parietal pleura.

The parietal pleura: covers the ribs, mediastinum and diaphragm; Thicker of the two pleura.

The parietal pleura is further named for the adjacent structures: mediastinal, costal, diaphragmatic, cervical pleura.

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5
Q

Nerve supply to the lung pleura layers?

A

Lungs and Visceral Pleura
o Parasympathetic – Vagus
o Sympathetic – Pulmonary Plexus – T1-6 via cardiopulmonary splanchnic
o Visceral pleura insensitive to pain

Parietal Pleura
o Costal aspect - Intercostal Nerves
o Mediastinum and Diaphragm – Phrenic Nerve
o Extremely Pain Sensitive
o Refers: thoracic or abdo wall or neck and shoulder, cervical spine (phrenic), diaphragm

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6
Q

What is pneumothorax and how does it occur?

A

The pressure in the pleural cavities (between visceral and parietal pleural layers) is subatmospheric. This helps to keep the lung distended during expiration.

  • If a hole opens up through the thorax (wound) or the lung (rupture of pulmonary lesion, rib fracture), air is sucked into the pleural cavity because of the negative pressure.
  • The surface tension adhering the pleural layers is broken and the lung will collapse (pneumothorax).
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7
Q

List the structures forming the root of the lung? and the role of each?

A

The root of the lung is a tubular collection of structures attaching the lung to the mediastinum. The hilum is the name given to the lung surface where the root is attached.

Each root contains:
• a pulmonary arteries (which carries deoxygenated blood from the right ventricle)
•two pulmonary veins (which deliver oxygenated blood to the left atrium)
•main bronchus (airway)
•bronchial vessels (arteries and veins provide nutrition to the root of the lung, support tissues and visceral pleura),
•nerves (innervate the lungs)
•lymphatics (drain lungs)

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8
Q

How many lobes does the Right and Left lung have? Name the fissures of the Right and Left Lungs.

A

The Right lung has three lobes: It has a horizontal fissure and a right oblique fissure. 3 lobes are: 1. Superior, middle and inferior lobes

The Left lung has two lobes: It has a left oblique fissure separating the 1. Superior 2. Inferior lobes.

Right lung shorter and wider due to the right dome diaphragm and the liver. However right lung overall is bigger due to the cardiac notch in the left and mediastinum is more left.

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9
Q

How can restriction at the thoracic inlet influence

lymphatic drainage from the lung?

A

The lymphatics from the lungs join the thoracic duct and the right thoracic duct meaning that all lymphatic fluid from the lungs must pass through the thoracic inlet before joining the venous system for return to the heart.

-Fascial restrictions of the thoracic inlet and pectoral fascia can cause lymphatic stasis of the upper limb.

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10
Q

What nerves supply the lungs? From what levels is the sympathetic innervation derived?

A

Posterior & Anterior Pulmonary plexus:

  • Vagus nerve (parasympathetic; Parasympathetic stimulation bronchoconstricts, vasodilates and is secretomotor)
  • T1-6 sympathetic via cardiopulmonary splanchnic nerves
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11
Q

What surface anatomy is used to identify the

extent of the normal lung fields?

A

Superior margin = Lung apices, cervical pleurae pass through the superior thoracic aperture deep to the supraclavicular fossa, and lateral to the tendons of SCM.

Anterior border= lie between 2nd and 4th costal cartilage. The anterior border than moves laterally and inferior to 6th costal cartilage.

Inferior border = midclavicular line of the 8th rib and the scapular line at the 10th rib, proceeding toward the 10th vertebra SP.

Oblique fissures= SP T2 to 6th costal cartilage anteriorly.

Horizontal fissure on right = from oblique fissure to the 4th rib and costal cartilage anteriorly.

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12
Q

Where would you place the stethoscope to

examine the lower lobes of the lung?

A

Posteriorly below rib 8– Left and Right
􀁸 Laterally- below rib 5
􀁸 Anteriorly- below rib 6
􀁸 Apex -first intercostal space
􀁸 Superior Lobes -Anterior above rib 4, posterior above rib 5
􀁸 Middle Lobe Right – Anterior midclavicular line- ribs 4-6

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13
Q

How does parasympathetic stimulation of the lung affect the bronchioles and pulmonary arteries?

A

Effect of sympathetic stimulation (fibres are post-synaptic, their cell bodies are in the paravertebral sympathetic ganglia of the sympathetic trunks. :

  • Inhibitory to the bronchial muscles= Bronchodilation
  • Motor to pulmonary vessels =Vasoconstriction
  • Inhibitory to the alveolar glands of the bronchial tree- type two secretory alveoli.

Effects of parasympathetic stimulation: (the parasympathetic fibres conveyed to the pulmonary plexus are presynaptic fibres from Vagus. They synapse with parasympathetic ganglion cells in the pulmonary plexus along the branches of the bronchial tree.

  • Motor to the smooth muscle of the bronchial tree =Bronchoconstrictor
  • Inhibitory to the pulmonary vessels = vasodilator
  • Secretory to the glands of the bronchial tree = secretomotor
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14
Q

List the structures found in the mediastinum?

A

-Heart, oesophagus, proximal bronchial tree, thyroid, thymus, trachea
-Vessels – aorta, sup and inf vena cava, brachiocephalic veins, azygous veins, thoracic duct
-Nerves – cardiac plexus, phrenic, vagus, recurrent laryngeal, symp chains – Tx splanchnic nerves that go
to cardiac plexus

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15
Q

Superior mediastinum: contents “PVT Left BATTLE”

A
Phrenic nerve
Vagus nerve
Thoracic duct
Left recurrent laryngeal nerve (not the right)
Brachiocephalic veins
Aortic arch (and its 3 branches)
Thymus
Trachea
Lymph nodes
Esophagus
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16
Q

Posterior mediastinum: contents “DATES”

A
Descending aorta
Azygos and hemiazygous veins
Thoracic duct
Esophagus
Sympathetic trunk/ganglia
17
Q

How is the mediastinum anatomically divided?

A

Superior
o Above T4/5 IVD
Inferior (anterior, middle and posterior)
o Below T4/5 IVD

  • Anterior: Anterior to heat and pericardium
  • Middle: Hear and pericardium
  • Posterior: Posterior to heart and pericardium.
18
Q

What is the somatic sensory nerve supply to the pericardium?

A

Serous pericardium:
o Phrenic Nerve – C3-C5 – primary source of sensory fibres – pain sensations conveyed by these
nerves are commonly referred to the skin (C3-C5 dermatomes) of the supraclavicular region,
same side.
o Vagus Nerve – function uncertain
o Sympathetic trunks – vasomotor (T1-4), referred as chest pain.

19
Q

Which arteries supply the heart muscle?

A

-The Right and Left Coronary Arteries off the
aortic sinuses (first part of the aorta) which give
branches to atria and ventricles

-Venous: Cardiac veins, via the coronary sinus
drains blood from the heart into the right
atrium.

20
Q

How does blood flow through the cardio-pulmonary

system?

A

Superior Vena Cava and Inferior Vena Cava, flow into the right atrium. The deoxygenated blood flows through the tricuspid valve into the right ventricle. The deoxygenated blood is then pumped up through the pulmonary valve into the pulmonary trunk. The trunk splits into left and right pulmonary artery and into the lungs where it becomes oxygenated and pumped back to the heart through the pulmonary veins to the left atrium. Once in the left atrium the blood is pumped into the right ventricle via the bicuspid valve. The blood then is pumped through the aortic valve into the ascending aorta, where it branches off to the brachiocephalic trunk, Left internal carotid and left subclavian before becoming the descending aorta

21
Q

Describe the innervation of the heart.

A

Nerve supply to the heart is derived via the cardiac plexus. Autonomic fibres from the cardiac plexus- The cardiac plexus is made up of both sympathetic and parasympathetic fibres en route to the heart , as well as visceral afferent fibres conveying reflexive and nociceptive fibres from the heart.

o It is supplied by the vagus (parasympathetic – decreases heart rate, force and constricts
coronary arteries)
o Sympathetic fibres from the sympathetic trunk of the thoracic and cervical regions (T1-4 -
increases heart rate and force).

  • Pain sensation from the heart is conducted with the
    sympathetic fibres which can refer to the cutaneous
    regions supplied by the same spinal cord levels.
22
Q

Where would you place the stethoscope to auscultate the

mitral valve?

A

Aortic – 2nd Intercostal space – Right Midclavicular Line
Pulmonic – 2nd intercostal Space – Left Midclavicular Line
Tricuspid – 3rd - 5th Intercostal Space – Left Sternal Border
Mitral - Midclavicular line in 5th Intecostal Space (apex)

23
Q

What is a pulmonary embolism? What signs and symptoms may occur? What are the risk factors for
pulmonary embolism?

A

Pulmonary embolism - Obstruction of a pulmonary artery by a blood clot (embolus) is a common cause of morbidity and mortality. An embolus in a pulmonary artery forms when a blood clot, fat globule, or air bubble travels in the blood to the lungs e.g from the leg after a compound fracture. Consequently the immediate result of a PE is partial or complete obstruction of blood flow to the lung. Therefore a section of the lung is ventilated but not perfused with blood.

SSx:
o Often non-specific, SOB, dyspnoea, chest pain, coughing, haemoptysis, arrhythmia
o Complications: Right sided heart failure (friction rub, swelling of ankle)

Risk factors:
o Old age, cancer, genetic predisposition, immobilization, pelvic or leg trauma, pregnancy and
surgery.

24
Q

What is a pulmonary embolism? What are the risk factors for pulmonary embolism?

A

Pulmonary embolism - Obstruction of a pulmonary artery by a blood clot (embolus) is a common cause of morbidity and mortality. An embolus in a pulmonary artery forms when a blood clot, fat globule, or air bubble travels in the blood to the lungs e.g from the leg after a compound fracture. Consequently the immediate result of a PE is partial or complete obstruction of blood flow to the lung. Therefore a section of the lung is ventilated but not perfused with blood.

Risk factors:
o Old age, cancer, genetic predisposition, immobilization, pelvic or leg trauma, pregnancy and
surgery.

25
Q

Define dyspnea

A

-Difficult, painful breathing or shortness of breath

26
Q

Define Orthopnea

A
  • Difficulty breathing when in any position except sitting or standing upright
27
Q

What is angina?

A

Chest pain caused by reduced flow of blood to the heart muscle (ischemia) – can be stable
(requires exertion), unstable (can occur at night) or variant (like unstable but relieved by angina meds)

28
Q

What are the clinical signs and symptoms of Angina?

A

Symptoms associated with angina include:

Chest pain or discomfort
Pain in your arms, neck, jaw, shoulder or back accompanying chest pain
Nausea
Fatigue
Shortness of breath SOB SOBE
Sweating
Dizziness

The chest pain and discomfort common with angina may be described as pressure, squeezing, fullness or pain behind the sternum. Some people with angina symptoms describe angina as feeling like a vise is squeezing their chest or feeling like a heavy weight has been placed on their chest.
For others, it may feel like indigestion. Indigestion, nausea, fatigue, sweating, light-headedness or weakness may also occur.

Often pain / discomfort occur during physical exertion but it last less than 20 minutes. Pain is usually
relieved by rest.

29
Q

What is myocardial infarct? What are SSX?

A

-Destruction of heart tissue resulting from obstructed blood supply to the heart muscle AKA heart attack.

-Central crushing chest pain, possibly referred pain (shoulder, jaw, back), SOB, toothache,
headache, nausea, vomiting, epigastric pain, sweating, heartburn, malaise

30
Q

What is heart failure?

A

Heart failure is a chronic, progressive condition in which the heart muscle is unable to pump enough blood through to meet the body’s needs for blood and oxygen. Basically, the heart can’t keep up with its workload.

31
Q

What is the difference between Right and Left Heart failure?

A

Right – Failure to pump returning blood to & through lungs, increased back pressure on the body–>
oedema (esp. ankles, central obesity), pitting oedema, hepatomegaly, dry skin on legs (due to tissue
stretch & pressure), SOB, central cyanosis, decreased ability to move, distended neck veins.

􀁸 Left – Failure to pump oxygenated blood around the body –> Peripheral cyanosis, SOBE, fatigue
chronic pulmonary congestion –> Cough with frothy sputum, ( from increased back pressure on
pulmonary circulation), dyspnoea, increase of symptoms at night (due to intrapulmonary oedema)

32
Q

What is the difference between Right and Left Heart failure?

A

RIGHT – Failure to pump returning blood to & through lungs, increased back pressure on the body–>
oedema (esp. ankles, central obesity), pitting oedema, hepatomegaly, dry skin on legs (due to tissue
stretch & pressure), SOB, central cyanosis, decreased ability to move, distended neck veins.

LEFT – Failure to pump oxygenated blood around the body –> Peripheral cyanosis, SOBE, fatigue
chronic pulmonary congestion –> Cough with frothy sputum, ( from increased back pressure on
pulmonary circulation), dyspnoea, increase of symptoms at night (due to intrapulmonary oedema)