Week 6 - ABI/TBI and balance Flashcards

1
Q

Who is more at risk for falls and why?

A

Older people are more at risk due to:
- impaired vision
- dizziness
and other de-stabilizing health problems.

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2
Q

Where are the most common areas for fractures or injuries?

A
  • 90% are hips and wrist fractures
  • 60% are head injuries
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3
Q

what condition is the most in danger when falling?

A

Osteoporosis

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4
Q

why is Osteoporosis called the silent disease?

A
  • bones may become weak with no symptoms
  • may not be aware until they have a strain, bump, or fall that causes a bone to break
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5
Q

How common is a second fall after a one resulting in head injuries?

A

1/3 of older adults end up back I’m the ER within 90 days

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6
Q

what effects does ageing have on our bodies?

A
  • Loss of muscle bulk (mass starts to deteriorate unless we stay active)
  • Loss of agility
  • losses of muscles in tib anterior
  • Spinal degenerative disc disease (limiting flexibility and may cause pain)
  • Diminished vision such as cataracts and glaucoma
  • reaction time and reflexes
  • possible chronic medical conditions
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7
Q

Cataracts vs Glaucoma

A

Cataracts
- eye condition where cloudy or opacity blocks light entry

Glaucoma
- Group of eye conditions that gradually steal sight without warning
- often without symptoms
- damage to optic nerve

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8
Q

Why might glasses not help improve vision for falls and how could you fix this?

A
  • glasses might be bifocals or trifocals
  • when they look through the bottom half, depth perception may be altered
  • making it easier to lose balance
  • to prevent, practice keeping eyes forward and head down to look
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9
Q

Altered reaction time vs reflexes

A

Altered Reaction:
- Ageing slows reaction time
- makes it harder to regain balance following a sudden shift of weight.

Altered Reflexes:
- May result in a fall
- As people age, reflexes go down

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10
Q

how might taking medication for a chronic medical condition increase the risk of falls?

A

some may also increase the risk of falls related to side effects such as:
- dizziness
- confusion
- disorientation
- slowed reflexes

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11
Q

What are the three types of falls?

A

Physiological (anticipated):
- Most in-hospital falls
- occur in patients who have risk factors for falls identified in advance, such as altered mental status, abnormal gait, frequent toileting needs, or high-risk medications.
- close supervision with attempts to address the patient’s risk factors.

Physiological (unanticipated):
- occur in a person who is otherwise at low fall risk, because of an event whose timing could not be anticipated
- seizure, stroke, or syncopal episode
- post-fall care with injury prevention strategies.

Accidental:
- low-risk people due to an environmental hazard
- Improving environmental safety will help reduce fall risk in these people and for all health providers.

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12
Q

How can we improve balance?

A
  • whole body muscle strength (lower limbs and core exs)
  • max vision correction
  • practice daily balance exercises that are challenging enough, and work on dynamic trunk control, sitting and standing, dynamic balance
  • correct abnormal posture
  • correct abnormal movement patterns
  • strengthen self efficacy in balance control –> this leads to reduced fear of falling, increase walking speed, improve physical function, and improved QOL
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13
Q

4 types of balance strategies

A
  1. ankle
  2. hip
  3. suspensory
  4. stepping
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14
Q

when can balance exercises be done?

A

every day or as many days as you like and as often as you like

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15
Q

when should older adults do balance training?

A
  • 3 or more days a week
  • strength/flex/balance exs from a standardized program demonstrated to reduce falls
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16
Q

what are the 3 things that can be used synonymously with balance?

A
  • postural control
  • postural stability
  • equilibrium
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17
Q

what is needed to have good balance?

A
  • ability to maintain one’s line of gravity within a base of support
  • maintain equilibrium - all acting forces are cancelled by each other resulting in a stable balanced system
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18
Q

how much do healthy subjects rely on somatosensory, vestibular, and vision ON FIRM SURFACE?

A
  • 70% on somatosensory
  • 20% Vestibular
  • 10% on Vision
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19
Q

how much do healthy subjects rely on somatosensory, vestibular, and vision ON UNSTBALE SURFACES?

A
  • 60% vestibular
  • 30% Vision
  • 10% somatosensory
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20
Q

how is the somatosensory system important for balance?

A
  • Proprioceptive information from spino-cerebellar pathways, processed unconsciously in the cerebellum
  • required to control postural balance.
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21
Q

what is the time delay for Proprioceptive information?

A
  • monosynaptic pathways that can process information as quickly as 40–50 ms and hence the major contributor for postural control in normal conditions.
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22
Q

What is the function of the Vestibular System?

A

Measures head rotation and head acceleration through semicircular canals and otolith organs (utricle and saccule)

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23
Q

what are the three generates compensatory responses to head motion?

A

Postural responses (Vestibulo-Spinal Reflex): keep the body upright and prevent falls when unexpectedly knocked off balance.

Ocular-motor responses (Vestibulo-Ocular Reflex): allows the eyes to remain steadily focused while the head is in motion.

Visceral responses (Vestibulo-Colic Reflex): help keep the head and neck centered, steady, and upright on the shoulders.

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24
Q

in relation to balance, what is the Visual System dependent on?

A
  • characteristics of the visual environment
  • the support surface
  • including the size of the base of support and its rigidity
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25
Q

what is the time delay for the visual system information?

A

has longer time delays as long as 150-200 ms

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26
Q

what systems can adapt to composite for visual disorders?

A
  • peripheral
  • vestibular
  • somatosensory perception
  • cerebellar processing

help compensate for their visual information deficit and to provide good postural control.

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27
Q

what is internal and external Pertubation-based Balance Therapy (PBT)?

A

Internal: Patient perturbs their own balance

External: Other forces are applied to a patient to perturn their balance

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28
Q

what is Treadmill-based PBT?

A
  • applied by therapists are the most feasible forms of PBT.
  • constructs a safe environment with a harness and handrails, and is highly task-specific for fall prevention (eg, provoking slips or trips through unexpected accelerations)
  • Improves control of step reactions
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29
Q

how frequent is Treadmill-based PBT done?

A

2-3 training sessions a week

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30
Q

What is a Acquired Brain Injury (ABI)?

A
  • Damage to the brain, which occurs after birth
    and is not related to a congenital or a
    degenerative disease.
  • may be temporary or permanent
  • cause partial, functional disability or psychosocial
    maladjustment
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31
Q

what are two ways a Acquired Brain Injury (ABI) can happen?

A

Traumatic Brain Injury - TBI (e.g. car accident or fall)

Non-traumatic (e.g. Tumor or stroke)

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32
Q

what deficits can a severe injury cause?

A
  • thought
  • behaviour
  • motivation
  • personal traits
  • talents
  • movement
  • cognition
  • social skills
33
Q

What are the mechanisms of injury?

A
  • We have to know that brain tissue is contained within boney skull

-Any head movement that suddenly halted or any direct blow produces displacement and distortion of tissue

34
Q

What can determine an injury?

A
  • Nature
  • Direction and magnitude of forces determine degree of primary and secondary damage
35
Q

Where are the two sites where an ABI can occur?

A

Coup injury

Contrecoup injury

36
Q

What are the types of focal - local injury

A

Cerebral contusions (of gray matter)

Lacerations

Hematomas

37
Q

What is a cerebral contusion?

A

Damage under point of impact

Results in underlying hemorrhage

Mild → severe

38
Q

What is a laceration?

A

Internal bony irregularities of skull abrade brain surface

39
Q

What is a hematoma?

A
  • Tearing of vascular structures causes swelling or mass of blood

-Occupy space and compress brain tissue; in severe cases can cause additional brain damage, shift or herniation

40
Q

What are the 3 types of a hematoma?

A
  • Epidural → blood collects between skull and dura mater (outer layer of meninges that protect the brain)
  • Subdural → blood pools between dura mater and arachnoid mater (middle layer of meninges)

-Intracerebral → blood accumulates within the brain tissue itself

41
Q

What is a cerebral herniation?

A

Brain tissue, blood, and csf shift from normal position inside the skull

Serious medical emergency, and fatal

42
Q

How does a cerebral herniation present?

A
  • Decreased level of consciousness → leads to coma
  • Progressive motor dysfunction
  • Vegetative disturbances
  • Abnormal posturing
43
Q

What is a diffuse brain injury?

A
  • Rotational and shaking forces on brain
  • Widely scattered shearing of axons within myelin sheaths

-This results in severe widespread degeneration of white matter, microscopic structural disruption

44
Q

Clinical picture in a DAI?

A

Coma

Abnormal extensor posturing of limbs

Autonomic dysfunction

45
Q

What is a coma in a DAI?

A

Different from sleep as person can not wake up

Not the same as brain death

Person is alive, but can not respond in a normal way to environment

Can persist in vegetative state for weeks → months

If regains consciousness, widespread functional impairment: attention, endurance, coordination, speed, judgement, insight

46
Q

What is pathological posturing?

A

Sign of severe damage to brain

Rigidity is present

47
Q

How in response can pathological posturing occur to noxious stimuli from external / internal sources?

A

Abscess
Hematoma
Hydrocephalus
Raises intracranial pressure
DAI
Tumor
Hemorrhage
Encephalitis
Lead poisoning
Meningitis

48
Q

Abnormal posturing

A

Decorticate
Decerebrate

49
Q

What is decorticate?

A

Rigid with bent arms turned in toward the body, clenched fists held on to chest, and legs are held straight in extension

50
Q

What is decerebrate posturing?

A

Rigid with arms and legs held in extension, toes are pointed downward and head and neck are arched

51
Q

What is a coma emergence?

A

Gradual process of regaining consciousness after a coma

52
Q

What would pt experience in a coma emergence?

A

Significant agitation, anxiousness and aggression during time after coming out of coma

This would be due to pt inability to process the immediate sensory information with their environment

Pt may overreact in presence of relatively minor requests or task

Pt may demonstrate periods of tactile defensive behaviour → minimize manual cues

53
Q

How should the treatment sessions be after coma emergence?

A

Quiet dark room

54
Q

Coma emergence treatment?

A

Pt progressively mobilized through use of tilt table / standing frame activities

Pt may begin on sitting schedule to gradually increase sitting time

Vital signs monitored for orthostatic changes and adverse physiological responses to positional changes → fluctuations in bp and diaphoresis (excessive sweating)

Requires 2pA initially for transfer, positioning, and standing activities

55
Q

Things to keep in mind post craniotomy?

A

Require helmet use when out of bed

When mobilized without helmet, care should be taken not to put excessive pressure over affected area

56
Q

What are the secondary brain damage, results from initial injury?

A
  1. Raised intracranial pressure
  2. Arterial hypoxia and brain ischemia
  3. Cerebral edema
  4. Arterial hypotension
  5. Impaired salt and water balance
  6. Intracranial infection
  7. Hydrocephalus
57
Q

What do clinicians use as recovery scales for ABI?

A

Glasgow coma scale

Ranchos los amigos scale

58
Q

How many levels for Ranchos Los Amigos scale?

59
Q

What is the Glasgow coma scale?

A

Eye Opening response

Verbal response

Motor response

60
Q

What are the factors in an ABI recovery

A

Age
Size of lesion
Extent of diffuse injury
Premorbid skills, intelligence, behavior
Generic inheritance
Neural plasticity
Nutritional history
Environment
Early medical management, rehab, fam involvement
Availability of support services

61
Q

ABI management

A

-Team approach is key
-Medical management: primary injuries, traumatic: address other injuries
-Early treatment: positioning, skin care, rom, pulmonary hygiene

62
Q

What are the clinical consequences of a diffuse ABI?

A

Coma, persistent vegetative state

Confusion and cognitive stages

Mixed sensory and motor deficits

63
Q

What are the clinical consequences of a local ABI?

A

Frontal lobe: confusion, impaired judgement, insight, safety; behavioural problems

Other brain areas: mixed sensory, motor and language deficits

64
Q

What are the treatment considerations of a diffuse ABI?

A

Treatment and activity should be related to cognitive status

Emphasize movement, functional activities; deemphasize component deficits

65
Q

What are the treatment considerations of a local ABI?

A

Behavioural focus, safety training

Sensory integration, compensation, function

66
Q

What do PT like to focus on for TBI rehab and why?

A

Vestibular rehab

Why? → address pt dizziness and balance problems

67
Q

What are some management considerations that PTs should keep in mind when treating?

A

Behav management
Motor learning
Motivation
Attention
Memory
Motor control
Family education

68
Q

What are some of cognitive- behavioral issues and tips?

A

Impulsivity:
-Stay aware at all times
-Give patients 1 command at a time

Decreased initiative:
-Give patients choices

Emotional lability:
-Reassure patients this is not unusual; carry on

Decreased memory
-Use memory aids; ++repetition

Decreased attention
-Use memory aids; ++repetition

Decreased problem solving
-Provide choices, cues and prompting

Decreased abstract thinking
-Keep interactions concrete

Cognitive - communication issues
-Provide feedback; encourage turn taking

Agitation
-Recognize triggers, sign
-Use voice, distractions to de-escalate situations

69
Q

Integration of cognitive and neuromuscular interventions

A

Cognitive impairments may be substantial

Sleep disorders may be interfered with treatment

Give time for problem solving; process slowly, needs more time to perform task

Diminished attention span → need ongoing redirection to the task

Appropriate time and cue to make the skill easier

Poor judgment can create a dangerous situation if left unsupervised

Gait interventions; pt performed necessary speed changes needed during an emergency situation like a fire drill

Reinforce safety strategies during sessions, turning on light when going to bathroom at night, hanging on to railing on stairs, brakes on wheelchair

70
Q

The agitated patient

A

Be aware of the sensory experiences of pt

Pt biome anxious and aggressive when they can not process immediate sensory info with the environment

Pt may overreact in presence of minor requests or tasks

71
Q

Treatment of agitated pt

A

Assess environment

Strategize deliver proper sensory experiences

Sessions to prevent sensory overload

Quiet areas are good to reduce distractions

Reducing volume of the voice is more calming

Multiple shorter sessions are more preferred

Time outs can be implemented when undesired behaviour is unable to be redirected

Be careful with bernal and manual cues during mobility

72
Q

What communication disorders would you see in ABI issues and tips?

A

Aphasia: wernicke’s or broca’s
- for wernicke’s: gestures and write it out
- for broca’s simple, clear sentences

Apraxia: Hard to coordinate movement of speech muscles. Hard to get message out. Often accompanies aphasia
- Encourage breath control, pauses
- Ensure understanding ; clarifying words

Dysarthria: Speech muscles are damaged, paralyzed or weakened. Often accompanies aphasia
- Encourage breath control, pauses
- Ensure understanding ; clarifying words

Dysphasia: Partial loss of ability to produce and understand spoken language
- Use tools / diagrams ; pen / paper, dichotomous choices ;gestures
- Liaise with SLP and follow their recommendations
- Supported communication techniques

Paraphasia: Production of unintended syllables, words or phrases during the effort to speak
- Encourage breath control, pauses
- Ensure understanding ; clarifying words

Dysphagia: Difficulty swallowing and / or painful swallowing
- Encourage patient to follow recommendations from SLP and dietician

73
Q

Who is the first and second in line for a risk of a TBI and why?

A

first: Young adults → Motor vehicle collision

second: Elderly 65+ → Ground level fall

74
Q

How are younger people different than elder in a hospital setting when recovering?

A

Elder:

They recover slowly and more likely to be hospitalized

They also exhibit worse outcomes

75
Q

What is known as a mild tbi?

A

concussion

76
Q

What is the cause of a concussion?

A

Traumatic biomechanical force with or without a loss of consciousness

77
Q

What are the 5 stages in the return to play post concussion and the objectives?

A
  1. No activity → complete cognitive and physical rest → brain recovery
  2. Light aerobic conditioning → walking and swimming → add movement
  3. Short specific exercise → running sprint in soccer or lacrosse → add movement
  4. Non contact training drills → passing in football or lacrosse → multitask intensity
  5. Full contact practice → normal training activities with medical clearance → return to sport specific intensity
78
Q

What are the long term rehab goals in a severe ABI?

A

Range of short to long term limitations in physical and neuropsychological abilities

Rehab leads the severe ABI survivors to a better QOL from physical, functional, social and emotional POV that increases possibilities and options for the patients future

Holistic multidisciplinary approach is recommended with aim of not only functional recovery also the social reintegration of ABI survivors

79
Q

what is resilience in pt with tbi?

A

may be a measure of “stress coping ability”

people with tbi have less resilience than the general population

resilience requires skills and characteristic like patience, tolerance, fatih, etc