WEEK 5 - post-procedural wound assessment and management Flashcards

1
Q

What are standard precautions?

A

prevents microorganisms on hands, surfaces, equipment

  • hand hygiene
  • ppe
  • gloves
  • goggles
  • masks
  • disposable equipment
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2
Q

What are platelets?

A

small, colourless cell fragments in our blood that form clots and prevent bleeding.

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3
Q

What are neutrophils?

A

a type of white blood cell (leukocytes) that act as your immune system’s first line of defence.

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4
Q

What are cytokines?

A

small proteins that are crucial in controlling the growth and activity of other immune system cells and blood cells.

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5
Q

What are enzymes?

A

proteins that help speed up metabolism, or the chemical reactions in our bodies.

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6
Q

What are fibroblasts?

A

a type of cell that contributes to the formation of connective tissue.

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7
Q

What is the cell matrix?

A

dynamic gel in the cytoplasm of the cell.

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8
Q

What is collagen?

A

main structural protein in skin/connective tissues.

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9
Q

what is actin?

A

cellular protein found especially in actin/microfilaments and active in muscular contraction, cellular movement, maintenance of cell shape.

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10
Q

What is anaemia?

A

condition; lack enough healthy red blood cells to carry adequate oxygen to body tissues; tired and weak.

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11
Q

What is sepsis?

A

life threatening organ dysfunction caused by a person’s impaired immune or metabolic response to an infection, anywhere in the body.

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12
Q

What is dehiscence?

A

the partial or complete separation of the wound edges and the layers below the skin.

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13
Q

What is granulation?

A

the wound is progressing from the inflammatory phase of healing to the proliferative phase of healing. Pink in colour, an indication of healing.

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14
Q

What is evisceration?

A

when the person’s viscera protrude through the disrupted wound.

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15
Q

What is necrotic tissue?
Eschar
Slough

A

dead cells in wound (should be removed to allow new tissues to form and cover wound bed)

eschar (dry, thick, leathery tissue that is often tan, brown or black)

slough (yellow, tan, green or brown in colour and may be moist, loose and stringy in appearance).

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16
Q

3 types of healing + descriptions:

A

Primary healing

  • Clean wound – no infection
  • +- sutured
  • Minimal disruption to basement membrane

Secondary healing

  • Needs to remain open for an extended period
  • Extensive loss of tissue
  • Increased infection risk – longer for granulation to fill deficit

Tertiary healing

  • Loss of tissue
  • Required to remain open for observation
  • Possibility of primary closure at a later stage
17
Q

What are the 4 stages of the healing process?

A
  1. Haemostasis (-burns)
  2. Inflammatory phase
  3. Proliferative phase
  4. Remodelling phase
18
Q

THE HEALING PROCESS: HAEMOSTASIS (3pts)

A
  • Initiates smooth muscle contraction of vessels to halt the bleeding.
    Clotting causes platelets to go to the wound.
  • Tissues become hypoxic and acidic causing vasodilation.
19
Q

THE HEALING PROCESS: INFLAMMATORY PHASE (4pts)

A
  • Body recognises an injury (not necessarily when an infection is present).
  • Neutrophils attracted from the release of cytokines and enzymes.
  • Neutrophils remove foreign materials, pathogens and any damaged tissue through phagocytosis.
  • Apoptosis, which is programmed cell death, or appear as slough.
20
Q

THE HEALING PROCESS: PROLIFERATIVE PHASE (4pts)

A
  • Fibroblasts ingrate to form a matrix.
  • Enable collagen and actin into bundles.
  • The intracellular matrix is the foundation of wound repair.
  • Angiogenesis occurs where new blood vessels occur across wound.

(Wounds do not have blood supply in its centre, relies on viable tissue at edges of wound – they perfuse the area and little capillaries sprout out into the wound).

21
Q

THE HEALING PROCESS: REMODELLING PHASE (2pts)

A
  • Development of epithelium and scar formation.

- Blood vessels in scar areas die off and collagen returns to normal (not as strong as original collagen matrix).

22
Q

Local vs systemic factors for wound healing:

A

Local factors
o Position of wound?
o Dehiscence risk or wound infection?

Systemic factors
o Consider increasing age, malnutrition/nutritional deficiencies; smoking, obesity; immune disorders; stress; anaemia; immobility; shock

23
Q

TIMERS

A

T – tissue is it deficient or non-viable (not capable of living, growing, developing, or functioning successfully)

I – infection or inflammation

M – moisture imbalance

E – edge of wound: is it undermined or non-advancing

R – repair of tissue and regeneration

S – social factors: including patient’s knowledge, language, mental health, living conditions

24
Q

HOPE

A

o Heat – large amount of exudate/discharge
o Oedema
o Pain
o Erythema

25
Q

HOPE

A

o Heat – large amount of exudate/discharge
o Oedema
o Pain
o Erythema

26
Q

To heal a wound must have:

A

o Cellular replication
o Supporting matrix
o Foreign/damaged material elimination

27
Q

Conditions that may affect wound healing:

A

o Cardiovascular and reparatory failure – systemic
o Peripheral vascular disease – regional
o Poor angiogenesis in the wound – locally (formation of blood vessels)

28
Q

LAYERS OF THE SKIN: EPIDERMIS

A

o Epidermis:

outer sheet of dead cells called stratum corneum
+
basal layer - deeper level of rapidly dividing cells (basal layer cells rise to replace stratum corneum cells following injury/scrapes – initial barrier to invading foreign substances)

29
Q

LAYERS OF THE SKIN: DERMIS

A

o Dermis:

made of dense connective tissue, sensory receptors embedded inside

(e.g., thermos, Meissner’s corpuscles are sensitive to touch, free nerve endings that detect pain + tissue damage), sweat glands, hair follicles, blood vessels