WEEK 5 Flashcards
peptic ulcer disease : what is it ?
a condition characterized by erosion of the gastric or duodenal mucosa
the mucosal barrier is broken and HCL enters the tissues causing injury to the tissues
why does PUD happen ?
can be precipitated by what ?
drugs
stress
bacteria ( H. pylori )
just read : gastric ulcer vs duodenal
Gastric
* Peak at age 50-70 years
* Pain can be aggravated by food
* May be malnourished
* N or decreased gastric secretion
* Pain 30-60 min after a meal
* Heals with tx -if it recurs it is in the same location
Duodenal
* Peak at age 20-50 years.
* Pain relief with antacid and food. * Usually well nourished.
* Increased Gastric secretions
* Pain 1 1⁄2-3 hrs after meal
* Often has remissions and
exacerbations
yes
recognize cues : assessment diagnostic tests
explain it
pt hx
lab assesments
esophagogastroduodenoscopy
nuclear medicine scan
what are the complications of PUD
explain it
hemorrhage/upper gi bleed
perforation
gastric outlet obstruction
GI bleed
can be apply to a patient that comes in for a GI bleed that perhaps is not as a result of an ulcer as well. so you can apply what you learn here to any patient that comes in with a GI bleed
yes
what are the recognize cues: assessment
if the blood has not hit the stomach acid yet, then it is bright red
and vomiting lots of bright red blood
what undergoes it
- Hematemesis/ coffee ground emesis
- Tarry black stool (bleed from higher up/longer duration) - higher up intestinal bleed
- Abdominal pain
- Can proceed to shock
take action : stabilize patient ( upper GI bleed )
- Treat like for hypovolemic shock
- Calm approach, frequent VS, O2,
- IV fluids +++ (monitor for fld overload)
- I&O, monitor urine output
- Monitor stools/emesis
- Monitor lab work
upper GI bleed : take action : stop the bleed
explain each and everything
endoscopic therapy ( primary treatment procedure )
surgery
drug therapy
antacids, PPIs, h2 receptor blockers
helpful decreases the irritants and help that bleed stop
what is octreotide ( sandostatin )
used to decrease blood flow to the abdominal organs it causes phases
constriction of vessels and its given IV over five to six days after the initial bleed
and helps with that bleed helps that bleed to stop
true or false. vasopressin , causes vasoontriction but vasopressin we have to watch carefully because it can also contrict all other vessels
so it needs to be given with caution and that pt needs to be monitored
true
perforation is the spillage of gastric contents into peritoneal cavity
this is where stomach mucosa erose away until theres a whole in the stomach
it spill into the peritoneal cavity
recognize cues : what undergoes perforation
Sudden severe abdominal pain
* rigid board like abdomen
* pain
* increasing distention
what undergoes take action for perforation
- Monitor for hypovolemic and /or septic shock and treat
- Maintain NG for gastric decompression
- Antibiotic therapy
- Prepare for surgery
what is the contraindication if mucosa is perorated , what should the nurse not do ?
is put anything down that ng tube
because whatever goes down that stomach is just going to spill out into the peritoneal cavity
just read : complication of pud : gastric outlet obstruction
Gastric Outlet Obstruction
* Narrowing of pylorus from…
Recognize Cues
* Pain that progresses and becomes
* Swelling of upper abdomen
* Projectile vomiting
* undigested particles from hours or days ago
Take Action
* Decompress with NG tube
* IV fluid and electrolyte replacement
* Surgery to open obstruction and remove scar tissue
trueeee
PUD pharmacological therapy
HAPCA
h2 receptors
antacids
PPIs
cytoprotective
antibiotics for H pylori
just read for the discharge planning for PUD :
Discharge Teaching of PUD: Conservative Therapy
* Dietary Modifications
* Avoid spicy foods, acidic foods, caffeine, alcohol
* Stop Smoking
* Avoid OTC Meds
* Take all meds as prescribed
* Report bloody emesis, tarry stools, increased epigastric pain
* Encourage patient to share concerns about following lifestyle changes
explain what pud surgical procedures are
billroth I
billroth II
vagotomy
pyloroplasty
- Billroth I: Gastroduodenostomy
- Partial gastrectomy with removal of distal
2/3 stomach and anastomosis of gastric stump to duodenum. Antrum and pylorus removed. - Billroth II: Gastrojejunostomy
- Partial gastrectomy with removal of distal 2/3
stomach and anastomosis of gastric stump to Jejunum. Antrum and pylorus removed. - Vagotomy
- Severing of the vagus nerve
- Eliminates the stimulus to secrete HCL
- Pyloroplasty
- Surgical enlargement of the pyloric sphincte
Take Action:Post-op care after surgery for PUD
just read :
* Will have NG to decompress stomach
* Monitor bowel sounds
* Monitor N/G content (color and amounts)
* Ensure patency of NG tube
* Remove NG tube when peristalsis returns
* IV fluids with K+ and vitamin replacement
* Introduce foods when ordered
* Care of abdominal incision
* Encourage DB&C with splinting
* Encourage ambulation to increase peristalsis
explain ensure patency of ng tube
may need saline irregations every 4 hours
if the tube plugs and the stomach becomes distended this could cause rupture of the suture lines and leakage of the gastric contents of peritoneal cavity
why is there pernicious anemia ?
when we remove so much of the stomach and the bowel were losing a lot of parietal cells
where the intrinsic factor is produced , we need that intrinsic factor b12 and b12 is needed to synthesize hemoglobin needed to synthesize hemoglobin so this patients ends up with pernicious anemia
true or false. putting up pillow against their stomach while they’re doing their deep breathing and coughing really helps them when it comes down for surgery pud
true
post op complications for billroth I and billroth II
dumping syndrome
post prandial hypoglycemia
bile reflux gastritis
what is the variant of dumping syndrome ?
postprandial hypoglycemia
what is this describing
; Hyperglycemia releases insulin resulting in secondary hypoglycemia.
post prandial hypoglycemia
gastric cancer :
recognize cues
- Anemia
- Vague epigastric fullness
- feelings of early satiety after meal
- weight loss, dysphagia, dyspepsia
true or false. * During an endoscopic ultrasound can be performed to evaluate
depth of tumor and presence of lymph nodes
true
true or false. If in early stages laparoscopic surgery may be all that is needed however in late stages a total gastrectomy may be required.
true
Total Gastrectomy with Esophagojejunostomy (removal of stomach with resection of esophagus to jejunum)
- will have N/G (drainage minimal)
- will have chest tubes because enter through chest wall.
- Clear fluids initiated after several days to solid foods
- Radiation/ chemotherapy to adjunct or if surgery is not an option
- At risk for poor nutritional status, wt loss, vitamin
deficiency, pernicious anemia, dumping syndrome, postprandial hypoglycemia : is this true or false when it comes down to surgical management of gastric cancer
true
what are the two autoimmune disorders of GI tract
ulcerative collitis
crohn’s
recognize cues : ulcerative collitis
- Peaks between the ages of 15 & 25
- Bloody diarrhea
- Abdominal pain
- Located distally in the rectum and spreads proximally in a continuous fashion of the colon
- Cured with Surgery
recognize cues : crohn’s disease
- Peaks between the ages of 15 & 30
- Non-bloody diarrhea
- Abdominal pain
- Can occur any where in the colon from mouth to anus (most often in the ilium)
true or false. ulcerative collitis, gone for good in the bowel when it comes to surgery
true
crohn’s disease, reoccurrence after surgery
more lessions may form multiple surgeries ( less and less bowel to work with )
Recognize Cues: Intra-intestinal complications of IBD
ulcerative collitis
hemorrhage
perforation
colonic dilation
colorectal cancer
this is an extensive dilation and paralysis of the colon
toxic megacolon
true or false. If they have UC for greater than 10 years, the pt is at risk
true
often associated with the deep longitudinal ulcers
that penetrate in this inflamed mucosa and it looks like kind a cobble stone appearance
this is crohn’s disease
what undergoes intra intestinal complications of IBD: recognize cues
- Stricture, obstruction
Perforation, intra-abdominal abscess
Malabsorption ,
Fistulas (Cardinal feature)
- Stricture, obstruction
- May communicate with loops
of bowel, vagina, urinary
tract causing feces in urine
and UT
what are the extra intestinal complications of ulcerative colitis and crohn’s
anthroplasty
arthritis
ocular manifestations
ostepenia./osteoporosis
skin manifestations
thromboembolic events
take action for acute phase : uc and crohns
**Frequency, amount and color of stools most important to monitor because the severity of diarrhea
determines how much fluid replacement is necessary **
what should be utilized ?
nutrition and fluid an electrolyte balance
daily wts
meticulous perianal care
what should u monitor during acute phase of uc and crohn’s for taking action
dehydration
fatigue
skin breakdown
ineffective coping strategies
intra/extra intestinal complications
blood in stools
take action for maintenance phase ( uc and crohn’s)
chronic/long term disease … remissions and exacerbations
what are the maintenance drugs
C
I
I
V
A
- ***5-ASA-Sulphasalazine–longterm(worksbestinlargeintestinetodecreaseinflammation)
- Corticosteroid Drugs -prednisone
- Immunosuppressant’s–cyclosporine(Neoral)
- Immunomodulators-infliximab(Remicade)
- Vitamins- oral iron (ferrous gluconate), IV iron (iron dextran)
- Antidiarrheal-diphenoxylate (Lomotil)