Week 4 TS and TR Flashcards

1
Q

What 2 TV leaflets do you see in each of the following views:
LAX - RVIT
SAX
SC SAX
4ch
SC 4ch

A

RVIT - Ant and Post

all the other views - Ant and Sep

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2
Q

What are the names of the TV leaflets?

A

Ant
Post
Septal

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3
Q

TS hemodynamics

A

TS = incr RA press

RA dilates

Decr venous return to heart
= systemic venous congestion (jugular venous distension, peripheral edema, ascites)

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4
Q

TS signs and symptoms

A

Fatigue bc low CO
Abd discomfort bc venous congestion
Arrythmias bc large RA

if MS too (rheumatic), then dyspnea

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5
Q

TS etiology

A

Rheumatic fever
Carcinoid TV disease
Cor Triatriatum Dexter
Mass/endocarditis
Congenital (rare)

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6
Q

How does rheumatic fever cause TS?

A

Streptococcus infection
untreated strep throat becomes rheumatic fever

years later autoimmune system response causes valve disease

thickened (bright), stiff leaflets

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7
Q

How do carcinoid tumors cause TS?

A

Tumor in GI tract (carcinoid tumor)

Metastasizes in liver

Metastases produces substances that harm heart (deposit fibrous tissue on endocardium)
= thickened leaflets that don’t move
= carcinoid TV disease

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8
Q

What is Cor Triatiatum Dexter

A

Cor Triatriatum = thin membrane
Dexter = Right side

membrane across RA that mimis TS

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9
Q

Role of Sonography in TS

A
  1. Determine etiology - rheumatic?
  2. Assess RA and RV size
  3. Estimate severity of TS
    - CW of TV inflow; trace to get mean gradient (can also measure PHT)
    - continuity eqn (not routine in BC)
  4. Assess for associated findings
    - TR
    - IVC dil
    - AoV and MV involved if rheumatic (pulm press?)
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10
Q

What is the one extra step in an echo if you have patient with TS? What do you measure?

A

CW through TV inflow
- trace to get mean gradient
- line to get PHT (not routine in BC)

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11
Q

Continuity eqn for TV is not routine in BC….if you have a patient with TS and you wanted to do the continuity eqn, how would you do it?

A

SV(TV) = SV (LVOT)

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12
Q

What are the associate findings for TS?

A

TR

IVC dil (bc high RA press)

if rheumatic… AoV and MV involved
if MV involved… high pulm press

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13
Q

What are the treatment options for TS?

A

Meds - symptoms

Percutaneous - balloon dilatation

Surgical - valve replacement

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14
Q

Organic vs. functional cause of valve disease

A

Organic = primary = smt wrong with valve

Functional = secondary = smt wrong with heart (dilated)

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15
Q

What are some functional causes of TR?

A

chronic A-fib
ASD
Dilated cardiomyopathy
Pulm HT
RV infarction

**these all cause RA/RV dilation and therefore cause TR

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16
Q

What are some organic causes of TR?

A

Carcinoid TV disease
Endocarditis (vegetation)
Ebstein’s Anomaly
Atrioventricular Septal Defect
Rheumatic fever
RV infarct / trauma (eg. ruptured pap)
Iatrogenic

17
Q

Ebstein’s Anomaly

A

one or both of TV leaflets insert more inferiorly than normal

18
Q

Atrioventricular septal defect

A

instead of the MV and TV, there is one valve with 5 leaflets that crosses the entire heart

also have ASD and VSD

19
Q

Iatrogenic

A

Caused by medical treatment (doctor caused it)

eg. pacemaker wire usually causes TR

20
Q

Hemodynamic consequences of TR

A

significant TR = incr blood in RA
= RA dilates and normal RAP (no symptoms)

Over time…
Decr RA compliance and incr RAP

Incr RA press and venous press
Diastolic vol overload = Incr RV vol and press

Right heart failure

21
Q

Symptoms of severe TR?

A

**blood / fluid stuck in body

pulsatile, tender liver
peripheral edema
ascites
cyanosis
SOB
fatigue

22
Q

Role of echo in TR

A

Assess the amount of TR

Visual - colour: mild, mod, severe TR?
*only thing we do in BC

you can also look at:
- Regurgitant area
- Jet contour/intensity from CW trace
- Hepatic vein flow
- Tricuspid inflow vel

23
Q

How would you determine the regurgitant area for TR?

A

Colour on
take cine of largest TR jet
freeze and trace entire TR jet

24
Q

If you did a CW of TR…. what would you expect the trace to look like for mild TR vs severe TR

A

mild TR - parabolic shape and less bright (dense)

severe TR
- more triangular shape with early peaking
bc high RA press but PG equalizes fast
- brighter (more dense)
bc more RBCs

25
Q

Hepatic vein flow

how to you measure it?
normal vs significant TR?

A

SC - PW at hepatic vein

Normal = S and D below the baseline and A above baseline
*opposite to pulm vein (bc of angle of doppler)

significant TR = S wave reversal

26
Q

If severe TR, how would tricuspid inflow change?

A

Increase peak E velocity

27
Q

Treatment for TR

A

Meds

Percutaneous
- valve replacement
- valve repair (new): “tampon” on a wire act as plug

Surgical
- prosthetic or mechanical valve replacement