WEEK 4 Soft Tissue Disorders & Osteochondroses Flashcards

1
Q

What is the term for stretching & tearing of musculotendinous unit?

A

strain

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2
Q

Which tendon injury reflects a more chronic condition w/ minimal or no inflammatory process detected histologically?

A

tendinosis or tendinopathy

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3
Q

Changes w/ tendinosis or tendinopathy at the cellular level involve expansion of local cells & thinner _________ fibrils.

A

collagen

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4
Q

What is the term for bruising w/ intact skin?

A

muscle contusion

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5
Q

Myofascial compartment syndrome is when there’s increased ________ pressure within closed compartment –> compromising nerves, muscles, vessels.

A

interstitial

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6
Q

Where does myofascial compartment syndrome usually occur?

A
  • envelopes of lower leg
  • forearm
  • thigh
  • foot
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7
Q

What is the earliest clinical symptom of impending acute compartment ischemia?

A

disproportionate deep, throbbing pain

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8
Q

In severe compartment syndromes, which objective signs are visible?

A

swollen extremity w/ smooth, shiny, or red skin

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9
Q

What is the standard intervention of severe compartment syndromes?

A

surgical decompression

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10
Q

During adolescent growth spurts, the cartilage cells of ________ become more active & prone to injury.

A

physis

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11
Q

Where does osteochondritis dissecans happen?

A

articular surface

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12
Q

Where does osgood-schlatter disease happen?

A

apophysis

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13
Q

What is the term for partial disruption of anatomic relationship within a joint?

A

sublaxation

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14
Q

What joints are most at risk for sublaxation?

A
  • GH
  • AC
  • SI
  • AA
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15
Q

What joint does dislocation occur most often at?

A

GH

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16
Q

Where are congenital dislocations most freq seen at?

A

hip

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17
Q

Movement during the first ___-___ days should be done w/ care to avoid stretching injured muscle.

A

3-7

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18
Q

B/t ___-___ days, gradually progress to using injured muscle more actively.

A

7-10

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19
Q

What type of training should be done first? What should it progress to? What’s last?

A

isometric –> isotonic –> isokinetic

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20
Q

_________ contraction is advocated for chronic tendinopathies.

A

eccentric

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21
Q

What is the term for bone formation in nonosseous tissues (usually muscles & other soft tissue areas) after trauma?

A

heterotopic ossification

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22
Q

Where does myositis ossificans occur?

A

bruised, damaged, or inflamed muscle

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23
Q

What are the 2 most common presenting symptoms of HO?

A
  • muscle pain
  • loss of motion
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24
Q

What type of end-feel is seen in individuals 3-6 months in w/ HO?

A

rigid or abrupt w/ pain

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25
Q

What sites are most affected by HO?

A
  • hip
  • elbow
  • knee
  • shoulder
  • TMJ
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26
Q

What is the term for at least 2 connective tissue diseases @ same time or in diff time frames?

A

mixed connective tissue disease or overlap connective tissue disease

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27
Q

OCTD frequently includes overlapping features of which diseases?

A
  • SLE
  • scleroderma
  • polymyositis
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28
Q

Who is predominantly affected by connective tissue disease?

A

adult women

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29
Q

What type of connective tissue disease has clinical & serologic characteristics of multiple autoimmune diseases?

A

undifferentiated

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30
Q

Which disease is present in 75% of cases of OCTD?

A

RA

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31
Q

A _________ sensory neuropathy appears to occur much more freq in MCTD/OCTB than in other rheumatic diseases.

A

trigeminal

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32
Q

What is the disorder marked by diffuse pain & stiffness in multiple muscle groups that primarily affects the shoulder & pelvic girdle?

A

polymyalgia rheumatica

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33
Q

The initial symptoms of PMR are subtle but a significant # of ppl w/ it also develop giant cell ________.

A

arteritis

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34
Q

At what age do most cases of PMR occur? Which gender?

A

over 70, women

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35
Q

PMR painful stiffness lasts more than ___ hour(s) in morning on arising.

36
Q

The symptoms of PMR are often bilateral & (symmetric/asymmetric) affecting the neck, SC jt, shoulders, hip, low back, & buttocks.

37
Q

What is the rapid breakdown of skeletal mm tissue bc of mech, physical, or chem traumatic injury?

A

rhabdomyolysis

38
Q

Injury in rhabdomyolysis results in release of _________ _________ enzymes, myoglobin, & other by-products into blood –> renal failure.

A

creatine phosphokinase

39
Q

What is the change in urine color associated w/ rhabdomyolysis?

A

tea or cola colored

40
Q

Injured muscle in rhabdomyolysis leaks _________ –> disruptions in heart rhythm.

41
Q

What is the term for nonspecific muscle weakness secondary to identifiable disease or condition? What about if it’s inflammatory?

A
  • myopathy
  • myositis
42
Q

What is the term for nonnecrotizing myopathy accompanied by fiber atrophy, fatty degeneration of mm fibers, & fibrosis that prolongs ICU stays?

A

critical illness myopathy

43
Q

What are the 4 types of acquired myopathies?

A
  • inflammatory
  • endocrine
  • associated w/ systemic illness
  • drug-induced or toxic
44
Q

Myopathy has more (distal/proximal) muscle weakness while myositis has more (distal/proximal).

A
  • pathy: proximal
  • sitis: distal
45
Q

________-induced myopathy –> respiratory myopathy.

46
Q

What is the term for hyperirritable spots in a taut band of skeletal muscle that is painful on compression, stretch, overload, or contraction of tissue which usually responds w/ referred pain distal?

A

trigger points

47
Q

TrPs are _________ when they cause spontaneous local & referred pain & __________ when they cause pain only on stimulation.

A

active & latent

48
Q

Both active & latent TrPs can cause allodynia & primary & secondary hyperalgesia which implies that __________ fibers from TrP nociceptors can make new effective connections w/ dorsal horn neurons.

49
Q

TrPs have been reported in all age groups except which?

50
Q

The current thinking & attention of TrPs has shifted more from mech factors –> __________ factors.

A

biopsychosocial

51
Q

What should be the last step in physical examination process?

A

palpation of relevant muscles

52
Q

What are the 3 criteria for identification of TrPs?

A
  • taut band in relevant muscle
  • presence of TrP in band
  • referred pain
53
Q

The most common metabolic & hormonal factors in myofascial pain are (hyper/hypo)thyroidism, gonadal hormone conditions, protozoal infections, & iron, mag, B12, D, estrogen, test deficiencies.

54
Q

Possible side effects of meds such as w/ ________ drugs may induce widespread myalgias.

55
Q

What muscles are in the superficial layer of PF?

A
  • external anal sphincter
  • sexual (bulbocav & ischiocav)
  • superficial transverse perineal
56
Q

What muscles are in the urogential diaphragm (2nd layer) of PF?

A
  • sphincter urethra
  • urethrovaginal sphincter
57
Q

What muscles are in the levator ani (deepest layer)?

A
  • puborectalis
  • pubococc
  • coccygeus
  • ishococc
58
Q

Which layer participates in urinary continence? Which supports pelvic viscera?

A
  • continence: 2nd
  • viscera: deep
59
Q

Urinary or fecal incontinence & pelvic organ prolapse is due to (under/over)activity of PFM.

60
Q

Obstructive voiding or defecation, dyspareunia, & pelvic pain is due to (under/over)activity of PFM.

61
Q

What is the most common factor of underactivity of PFM?

A

birth-related trauma

62
Q

What are the 2 categories of origin of dysfunction of overactivity of PFM? Which one has evidence said is the primary origin?

A
  • tissue-based/nociceptive
  • neuropathic/central sensitization*
63
Q

What is the term for pain related to coccyx & muscles attached there?

A

coccygodynia

64
Q

What are the categories of causes of coccydynia?

A
  • MSK
  • direct trauma (childbirth or fall)
  • inflammation
  • infections
  • referred pain from visceral sources
  • neoplasm
  • centralized pain syndrome
65
Q

What is coccygeal spicule?

A

hook on end of coccyx

66
Q

Sacral _________ is a rare, slow-growing tumor that should be considered in diff dx of coccygodynia.

67
Q

What is the term for where 2 pelvic sympathetic trunks converge ending in ganglion @ front of coccyx (which can cause chronic pain w/ overactivity)?

A

ganglion impar

68
Q

What is the most susceptible area for osteochondroses?

69
Q

What is the disorder of 1 or more ossification sites w/ localized subchondral necrosis followed by recalcification?

A

osteochondritis dissecans (OCD or OD)

70
Q

In OCD/OD, piece of _________ cartilage & fragment of bone separate & pull away from underlying bone –> loose in joint.

71
Q

Where are the most common sites of involvement of OCD/OD?

A

concave surfaces of synovial jts:
- medial femoral condyle
- talar head
- capitellum of humerus

72
Q

OCD/OD is caused by repetitive __________ resulting in ischemia & disruption of subchondral growth.

A

microtrauma

73
Q

What is the term for when pain is increased w/ passive knee extension & tibial IR & relieved w/ tibial ER?

A

wilson sign

74
Q

What is the term for death of bone & bone marrow cellular components bc of loss of blood supply in absence of infection?

A

osteonecrosis (avascular/aseptic)

75
Q

What is the most common site of osteonecrosis? What is the name for this?

A

femoral head (chandler disease)

76
Q

What are other sites in which osteonecrosis is common?

A
  • scaphoid
  • talus
  • prox humerus
  • tibial plateau
  • small bones of wrist & foot
77
Q

What type of gait abnormality is seen w/ osteonecrosis in femur?

78
Q

What type of surgical interventions are done for osteonecrosis?

A
  • core decompression
  • hemiarthroplasty
  • total jt replacement
79
Q

What are other names for legg-calve-perthes disease?

A

coxa plana (flat hip) & osteochondritis deformans juvenilis

80
Q

Legg-calve-perthes disease is epiphyseal aseptic necrosis of proximal end of __________.

81
Q

Who does legg-calve-perthes disease occur in the most?

A

boys b/t 5-8 yrs

82
Q

Legg-calve-perthes disease has insidious onset w/ intermittent appearance of a limp on involved side & ________ pain w/ soreness, aching, & stiffness.

83
Q

Pain of legg-calve-perthes disease follows the path of which nerve?

84
Q

What disease results from fibers of patellar tendon pulling small bits of immature bone from tibial tub (considered as tendinitis)?

A

osgood-schlatter

85
Q

Which age groups in boys & girls is osgood-schlatter seen in? Who is it seen in more often?

A
  • boys: 10-15*
  • girls: 8-13
86
Q

What 3 abnormal alignment conditions can lead to osgood-schlatter?

A
  • genu valgum
  • flat-footed
  • high-riding patella (patella alta)
87
Q

Avulsion of _________ ossification center of tibial tub occurs when patellar tendon fibers pull fragments away from tibial epiphysis.