Week 4--NEURO --(MR. LIEB) Flashcards
TWO MECHANISMS OF INJURY WITH REGARD TO NEURO INJURIES
- BLUNT TRAUMA
- mvc
- Falls
- Sports injuries
- Assault
- PENETRATING INJURIES
- unintentional injuries
- firearms (ie, guns)
Since NEURO injuries require ALOT OF FORCE to become injured…there are other “concurrent” injuries that nurses need to be mindful for.
- CERVICAL SPINE INJURIES
- Must maintain inline cervical spine stabilization using C-Collar and safe pt transportation until injury has been cleared
- VERTEBRAL COLUMN
- FACIAL FRACTURES
- EYE INJURIES
- RESPIRATORY/AIRWAY COMPROMISE
Primary vs Seconday Injuries
-
Primary <u>(this is the injury we cant change)</u>
-
Neuronal destruction is permanent once neurons die
- Direct transfer of energy leading to injury
- Fractures, bleeds
-
Neuronal destruction is permanent once neurons die
-
Secondary (this outcome can be made better with good care management)
-
Contains PENUMBRA (area surrounding dead area)
- It is at risk for decreased oxygen, decreased glucose and could also die in addition to the already dead tissue
- Region of impaired/ischemic tissue
-
It is viable and may be salvaged
- Caused by pathophysiologic changes after primary injury which lead to:
- Hypotension
- Hyperthermia
- Hypoxia
- Cerebral edema
- Increased intracranial pressure (ICP)
- Caused by pathophysiologic changes after primary injury which lead to:
- It is at risk for decreased oxygen, decreased glucose and could also die in addition to the already dead tissue
-
Contains PENUMBRA (area surrounding dead area)
These changes can further damage vulnerable cells
With PRIMARY INJURIES–Neuronal destructrion is permanent once neurons die
True or False
true
SECONDARY INJURIES contain an area called _______________ that are found around the central core of dead neurons
PENUMBRA
“4” types of FOCAL BRAIN INJURIES
- EPIDURAL HEMATOMA
- SUBDURAL HEMATOMA
- CEREBRAL Contusion
- Intracerebral Hematoma
EPIDURAL HEMATOMA
- 90 % occur as a fracture to temporal/parietal bone
- Laceration of the middle meningeal artery (rapid accumulation of blood)
- Bleeding between DURA and SKULL
ASSESSMENT
- TRANSCIENT loss of consciousness followed by LUCID period (acting normal)
- Unilateral fixed / dilated pupil
- Rapid Neuro decline
TREATMENT
- surgical intervention is required
- burr hole to lower ICP
SUBDURAL HEMATOMA
- Injury to “Bridging Veins” …BELOW THE DURA
- Direct Injury to brain tissue
ACUTE
- S/S begin to appear within 48 hrs (2 days)
- Decreased Cerebral blood flow
- HIGH MORTALITY RATE 30-90%—(lower if surgery happens)
CHRONIC
- S/S may take 2 weeks (or more) to be symptomatic
- slow accumulation of blood
AT RISK POPULATION
- anticoagulant use
- older adults and chronic ETOH abuse (shrinks brain putting extra pressure on bridging veins causing them to break open
TREATMENT
- If SMALL……..Observe and allow body to reabsorb
- IF LARGE or Causing significant neuro s/s…..surgical evaluation
DIFFERENCE
between
EPIDURAL HEMATOMA
AND
SUBDERMAL HERMATOMA
EPIDURAL = arterial blood SUBDURAL= Venous blood flow
- DURA is peeled back from skull DURA still attached to skull
- fractured bone
CEREBRAL CONTUSION
- Bruising of the brain
- Capillaries are damaged
- Hemorrhage or Infarction
S/S will peak at 18-36 hours and they depend upon the size and location of contusion
INITIALLY, pt is often unconscious
Behavioral changes / irritability
motor or speech
INTRACEREBRAL HEMATOMA
**similar to hemorrhagic shock
Occurs deep within the brain (Parenchyma)
May be associated with contusions
The hematoma compresses brain tissue- 2cm or greater causes great concern
ASSESSMENT
Decline in LOC as hematoma grows
TREATMENT
- IF SMALL—watch and wait if small or deep in the brain
- REPEAT CT’S q 4 hours
- SURGICAL REMOVAL IF LARGE OR EASILY ACCESSIBLE
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