Week 4 Flashcards

1
Q

What are UMN lesions associated with?

A

Normal increased reflexes
late and mild atrophy
Normal or increased tone

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2
Q

What are LMN lesions associated with?

A

Decreased or absent reflexes
Rapid and severe atrophy
Decreased or absent tone

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3
Q

What are the types of stroke?

A

Ischemic - clot/block
Hemorrhagic - bleed/burst
TIA- smaller blockage

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4
Q

Dysphagia in ______ of acute stroke cases

A

29-80%

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5
Q

How is improvement of dysphagia in strokes?

A

50-90% show rapid improvement to near baseline function

Persistent dysphagia continues in 20-50%

Of the persistent, 50% aspirate and 35% develop AP

AP is the leading cause of re-hospitalization in acute stroke

AP contributes to >50% of deaths in the first 30 days post stroke

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6
Q

What is silent aspiration?

A

aspiration w/o sensation (no awareness, no coughing, no throat clearing)
CN X damage (sensory)

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7
Q

Silent aspiration is present in _____ of post stroke dysphagia cases and commonly causes AP

A

2-66%

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8
Q

What are indications for instrumental assessment?

A

AP
cough
wet voice
diff w/ hydration/nutrition

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9
Q

Which is more detrimental to swallowing: cortical or brainstem stroke?

A

Brainstem

Location of many CNs

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10
Q

What are primary characteristics of GERD?

A

heartburn and esophagitis

Nightime, supine reflux

Associated with obesity

LES dysfunction and poor motility

Esophagus is protected against acid

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11
Q

What are primary characteristics of LPR?

A

70% of pt deny heartburn (silent); only 25% have esophagitis

Daytime, upright reflux

No Rx with BMI

UES dysfunction and seemingly good motility

Laryngeal/pharyngeal region is poorly protected from acid

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12
Q

What are GERD lifestyle modifications?

A
Elevate HOB 6 in.
Smoking cessation
Low-fat diet
Weight loss
Avoid lying down w/i 3 hrs of eating
Eat frequent small meals
Avoid refluxogenic foods: alcohol, chocolate, spicy, citrus
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13
Q

what are causes of esophagitis?

A
GERD - most common
Infection (ex. candida)
Foreign bodies (ex. pills)
Chemo and radiation
Eosinophilic esophagitis)
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14
Q

Eosinophilic esophagus

A

used to be rare, more common now

Allergic inflammation of esophagus

Cause unknown

referral to allergist

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15
Q

What is treatment for LPR?

A

Standard GERD reccomendations

Meds-h2 receptor antagonists and/or PPI

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16
Q

Which is more common to cause solid food dysphagia: A or B ring?

A

B ring - Tx: dilation

If A ring tx needed: botox

17
Q

What are common findings in TBI?

A

residue, bolus spill, delay swallowing

18
Q

What is the presenting complaint of myasthenia gravis?

A

Dysphagia

high risk of AP, aspiration typically silent

not good to repeat trials and exercises, can deplete ACh

19
Q

How do you decide if there is a presence of presbyphagia or dysphagia?

A
  1. overt safety issue

2. case Hx

20
Q

What are typical signs of presbyphagia

A
longer swallowing
muscle atrophy
increase in pharyngeal movement
decrease in pharyngeal muscle mass
loss of sensation
longer swallow trigger
increased cortical activity

increased or reduced kinematic output? Literature is controversial