Week 3 - Spasticity & History Flashcards

1
Q

A motor disorder characterised by a velocity dependent increases in tonic stretch, reflexes with exaggerated tendon jerks, resulting from hyperexcitability of the stretch reflex, as one component of UMN syndrome

A

Spasticity

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2
Q

The amount of resistance to passive elongation or stretching when a muscle is relaxed.
Resistance felt when passively stretching relaxed muscle

A

Tone

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3
Q

What are the objectives in treating spasticity?

A

To improve quality of life
- Relive symptoms and reduce disfigurement
- Ease personal care and positioning
- Improve function and mobility of a specific limb
- Reduce burden of care
- Enable ADL

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4
Q

What is spastic dystonia?

A

Stretch sensitive muscle contraction in absence of volitional command (at rest)

Involuntary muscle contractions and an inability to relax muscles

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5
Q

What is spastic co-contraction?

A

Inappropriate antagonist, actively using muscle and getting an inappropriate antagonist recruitment in the absence of stretch

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6
Q

Bidirectional resistance to passive stretch, relatively independent of the speed of movement; does not result from hyperactive stretch reflexes

A

Rigidity

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7
Q

What is cogwheel and ledpipe rigidity?

A

Cogwheel: tension in muscle gives way in little jerks as you try to straighten limb

Ledpipe: rigid whole way through movement, have to push quite hard “might feel like it’ll break”

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8
Q

Immobolisation in shortened position results in less longitudinal tension

A

Contracture

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9
Q

List 3 pathophysiology to spasticity

A
  1. Imbalance between excitatory and inhibitory impulses to the alpha motor neuron
  2. Descending pathways also influence Renshaw cells which suppress repeated firing of alpha motor neurone
  3. Descending pathways also inhibit Golgi Tendon Organ (GTO)
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10
Q

List 3 management of spasticity

A
  1. Muscle lengthening
  2. Muscle strengthening and retraining
  3. Local muscle relaxation
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11
Q

How does botox BTX-A help with spasiticity

A

Blocks neuromuscular transmission by inhibiting release of Ach from presynaptic terminals, resulting in a reversible denervation atrophy

Blocks chemical signal between nerves and muscles that makes muscle contract or tighten

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12
Q

What is Alexander Technique?

A

Patients learn to observe the way they move and make small changes to posture/coordination

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13
Q

What is Feldenkrais method?

A

Described as somatic education
2 components: awareness through movement, functional integration

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