Week 3 Heart Axis And Cardiac Flow Flashcards

1
Q

What is one lead

A

A set of TWO electrodes

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2
Q

What does a lead measure

A

Records a voltage shift
As waves spread over the heart

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3
Q

How are direction and strength of a signal related in a lead

A

When a wave is parallel (same direction) it sis stronger

Perpendicular - no signal

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4
Q

Why is einthovens triangle formation used

A

A wave travelling in any direction will be parallel to at least one of the three leads

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5
Q

Ground electrode - purpose

A

Acts as a baseline

Looks at the natural voltage of the ground

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6
Q

By convention - what are the ECG directions for depol and repol

A

Depol - positive (up)

Repol - negative (down)

Waves travelling away from the positive electrode - opposite ^

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7
Q

Why is lead 2 normally used in a single lead trace

A

Lead 2 has the strongest response to a healthy heart

Several issues can be detected on lead 2 - other leads can then be assessed

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8
Q

Q of QRS

A

Depolarisation in the direction of the negative electrode - downward slope

small magnitude

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9
Q

Upwards slope R of QRS

A

Ventricle is being depolarised

Happens in the direction of lead 2

Huge wave in the positive direction

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10
Q

Downward slope R of QRS

A

Ventricle is still depol
But wave points less at the positive electrode
So is seen returning to 0

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11
Q

S of QRS

A

Opposite direction to positive electrode
Wave travels toward atria
Small magnitude
Trace is negatice

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12
Q

What is the heart axis

A

The mean vector
Describing direction of voltage in the heart

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13
Q

Heart axis calculation

A

Calculates mean of each mean electrical wave
Normal is 0-90 degrees

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14
Q

what causes ventricular fibrillation

A
  • AP keeps circling back on itself
  • or cardiomyocytes are repeatedly contracting
  • or contracting irregularly so ventricle is out of synch
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15
Q

when to use a defibrillator

A
  • not for a flatline
  • is used for ending FIBrillation
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16
Q

how does a defib work

A

depolarises all cardiac cells at once

to create a fresh signal from myogenic pacemaker cells

to bring back synchrony in beating

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17
Q

what are the two valves

A

mitral/bicuspid - 2 cusps
tricuspid - 3 cusps

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18
Q

what is the wiggers diagram

A

a method to visualise several aspects of the heart at once

19
Q

systole

A

period of contraction of the ventricles

20
Q

diastole

A

relaxation of heart muscle + then chamber fills with blood

21
Q

phases of systole

A
  1. isovolumetric contraction
  2. rapid ejection phase
  3. slow ejection phase
22
Q

phases of diastole

A
  1. isovolumetric relaxation
  2. rapid filling phase
  3. slow filling phase
  4. atrial contraction
23
Q

what phase of diastole is the P wave

A

atrial contraction / depol

24
Q

what phase of systole is QRS

A

ventricular contraction

25
what phase of systole is the T wave
ventricular relaxation / repol
26
aortic pressures in 1) diastole 2) ejection phase 3) isovolumetric phase
1) diastole - 80mmHg 2) ejection phase - 120 3) isovolumetric relaxation - aortic valve closes, causes dicrotic notch
27
ventricular pressure 1) isovolumetric contraction 2) ejection phase 3) isovolumetric relaxation
1) ventricle contracts, closed chamber so pressure increases when ventricular pressure crosses (is higher) the aortic pressure, the aortic valve opens 2) ejection phase is the top peak ◠ curve when VP decreases and drops below the aortic pressure, the aortic valve closes 3) isovolumetric relaxation starts - pressure decreases - slope downwards when VP falls below atrial pressure (1-7 mmHg) - mitral valve opens - rapid and slow filling ventricle phase starts
28
Atrial pressure
starts high than VP mitral valve closes when ven pressure raises -> raises atrial pressure ejection phase - atrium is filled + pressure rises when pressure is higher than VP - mitral valve opens and filling phase of ventricle starts
29
ESPVR
end systolic pressure volume relationship the max pressure of left ventricle at any volume a measure of cardiac contractility
30
EDPVR. passive filling curve of the left ventricle during diastole and is a measure of passive chamber stiffness
end diastolic pressure volume relationship passive filling curve of the left ventricle during diastole measure of passive chamber stiffness
31
factors affecting cardiac output:
1. preload 2. frank starling mechanism 3. afterload 4. compliance 5. inotropy
32
what is cardiac output
amount of blood that the heart pumps in a minute
33
what is preload
how much blood is in the chamber at the end of diastole more blood entering heart = more preload so heart increases stroke volume
34
stroke volume
The volume of blood pumped out of the left ventricle of the heart during each systolic cardiac contraction
35
factors that increase preload
these factors increase ventricular filling time 1. increased atrial contractility 2. increased central venous pressure 3. decreased heart rate
36
frank starling mechanism
when the heart is stretched the heart contracts more so end systolic volume is maintained more contracting = more preload = increased stroke volume
37
afterload
the pressure the heart has to pump to push blood out during ventricular contraction (systole)
38
aortic pressure
the pressure at the root of the aorta caused by blood pushed from the LV to the aorta
39
how does afterload determine end systolic volume
increased aortic pressure - increased afterload 1. higher aortic pressure = aortic valve closes earlier 2. less ventricular emptying 3. volume is at the end of systole
40
what is compliance
the inverse of stiffness ie. MORE stiff, LESS compliant
41
how does compliance contribute to preload
it alters EDVPR hypertrophy: preload is reduced by having a stiffer heart ventricle walls do not stretch as far in response to more blood volume
42
what is inotropy
increases the speed of contractility lowers end systolic volume (ESV) affects ESPVR
43
inotropes
make the heart beat with more or less power
44
between which phases does ejection occur
spans from the end of isovolumic contraction to the beginning of isovolumic relaxation