Week 2 ECG Flashcards
How are electrical signals of the heart detected
- Electrodes are placed on skin
- Machine records heart rhythm
- By recording waves of electrical activity
- Produces an ECG
What does an ECG measure
Effect on anatomical features of the heart
Not cell membrane potential
Shows PQRS wave
What does the PQRS wave show
Movement of an electrical wave across skin
P of PQRS
Atrial depol / contraction
QRS of PQRST
Bundle of His, purkinje fibres
Ventricular depol
Contraction
T of PQRST
Ventricular relaxation / repolarisation
SAN relation to ECG
- Not seen much as there are not a lot of cells
- Also Not directional
Atrial muscle relation to ECG
Takes up most of the P wave
- lots of muscle tissue
- depol = upward
- Steep opening of sodium channels ⬆️
- L type calcium ion channels open = plateau
- Calcium channels close, K+ continues to flow out
AVN relation to ECG
Small mass - seen at the end of the P wave
Common bundle, branches and purkinje fibres + ECG
Not seen due to small area
Ventricular muscle relation to ECG
- largest muscle mass
- shows as PQRST complex
- depolarisation + repolarisation
What is happening at the level stage between S and T
L type calcium channels remain open for a long time
So there is no change = levelling on the ECG
What is happening during the T wave
Repolarisation but in the opposite direction
So is seen as upwards - the same as depol
What anatomical feature of the heart has the largest impact on the ECG
Ventricular muscle
What direction is an ECG
Too left to bottom right
^ affects how depol and repol are seen
- depol is normally up, repol is down
Reversed when going from bottom right to top left
What are segments on an ECG
Area between waves
1. PR
2. ST
What are the two intervals on an EVG
PR and QT
Named from the first wave
What is an atrioventricular block
Abnormal or absent conduction
So downstream tissue will not be excited by the travelling AP wave
How is an AV block managed
Pacemaking tissue further along will generate its own APs
Conduction in 1st degree AV blocks
Conduction through AVN is slowed but not interrupted
P-QRS in 1st degree AV block
One P for every QRS (as normal)
But there is a longer gap
Of more than 200ms
Conduction in 2nd degree AV block
Partially interrupted
But mostly proceeds
P-QRS complexes in 2nd degree AV blocks
Several QRS complexes have no P wave
Conduction in 3rd degree AV blocks
Conduction through the AVN is blocked