Week 3 - Arrhythmias Flashcards

1
Q

What is the most common arrhythmia

A

AF

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2
Q

What is the prevalence of AF

A

Increases with age.
10% ≥ 70 years; 23% ≥ 80 years

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3
Q

What causes AF

A

Supraventricular tachyarrhythmia
– Uncoordinated atrial contraction
– Irregular and frequently fast ventricular rate

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4
Q

What is the classification for AF

A

First diagnosis - Paroxysmal AF - Persistent AF - Long standing persistent AF - Permanent AF

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5
Q

What are symptoms of AF

A

Palpitations, Dyspnoea, Chest tightness, Fatigue / lethargy, Sleeping disturbance, Psychological effects

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6
Q

Aetiological factors which cause AF

A

Aging (structural remodelling), Heart failure • Hypertension and Diabetes mellitus • Valvular heart disease (esp. mitral) • Coronary artery disease (atrial ischaemia) • Alcohol excess • Hyperthyroidism (trigger) • Obesity and sleep apnoea • Autonomic activation

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7
Q

What is the diagnostic work up of AF
What investigations would you do

A

12-lead ECG • BP • Bloods - FBC, U&E, LFT, TFT, Coag • Echocardiogram • Holter monitoring
– Symptom / Rhythm correlation
– AF burden
– Ventricular rate control

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8
Q

How does AF increase risk of stroke?

A

Blood pools in the atria
Blood clot forms
Whole or part of the clot breaks off
Bloood clot travels to the brain and blocks a cerebral artery causing stroke

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9
Q

What is the drug of choice for AF when no structural heart disease

A

Flecainide

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10
Q

What is the best drug for AF with structural changes

A

Amiodarone

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11
Q

Heart rate control drugs examples

A

beta blockers
Verapamil
Digoxin

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12
Q

What is the score name for stroke risk in AF

A

CHA DS- VASc score

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13
Q

What is bradycardia

A

Lower than 60 bpm

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14
Q

What can cause bradycardia

A

Congenital
Acquired
Degenerative - Ischaemic heart disease - Drugs - Electrolye/metabolic - Infection (eg endocarditis) - Iatrogenic (ablation, cardiac surgery) - Infiltrative diseases (eg sarcoid,
amyloid) - Neuromuscular diseases (eg
myotonic dystrophy)

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15
Q

Which artery supplies the AV node

A

The right coronary

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16
Q

What symptoms does bradycardia present with

A

Asymptomatic
Dizziness
Breathlessness
Syncope
Falls
Exercise intolerance
Difficulty concentrating
Fatigue

17
Q

Tachycardia-bradycardia syndrome

A

Pacemaker for fast heart rate
Drugs for slow heart rate (beta blockers)
Anticoagulation for strokes

18
Q

Type 1 AV block

A

First Degree – PR interval is prolonged but all impulses are
conducted to the Ventricles

19
Q

Second degree AV block

A

Second Degree – Some (but not all) impulses are conducted to the ventricles

20
Q

Third degree AV block

A

Third Degree – No impulses are conducted to the ventricles

21
Q

Type 1 second degree hear block is called?

A

Wenckeback

22
Q

In Mobitz Ii

A

P to QRS interval is normal
Sudden p wave not followed by QRS - `Need pacemaker

23
Q

2:1 AV block

A

For ever 2 P waves there is only 1 QRS

24
Q

How do people with tachycardia present

A

Asymptomatic
SOB
Palpitations
Pre-syncope/ light headed
Chest pain
LOC
Cardiac arrest

25
Background history
Clinical context Age Medical background Family history Congenital abnormality Development of substrate Acquired abnormality
26
How would you describe AF
Tachycardia, narrow QRS, irregular, no P waves
27
What are differentials for narrow complex tachycardia
Sinus tachycardia AF A flutter Focal atrial tachycardia AVNRT AVRT
28
What are differential diagnosis for brad complex tachycardia
Ventricular tachycardia SVT with aberration Pre-excited tachycardia Pacemaker associated tachycardia
29
How do you cure arterial flutter
flutter ablation
30
How fast odes the heart beat in atrial flutter
150-200bpm
31
Management of AF and flutter, AVRT, AVNRT
Manage stroke risk Rate control Termination of re-entrant arrhythmia Anti0arrhythmic therapy Interventional electrophysiological procedures
32
what does VT present as
Previous MI which causes scar - areas don’t conduct - short circuit - bad ventricular can depolarise Broad complex Positive/negative concordance Basically regular Extreme axis Not a typical BBB AV disassociation - see P waves as SA node is normal Slurred initiation of QRS
33
How to tel between VT and RBBB
sharp initiation in RBBB and slow in VT
34
Above the AV node give adenosine
Shows the flutters but no affect
35
Att the AV bnove
Adenosine will work on the issue
36
Below the AV node
Adenosine makes not affect