Week 12: Coronary Vascular Disorders, Atherosclerosis, Angina, MI Flashcards
Flow of Blood from Heart to Body
IVC/SVC –> RA –> Tricuspid –> RV –> Pulmonary Valve –> Lungs –> LA –> Mitral Valve –> LV –> Aortic Valve –> Aorta
Main Coronary Arteries
RCA - Right Coronary Artery
Left Main Coronary Artery (LCA)
a. Left Circumflex Artery (LCx)
b. LAD - left anterior descending coronary artery
LAD
Left Anterior Descending Artery
Called the Widowmaker
Supplies so much blood to the left side of the heart that an acute change can cause death quickly
RCA
Right Coronary Artery
Can cause a lot of heart rate issues, so a patient with a low 50s HR would not necessarily mean anything bad it may mean they have bradycardic symptoms from RCA problems (d/t inactivity)
Leading Cause of US Death is
Heart Disease
CAD
Coronary Artery Disease
Buildup of plaque in coronary arteries - plaque buildup in the walls of coronary arteries
Blocks flow and is often unnoticed until the blockage is more than 70%
What makes up the plaque in CAD
Usually lipids, other fatty substances, fibrous material
What % of blockage of coronary arteries does it take to lead to symptoms being seen?
70% minimum usually
Could be 90-100% because of collateral circulation though
How does CAD differ in Men compared to Women
CAD manifests 10-15 years sooner
Initial cardiac event is more often an MI than angina
Higher incidence of LVH
How does CAD differ in Women compared to Men
CAD causes more death in women
Initial cardiac event more likely to be angina than MI
Complain of palpitations more frequently than men
LVH
Left ventricular hypertrophy
thicker musculature and hypertrophy of the heart causing it to get bigger and it tries to pump more blood - but it keeps getting bigger and lessens how efficient it is
Non Modifiable Risk Factors for CAD
Age
Gender
Ethnicity
Genetics and Family Hx - high risk for CAD and MI if in nuclear family
What is the gender disparity with CAD
men > women until age 60
What is the ethnicity disparity of CAD
AA > Caucasian
South Asian High - Japanese Low
Modifiable risk factors for CAD
serum lipids
HTN
tobacco
physical inactivity
obesity - waist circumference and BMI
also watch persons with DM, fasting BS > 100, psychological states
elements of metabolic syndrome
What are the 3 most common risk factors for CAD that 9/10 patients have
HTN
Hyperlipidemia
Obesity
Nursing Management of CAD Risk Factors
Health promotion - ID high risk people through risk screening and work on modifiable factors with lifestyle changes
Physical activity
nutritional therapy - lower LDL cholesterol
cholesterol lowering drug therapy
anti platelet therapy
FITT Formula of Physical Activity to counteract CAD
FITT Formula = Frequency, Intensity, Type, Time
Moderate exercise 30 min/day on 5 or more days/week - brisk walking, hikin, biking, swimming
What does following the FITT Formula lead to
Contributes to weight reduction, 10% drop in SBP, diabetics - better blood glucose
Nutrition education for CAD emphasizes what things
Decrease Sat Fat, Cholesterol, Red meat, eggs, whole milk products, alcohol, simple sugars
Increase Complex Carbs (whole grain, fruit, vegis) and Omega 3 FA
Fat intake 30% of calories - good oils (olive, canola)
What should be done if a patient with CAD has elevated serum triglycerides
alcohol intake and simple sugars should be reduced or eliminated
Where to get Omega 3 Fatty Acids
Eating fatty fish 2x a week - salmon and tuna
tofu, soybean, canola, walnuts, flaxseed
Angina
chest pain or pressure resulting from myocardial ischemia (reflects imbalance between cardiac oxygen demand and supply)
Directly related to myocardial ischemia - but not all chest pain is this (could be eating too much, gas, or somatic pain)
Types of Angina
Stable
Unstable
Intractable or Refractory
Variant (Prinzmetal’s Angina)
Silent Ischemia
Stable Angina
predictable, manageable episodes of chest pain
Relieved with rest and/or nitroglycerin
occurs with exercise
Unstable Angina
unpredictable
occurs when resting or with minimal activity or at varying levels
occurs with increasing frequency, duration, and severity
needs further workup and tx
comes and goes randomly
Interventions for Stable Angina Acute Episodes
stop all ativity and sit or rest in bed
Assess patient - VS including O2 sat, resp distress, assess pain, pain, diaphoresis?, sudden LOC change
Administer supp. O2
12 lead EKG
pain assessment and relief - nitrate then opioid
auscultate heart sounds
Unstable Angina has a high risk for…
Myocardial infarction
Prinzmetal;s Angina
occurs at night in clusters
from artery spasms
Refractory Angina
Reoccurs despite treatment
Silent Angina
No s/s
no chest pain but myocardial ischemia is occurring!
Tests pick up ischemia affecting heart and perfusion
Acute Coronary Syndrome (ACS)
Prolonged ischemia that causes DAMAGE to the heart!
An umbrella term for damage to the heart from ischemia
3 categories of ACS
unstable angina
STEMI
NSTEMI
STEMI
ACS
Complete occlusion of coronary vessel(s)
ST elevation on EKG
NSTEMI
ACS
partial occlusion of coronary vessel(s)
No ST Elevation, but other EKG changes are possible