week 10- learning and memory Flashcards
case hm- bilateral med temp lobectomy
sufferer eplepsy
removed hippocampus’
amygdala
adjacent limbic cortex
treated but caused amnesia
effecting
–STM in tact- normal 7 plus minus 2 range)
–LTM- impaired-unable recall recent past events last 2 years- RETROGRADE
unable new mem- ANTERGRADE
implicit LTM intact ( improved performance not remember how)
findings
medical temporal lobes= specific mem
effect bilteral MT labetroy but STM mem in tact
supporting modal theory
and two parrell LTM- exlicit and implicit
whats declarative mem ( explicit)
- hippocampus role in formation of LTM declarative
- inloved in consolidation- STM – LTM
Consolidation
disrupted- lost mem
only involves tem story area for LTM, when fully formed declarative long term store permanently
hippocampus and spatial memory
key structure
- lesions morris water maze
emotional mem
amygdala- provides hippocampus in mem with emotional context (flashbulb)
amnesia of Kd syndrome
vit B deficiency/ associated alcohol
antegrade amnesia- explicit
serve retrograde amnesia- if progression
motor issue
confusion
KS damage to
medical hypothalamus
hippo
cerebellum’ neocortex
warehouse theoyr
mem result of sensory and motor and cerbral cortext
learning=
interconnect cell— all activated— strength connection— create mem
reverberate= create LTM
prolonged reveration- creation
Potential long term
change strength synpase= result firing or pre and post synaptic neurons
medical temporal lobes
MTL not involve in STM/implicit mem
MTL not permanent for explicit (dec)
MTL involve in formation in LTM explicit (STM to LTM)
MTL medicated consolidation process is gradual