Week 10; Acute Care Endocrine Flashcards

1
Q

Most common causes of eye injury

A

– Abrasions
– Lacerations
– Foreign bodies
* Traumatic injury can also be caused by penetrating object, blunt force, burns

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2
Q

Prevention of eye injury

A

Protective eyewear prevents >90%
– >78% of individuals not wearing eyewear at time of injury
* Type of protection depends on activity
– Home use: eyewear labeled “ANZI Z87”
– Sports and recreation: depends on sport
– Proper UV protection when water or snow skiing
– OSHA determines what eye protection is required in workplace

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3
Q

Corneal abrasion

A

– Disruption of superficial epithelium
– Caused by objects such as contact lenses, eyelashes, small foreign bodies
– Superficial abrasions painful but heal rapidly without complications, scarring
– Photophobia, tearing common
– Stroma damage
▪ Increased risk of infection
▪ Slowed healing, scar formation

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4
Q

Burns to the eye

A

– Caused by heat, radiation, explosion, chemical
– Chemical burns most common
▪ Both acid and alkaline substances
▪ Alkaline eye burns particularly serious
▪ Acid burns: rapid damage, less serious
– Explosions, flash burns → greatest risk for thermal burns

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5
Q

UV damage to eyes

A

UV rays → corneal damage
▪ Depending on source: snowblindness, welder’s arc burn, flash burn
– History of face, eye contact with caustic substance or other burning agent
– Eye pain, decreased vision
– Eyelids, face, lips may be affected
– Sloughing with chemical burns
– Cloudy, hazy cornea with ulcerations

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6
Q

Penetrating injury

A

▪ Layers of eye spontaneously reapproximate
▪ Single entrance wound
– May be hidden because of tissue swelling
– May be missed when patient has other significant injuries
– Vital to inspect underlying eye tissue for damage
– Pain
– Partial or complete loss of vision
– Possible bleeding

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7
Q

Perforating injury

A

▪ Layers of eye do not spontaneously reapproximate → rupture of globe,
potential loss of ocular contents

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8
Q

Orbital blowout fracture

A

– Diplopia
– Pain with upward movement of eye
– Enophthalmos, limited movement

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9
Q

Hyphema

A

– Eye pain, decreased acuity, reddish tint

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10
Q

Detached retina

A

– Separation of retina from choroid
– Usually occurs spontaneously but may be precipitated by trauma
– Retina may tear, fold back on itself or may remain intact but not adhere to
choroid
– Detached area may enlarge rapidly, increasing vision loss
– Permanent vision loss unless contact reestablished
– Floaters, spots, lines, flashes of light
– Sense of curtain drawn across vision
– No pain, eye appears normal

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11
Q

Detached retina risk factors

A

▪ Aging
▪ Myopia
▪ Glaucoma
▪ Trauma
▪ Previous retinal detachment
▪ Aphakia

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12
Q

Eye injury dx

A
  • Visual acuity tests
  • Extraocular movement evaluation
  • Flashlight or ophthalmoscope: pupil reactivity, size
  • Ophthalmoscope: red reflex
  • Slit lamp, fluorescein stain: corneal defect
  • Facial x-rays
  • CT scans
  • Ultrasonography
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13
Q

Severe chemical burns to the eye treatment

A

– Debridement
– Tissue grafting
– Corneal transplant
Pain medication, steroids, cycloplegic drops

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14
Q

Retinal detachment: treatment

A

– Cryotherapy
– Laser photocoagulation
– Scleral buckling
– Pneumatic retinopexy
Steroids to reduce inflammation

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15
Q

Corneal abrasion treatment

A

After removal of foreign body, antibiotic ointment

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16
Q

Children eye injuries

A

Common causes of eye injuries
▪ Blunt trauma from ball or fist
▪ Sharp trauma from projectiles, sticks
▪ Chemical trauma from household chemicals
▪ Burns from fireworks
– Treatment same for all ages
– Help prevent injuries via patient teaching
– External eye injuries
▪ Two black eyes may suggest abuse

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17
Q

Older adults and eye injury

A

Older adults
– Most frequent cause: falling
▪ Slipping on wet surfaces
▪ Falling down stairs
– More at risk for falls
▪ Poor eyesight
▪ Bifocals that may alter depth perception
▪ Decreased cognition
– Patient teaching to prevent falling

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18
Q

Eye assessment injury

A

Nursing history
– Time, type, extent of injury
– Circumstances of injury
* Physical assessment
– Vision assessment
– Eye movement unless penetrating object
– Inspection
– Early manifestations of retinal detachment

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19
Q

Reduce risk for impaired vision

A

– Assess vision in each eye and both eyes: with, without correction
– Inspect eyes
– For burn or foreign body, consider anesthetic drops, irrigating eye
– Remove loose foreign bodies
– For severe or penetrating injury, promote rest, stabilize injured eye
– Apply eyedrops, ointment as prescribed

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20
Q

Interventions for retinal detachment

A

– Notify healthcare provider, ophthalmologist immediately
– Position patient so area of detachment is inferior
– Maintain calm, confident attitude
– Reassure patient that most retinal detachments are successfully treated
– Explain all procedures fully
– Allow supportive family members to remain
Discuss preparations for home care
▪ Limitations on positioning head before and after repair
▪ Activity restrictions
–No bending
–No straining at stool
▪ Use of eye shield
▪ Early manifestations, importance of immediate care and follow-up treatment

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21
Q

DIABETES MELLITUS

A

IS A COMMON CHRONIC ENDOCRINE
DISORDER OF IMPAIRED GLUCOSE REGULATION

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22
Q

Complications of DM management

A

CAN BE GREATLY REDUCED WITH GLYCEMIC
CONTROL BY MANAGEMENT OF HTN AND HYPERLIPIDEMIA AND LIFESTYLE CHANGES

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23
Q

Blood glucose regulation

A

ALL BODY PARTS REQUIRE CONSTANT SUPPLY OF GLUCOSE. SKELETAL MUSCLE, CARDIAC MUSCLE AND ADIPOSE TISSUE REQUIRE INSULIN FOR GLUCOSE TO MOVE INTO CELL.
* NORMAL BLOOD GLUCOSE IN HEALTHY CLIENTS REGULATED BY INSULIN AND GLUCAGON.
* INSULIN IS LIKE A “KEY” THAT OPENS LOCKED MEMBRANES TO GLUCOSE ALLOWING BLOOD GLUCOSE TO MOVE INTO CELLS TO
GENERATE ENERGY

24
Q

Type 1 DM

A
  • BETA-CELL DESTRUCTION LEADING TO ABSOLUTE INSULIN DEFICIENCY
  • AUTOIMMUNE
  • IDIOPATHIC
25
Q

TYPE 2 DIABETES

A

RANGES FROM INSULIN RESISTANCE WITH RELATIVE INSULIN DEFICIENCY TO SECRETORY
DEFICIT WITH INSULIN RESISTANCE

26
Q

Acute complications of diabetes

A

HYPOGLYCEMIA, DIABETIC KETOACIDOSIS (DKA), HYPERGLYCEMIC HYPEROSMOLAR STATE (HHS)

27
Q

Chronic complications of diabetes

A

NEPHROPATHY, RETINOPATHY, CHRONIC HIGH BLOOD SUGARS – MACROVASCULAR COMPLICATIONS, CARDIOVASCULAR DISEASE, CEREBROVASCULAR DISEASE, REDUCED IMMUNITY, GI SYMPTOMS – GASTROPARESES – DELAY IN GASTRIC EMPTYING, SLUGGISH MOVEMENT, BACTERIAL OVERGROWTH,
BLOATING, GAS, DIARRHEA, CAN ALSO LEAD TO CONSTIPATION

28
Q

NEUROPATHY

A

PERIPHERAL NEUROPATHY – PROGRESSIVE
RESULTS IN LOSS OF SENSORY PERCEPTION AND NERVE DAMAGE

29
Q

Hypoglycemia

A

LOW BLOOD GLUCOSE LEVEL THAT RESULTS IN SPECIFIC NEUROGLYCOPENIC AND NEUROGENIC SYMPTOMS AND RESOLVES WHEN GLUCOSE LEVELS RISE ABOVE 70 MG/DL.
* COMMON IN TYPE 1 DM
BLOOD GLUCOSE <70 MG/DL

30
Q

Causes of hypoglycemia

A

INTAKE OF ALCOHOL
DRUGS CAN CAUSE –BETA BLOCKERS,
SULFONYLUREA MEDICATIONS
TOO MUCH INSULIN COMPARED TO FOOD INTAKE AND PHYSICAL ACTIVITY
WRONG TYPE OF INSULIN INJECTED AT WRONG TIME
INSULIN INJECTION NOT CORRELATED WITH FOOD INTAKE.
DECREASED INSULIN CLEARANCE FROM
PROGRESSIVE KIDNEY FAILURE.

31
Q

Hypoglycemia s/s

A

SHAKY/TREMULOUS, TACHYCARDIA, SWEATY, HUNGRY, WEAKNESS, FATIGUE, ANXIOUS, NERVOUS, IRRITABLE, CONFUSION, SEIZURES WHEN CBG <20MG/DL, BRAIN DAMAGE, COMA, DEATH

32
Q

Hypoglycemia treatment

A
  • 15 G OF RAPID-ACTING SUGAR
  • HOSPITALIZED OR URGENT TREATMENT IF BLOOD GLUCOSE < 50 MG/DL
  • COMA, SEIZURES
  • ALTERED BEHAVIORS
  • CAUSED BY SULFONYLUREA DRUG
  • ADMINISTER 50% IV DEXTROSE
  • GLUCAGON 1 MG SC OR IM
  • IF UNCONSCIOUS – GIVE GLUCAGON 1 MG SC OR IM, REPEAT IN 10 MIN IF REMAINS UNCONSCIOUS, NOTIFY
    PROVIDER IMMEDIATELY
33
Q

Patient teaching r/t hypoglycemia

A

CAUSES OF HYPOGLYCEMIA, SYMPTOMS, SELF MANAGEMENT OF HYPOGLYCEMIA, ENCOURAGE WEARING A MEDICAL ALERT BRACELET, PEDIATRIC DM1 MANAGEMENT AT SCHOOL

34
Q

DKA

A

NO INSULIN THEREFORE GLUCOSE IS UNAVAILABLE FOR CELLULAR METABOLISM.
* IN DKA, THE BODY BREAKS DOWN ALTERNATE
SOURCES OF ENERGY. NO INSULIN. BREAKING DOWN FATS WHICH LEADS TO KETOSIS. BLOOD SUGAR >300 MG/DL
* HYPERGLYCEMIA, KETOSIS, ACIDOSIS
* KETONES ARE RELEASED, AND EXCESS KETONES ARE ELIMINATED IN THE URINE (KETONURIA) OR BY THE LUNGS (ACETONE BREATH)
* KETONES IN THE BLOOD ARE STRONG ACIDS THAT LOWER THE SERUM PH AND PRODUCE KETOACIDOSIS

35
Q

S/S of DKA

A
  • KUSSMAUL RESPIRATIONS – RAPID DEEP BREATHS, LOWER CO2 (SIGN OF METABOLIC ACIDOSIS)
  • FRUITY BREATH
  • NAUSEA
  • ABDOMINAL PAIN (FREQUENT IN PEDIATRIC PATIENTS)
  • DEHYDRATION OR ELECTROLYTE LOSS
  • POLYURIA
  • POLYDIPSIA
  • WEIGHT LOSS
  • DRY SKIN
  • SUNKEN EYES
  • LETHARGY
  • COMA
36
Q

Metabolic issues

A
  • HYPEROSMOLARITY
  • METABOLIC ACIDOSIS – PH LESS THAN 7.35
  • EXTRACELLULAR VOLUME DEPLETION
  • ELECTROLYTE IMBALANCES FROM OSMOTIC
    DIURESIS
37
Q

DKA risk factors

A
  • TYPE I DIABETES
  • NON-DIAGNOSED DIABETES – UNDETECTED, 3 P’S (POLYURIA,
    POLYDIPSIA, POLYPHAGIA)
  • CAN OCCUR WITH TYPE II
38
Q

DKA causes

A

*STRESSFUL SITUATION
* SKIPPING MEALS – “STARVATION MODE” – STARTS BURNING FATS
* CORTICOSTEROIDS
* INFECTION
* ACCIDENT
* TRAUMA
* OMISSION OF INSULIN
* MEDICATIONS THAT ANTAGONIZE INSULIN

39
Q

DKA vs HHS symptoms

A

DKA SYMPTOMS – OCCUR SUDDENLY AS OPPOSED TO HHS – OCCUR GRADUALLY

40
Q

DKA priority assessments

A
  • ASSESS THE AIRWAY
  • LEVEL OF CONSCIOUSNESS
  • HYDRATION STATUS
  • ELECTROLYTES
  • BLOOD GLUCOSE LEVEL
41
Q

DKA interventions

A
  1. CORRECT DEHYDRATION
  2. GLUCOSE LEVEL
  3. CORRECT METABOLIC ACIDOSIS
  4. ELECTROLYTE BALANCE
    * 8–10 L FLUID TO REPLACE LOSSES
    * IV FLUIDS WITH 0.9% NS TO 0.45% SALINE WITH 5% DEXTROSE
    * REGULAR INSULIN CONTINUOUS INFUSION USED UNTIL BLOOD GLUCOSE LESS THAN 200
    CARDIAC MONITORING IF HYPOKALEMIA
42
Q

HYPEROSMOLAR HYPERGLYCEMIC
NONKETOTIC STATE (HHS)

A
  • RESULTS FROM A SUSTAINED OSMOTIC
    DIURESIS.
  • SERIOUS, LIFE-THREATENING
  • SLOW ONSET
  • RESULTS IN SEVERE DEHYDRATION
  • PATIENT SECRETES JUST ENOUGH INSULIN TO
    PREVENT KETOSIS
  • EXTREME HYPERGLYCEMIA – BLOOD IS
    CONCENTRATED (HYPEROSMOLARITY)
  • MOSTLY IN TYPE II
  • PT WITH INFECTION OR OLDER ADULT
43
Q

HHS risk factors

A

TYPE II DIABETES, NON-DIAGNOSED DIABETES, OCCURS MOSTLY IN OLDER PATIENTS

44
Q

HHS causes

A
  • KIDNEY DISEASE
  • MYOCARDIAL INFARCTION
  • SEPSIS
  • PANCREATITIS
  • STROKE
  • SOME MEDICATIONS-GLUCOCORTICOIDS, DIURETICS,
    PHENYTOIN, BETA BLOCKERS, AND CALCIUM CHANNEL
    BLOCKERS.
45
Q

HHS s/s

A
  • POLYURIA
  • POLYDIPSIA
  • HYPOVOLEMIA
  • DEHYDRATION
  • HYPOTENSION
  • TACHYCARDIA
  • HYPOPERFUSION
  • WEIGHT LOSS
  • WEAKNESS
  • NAUSEA/VOMITING
  • ABDOMINAL PAIN
  • STUPOR
  • COMA
  • SEIZURES
46
Q

HHS lab findings

A
  • GLUCOSE >600 MG/DL
  • SERUM OSMOLARITY >320
    MOSM/L
  • URINE KETONES NEGATIVE
  • BUN ELEVATED
  • CREATININE ELEVATED
47
Q

HHS interventions

A
  • ADMIT TO ICU IF BLOOD GLUCOSE > 700 MG/DL
  • ESTABLISH, MAINTAIN VENTILATION
  • CORRECT SHOCK WITH ADEQUATE IV FLUIDS NORMAL SALINE IS PREFERRED.
  • 1LITER PER HOUR UNTIL CENTRAL VENOUS PRESSURE BEGINS TO RISE.
  • HALF-NORMAL SALINE FOR OTHERS.
  • IF CLIENT IS COMATOSE, NG SUCTION
  • MAINTAIN FLUID VOLUME
  • ADMINISTER INSULIN TO REDUCE BLOOD GLUCOSE
  • ASSESS MENTAL STATUS AND CONSULT PHYSICIAN IF CHANGES OCCUR.
48
Q

PATIENT TEACHING FOR BOTH DKA
AND HHS

A
  • PATIENT AND FAMILY TEACHING TO PREVENT DKA OR HHS EPISODES:
  • CHECK BLOOD GLUCOSE LEVELS EVERY 4-6 HOURS AS LONG AS SYMPTOMS SUCH AS ANOREXIA, NAUSEA, AND VOMITING ARE PRESENT AND GLUCOSE LEVELS GREATER THAN 250 MG/DL.
    *CHECK URINE KETONE LEVELS WHEN BLOOD
    GLUCOSE LEVELS EXCEED 300 MG/DL
    *PREVENT DEHYDRATION BY MAINTAINING FOOD AND FLUID INTAKE.
    *INSTRUCT PATIENT TO TAKE LIQUIDS CONTAINING BOTH GLUCOSE AND ELECTROLYTES.
  • INSTRUCT THE PATIENT TO CALL PHYSICIAN: CBG GREATER THAN 250 MG/DL THAT DOES NOT RESPOND TO TREATMENT. KETONURIA LASTS FOR MORE THAN 24 HRS, CANNOT TAKE FOOD OR FLUIDS, ILLNESS LASTS MORE THAN 1 TO 2 DAYS.
49
Q

Causes of hyperglycemia:

A

Illness, decreased physical activity, withholding
anti-diabetic drugs, use of corticosteroids, tube feedings, parenteral nutrition

50
Q

Complications of hyperglycemia

A

Reduced immunity, higher infection
rates, longer hospital stays, increased need
for intensive care, greater mortality

51
Q

Hypoglycemia in hospitalized patients

A

HYPOGLYCEMIA IN THE HOSPITALIZED PATIENT BELOW 40 IS ANOTHER RISK FACTOR FOR MORTALITY. IT IS CONSIDERED A “SENTINEL EVENT” BY THE REGULATORY BODIES.
* CAUSES:
INAPPROPRIATE INSULIN TYPE
MISMATCH BETWEEN INSULIN TYPE AND THE TIMING OF FOOD INTAKE
ALTERED EATING PLAN WITHOUT ADJUSTMENTS TO INSULIN DOSAGE.
GIVING SHORT OR RAPID ACTING INSULIN TO PATIENTS NOT EATING.

52
Q

HOSPITAL BLOOD GLUCOSE MANAGEMENT:

A

ESTABLISH TREATMENT PROTOCOLS TO MAINTAIN BLOOD GLUCOSE LEVELS 140-18 FOR CRITICALLY ILL PATIENTS.
CONTINUOUS IV INSULIN SOLUTIONS TO MAINTAIN GLUCOSE.
GLUCOSE TREATMENTS REVIEWED WHEN GLUCOSE LEVELS BELOW 100
GLUCOSE TREATMENTS MODIFIED WHEN GLUCOSE LEVELS BELOW 70
HOLD RAPID OR FASTING ACTING INSULIN FOR NPO
ADMINISTER BASAL INSULIN (LANTUS FOR EXAMPLE) WHEN PATIENT IS NPO

53
Q

BLOOD GLUCOSE MANAGEMENT
FOR SURGERY

A

SURGERY AND ANESTHESIA CAUSE PHYSICAL AND EMOTIONAL STRESS; STRESS RESPONSE RELEASES COUNTER REGULATORY HORMONES (ADRENALINE, NOREPINEPHRINE AND
CORTISOL) SUPPRESS INSULIN ACTION.

54
Q

Preop care for diabetics

A

PREOPERATIVE SCREENING, GLUCOSE MONITORING. SULFONYLUREAS ARE DISCONTINUED 1 DAY BEFORE
SURGERY. METFORMIN IS STOPPED WITHIN 24 HOURS PRIOR TO SURGERY AND STARTED WHEN KIDNEY FUNCTION IS NORMAL.
LONG-ACTING INSULIN MAYBE SWITCHED TO
INTERMEDIATE ACTING 1 TO 2 DAYS PRIOR TO SURGERY. PRE-OP BLOOD GLUCOSE < 200

55
Q

Intra-operative care for diabetics

A
  • REGULAR GLUCOSE MONITORING HOURLY
    TO MAINTAIN VALUES BETWEEN 140 AND
    180 MG/DL
  • IV INFUSION OF INSULIN, GLUCOSE, AND
    POTASSIUM MAYBE NEEDED.
  • MONITOR PATIENT’S TEMPERATURE.
  • MONITOR ARTERIAL BLOOD GASES.
56
Q

Post-op care for diabetics

A

HYPERGLYCEMIA MAY LEAD TO INCREASED
MORTALITY RATES POST SURGICAL. CONTINUE TO MONITOR BLOOD GLUCOSE TO MAINTAIN LEVELS BETWEEN 140-180. SHORT TERM INSULIN THERAPY MAYBE USED FOR THOSE PATIENTS ON ORAL ANTI-DIABETICS. CLOSE MONITOR OF PATIENTS WITH AUTONOMIC NEUROPATHY AND VASCULAR DISEASE. PREVENT HYPOTENSION AND RESPIRATORY
ARREST.
* CLOSE MONITORING OF PATIENTS TAKING BETA BLOCKERS-HYPOGLYCEMIA
* CLOSE MONITORING OF PATIENTS WITH INCREASED BLOOD PROTEIN OR NITROGEN- AT RISK FOR FLUID MANAGEMENT ISSUES.
* PATIENTS ARE AT HIGH RISK FOR CARDIAC AND
RENAL COMPLICATIONS.