W8 Mood and depression Disorders Flashcards

1
Q

What is depression

A

Low mood, sleeping less/more, eating less/more, loss of interest, hopelessness.

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2
Q

How to measure depression

A

Self-report scales

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3
Q

Causes of depression

A

Life events, genetics, chemical imbalance in the brain.

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4
Q

What are the symptoms of depression

A

Cognitive symptoms: difficulty with concentration or making decisions.
Behavioral symptoms: social withdrawal and agitation.
Somatic (physical) symptoms: insomnia or hypersomnia, eating too much too little.
Affective (mood) symptoms: depressed mood and feelings of worthlessness or guilt.

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5
Q

Reactive depression

A

triggered by a negative experience

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6
Q

Endogenous depression

A

no apparent negative life event

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7
Q

Unipolar affective disorder

A

depression

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8
Q

Bipolar affectve disorder

A

depression with periods of mania (energetic, impulsive, positive, very confident). Very cyclic, you might spend months in the mania period and then go through depressive period.

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9
Q

Iproniazid

A

originally developed to treat tuberculosis (didn’t work!) but patients felt less depressed about having tuberculosis.

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10
Q

Mono amine oxidase (MAO)

A

is an enzyme that breaks down monoamine neurotransmitters. MAO inhibitors stop it working. Leaves more neurotransmitter in the neuron so more can be released

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11
Q

What chemical structure is part of tricyclic antidepressants?

A

Imipramine: chemical structure includes a three-ring chain.

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12
Q

Tricuclic antidepressants

A

block reuptake and therefore leaves more transmitter at the synapse.

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13
Q

Prozac

A

blocks reuptake and therefore leave more transmitter at the synapse.

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14
Q

What is used to treat bipolar

A

Lithium: interferes with second messenger system

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15
Q

Monoamine theory of depression

A

Anti-depressants act on monoamines. Hypothesis: Depression is actually caused by a deficit of monoamine neurotransmission

Some evidence of elevated receptors in depressed patients (to compensate for low levels of transmission)

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16
Q

Seligman’s attributional model

A

Attribution of negative events: 3 dimensions are
Internal vs External
Global vs Specific
Stable vs Unstable

17
Q
A
18
Q

ADM (anti-depressant medication)

A

side-effects, no cognitive resilience, addictive

19
Q

Major/Unipolar depression

A

A psychological problem/disorder characterized by relatively extended periods of clinical depression which cause significant distress to the individual and impairment in social or occupational functioning.

20
Q

Bipolar disorder

A

A psychological disorder characterized by periods of mania that alternate with periods of depression.

21
Q

Dysthymic disorder

A

form of depression in which the sufferer has experienced at least 2 years of depressed mood for more days than not.

22
Q

Premenstrual dysphoric disorder

A

condition in which some women experience severe depression symptoms between 5 and 11 days prior to the start of the menstrual cycle. Symptoms then improve significantly within a few days after the onset of menses.

23
Q

Seasonal affective disorder

A

A condition of regularly occurring depressions in winter with a remission the following spring/summer

24
Q

Chronic Fatigue syndrome

A

A disorder characterized by depression and mood fluctuations together with physical symptoms such as extreme fatigue, muscle pain, chest pain, headaches and noise/light sensitivity.

25
Q

Tricuclic drugs

A

Drugs which block the reuptake of both serotonin and norepinephrine.

26
Q

Beck’s Cognitive theory

A

depression could be caused by biases ways of thinking and processing information.

27
Q

Negative schema

A

set of beliefs that tends individuals towards viewing the world and themselves in a negative way.

28
Q

Negative Triad

A

People hold negative view of themselves, of their future and of the world.

29
Q

Attribution theories

A

People who are likely to become depressed attribute negative life events to internal, stable and global factors

30
Q

Hopelessness theory

A

individuals exhibit an expectation that positive outcomes will not occur, negative outcomes will occur, and that the individual has no responses available that will change this state of affairs.

31
Q

Cyclothymic disorder

A

A form of depression characterized by at least 2 years of hypomania symptoms that do not meet the criteria for a manic episode and in which the sufferer experience alternating periods of withdrawal then exuberance, inadequacy and then high self-esteem.

32
Q

Olanzapine

A

Antipsychotic drug commonly prescribed in combination with antidepressant SSRI drug Fluxetine as a treatment for bipolar disorder.

33
Q

Fluxetine Prozax

A

A selective serotonin reuptake inhibitor (SSRI) which reduces the reuptake of serotonin in the brain and is taken to treat depression.

34
Q

Electroconvulsive theory

A

Method of treatment for depression/psychosis, 1930s, involves passing and electric current of around 70-130 volts through the head of the patient for around half a second.

35
Q

Stepped care models

A

treatments for psychopathology that emphasise that the type of treatment provided for those individuals should be tailored to the severity of their symptoms and their personal/social circumstances.

36
Q

Stepped care Models

A

treatments for psychopathology that emphasise that the type of treatment provided for those individuals should be tailored to the severity of their symptoms and their personal/social circumstances.

37
Q

Social skills training

A

theory for depression that assumes depression is partly resulting from an individual’s inability to communicatee and socialized appropriately and the addressing these deficits should help to alleviate the symptoms.

38
Q

Behavioural activation theory

A

a therapy for depression that attempts to increase clients’ access to pleasant events and rewards and decrease their experience of aversive events and consequences.