W1 - MSK inert Structures Flashcards

1
Q

What does FAI stand for

A

Femoral acetabulum impingement

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2
Q

What is an FAI

A

Pathological mechanical process where there is a morphological abnormality of the acetabulum or femur and vigorous hip motion that damages soft tissue structures around the hip

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3
Q

What are the 3 types of FAI

A

Cam lesion = abnormality to the anterior, lateral femoral head/neck (non-spherical head shape)

Pincer lesion = abnormality to the acetabulum causing an over coverage of the femoral head shape

Combined lesion = cam & pincer at the same time

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4
Q

What are the 3 main causes for a FAI

A
  1. Exposure to repetitive hip flexion & rotation during childhood development as a result of a high impact activity (sport). This triggers adaptive remodelling of the hip
  2. History of childhood hip disease following a femoral neck fracture as this alters the contour of the femoral head/neck
  3. Surgical over correction (hip dysplasia) can cause a pincer FAI
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5
Q

What is a type of ligament injury

A

Sprain

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6
Q

What is a sprain

A

Injury to the band of collagen tissue due to being suddenly forced outside its usual range of movement and the inelastic fibres are stretched through too greater range

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7
Q

What is the primary function of a ligament

A

Provide passive stabilisation of a joint

Plays an important role in proprioceptive function

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8
Q

What is a grade I ligament tear

A

Grade I = overstretched with micro-tears. Localised pain/tenderness. No visible bruising. Minimal swelling. Minimal loss of function. No loss of muscle strength & no ligament laxity

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9
Q

What is a grade II ligament tear

A

Partial tear with immediate onset of all inflammatory signs. Moderate swelling. Raising. Poorly localised pain. Impairment and painful ROM. Decrease in muscle strength and pain on contraction. Joint may be unstable

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10
Q

What is a grade III ligament tear

A

Complete rupture. I ability to contract the muscle. Separation may be evident. Immediate acute pain. Pop, crack or click sound. Cardinal signs. Later symptoms become less then a grade II and may require immobilisation or surgery

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11
Q

Non modifiable factors of a ligament injury

A

Intrinsic knee anatomy
Sex and hormones
Joint laxity

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12
Q

Modifiable factors that effect ligament injuries

A

Neruromuscular control = strength & Proprioception

Environment = sport specific skills, fatigue resistance

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13
Q

What is a frozen shoulder

A

Adhesive capulitis has initial pain and later progressively restricts active & passive glenihumeral joint ROME with spontaneous complete or nearly-complete recovery over a period of time

This inflammatory condition causes fibrosis of the GH joint capsule causing gradual stiffness & significant restricted ROM (external rotation)

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14
Q

What part of the shoulder does a frozen shoulder effect

A

Anterior, superior joint capsule
Axillary recess
Coracohumeral ligament

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15
Q

What causes frozen shoulder

A

Synovial inflammation followed by capsular fibrosis in which type 1 & 3 collagen is laid down with subsequent tissue contraction

Elevated levels of serum sytokines facilitate tissue repair & remodelling during the inflammatory process

An imbalance between aggressive fibrosis & loss of normal collagenous remodelling leads to the stiffness of the capsule and ligament structure

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16
Q

Who’s more likely to have a frozen shoulder

A

Females
35-65 year olds
Diabetics
People who have had a previous frozen shoulder

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17
Q

What are the 3 phases of a frozen shoulder

A
  1. Acute/freezing/painful phase
  2. Adhesive/frozen/stiffening phase
  3. Resolution/thawing phase
18
Q

What happens in the acute/freezing/painful phase of a frozen shoulder

A

Gradual onset of shoulder pain at rest
Sharp pain at extremes of motions
Painful at night and interrupts sleep
Last 2-9 months

19
Q

What happens in the adhesive/stiffening/ frozen phase of a frozen shoulder

A

Pain subsides
Progressive loss of GH motion in capsular pattern
Pain apparent only at extremes of motions Painful
Lasts 4-12 months

20
Q

What happens in the resolution/thawing phase of a frozen shoulder

A

Spontaneous progressive improvement in ROM
can last between 5-24 months and
Can last up to 3 years (self limiting condition)
15% of people will also have long term disability and persistent pain

21
Q

What is osteoarthritis

A

Chronic conditions which involves the break down of cartilage and eventually bone on bone rubbing

22
Q

Risk factors of osteoarthritis

A

Age
Female gender
Obesity
Anatomical factors
Muscle weakness
Joint injury

23
Q

What are the 2 types of OA

A

Primary = due to age
Secondary = specific trigger (previous injury)

24
Q

Clinical signs and symptoms ton of OA

A

Pain = when weight bearing and doing activities
Reduced ROM
Slight swelling
Clicking/grindimg

25
Q

What in a meniscal issue

A

Meniscal tears due to excessive force applied to a normal meniscus OR normal force applied to a degenerative meniscus

Commonly occurs in a twisting injury on a semi lexed limb through a weight bearing knee

26
Q

What are the 2 types of meniscus tears

A

Acute = result of trauma or sporting injury. Can have different shapes (horizontal, vertical, radial, complex). Treated with surgery in conservative management in ineffective

Degenerative = common in elderly. Minimal trauma to the knee. Treated with physio and anti-inflammatories

27
Q

What does PFPS stand for

A

Patellafemoral pain syndrome

28
Q

What is PFPS

A

Umbrella term for pain in the patellofemoral joint or adjacent soft tissue

Can be acute or chronic that’s characterised by overload

Worsens with activity (squatting, sitting, climbing)

29
Q

What causes PFPS

A

Combination of factors:
Overuse & overload of the joint
Anatomical or bio mechanical abnormalities
Muscular weakness
Imbalance or dysfunction
Main cause = patella orientation and alignment

30
Q

How does patella orientation and alignment cause PFPS

A

Different orientations cause it to glide more to one side of the femur causing overuse/overload on that part of the femur causing pain, discomfort or irritation

31
Q

What are the risk factors for PFPS

A

Knee hyperextension
Lateral tibial torsion
Genu Valgum of varus
Increased Q angle (women)
Tight iliotibial band, hamstring or gastrocnemius
Pronation or supination can provoke PFPS

32
Q

How would you manage PFPS

A

Education
Open vs closed chain exercises
Quads, hamstrings, glutes and calf strengthening
Patellar taping
Orthotics
Modalities
Manual therapy

33
Q

What causes shoulder instability

A

When the shoulder labrum and/or ligaments are stretched or teared leading to a greater chance of dislocations or subluxation (front, back or bottom)

34
Q

What is a bankart lesion

A

When the labrum is torn from the bone and results in an unstable shoulder leading to further episodes of dislocation

35
Q

How do you treat a bankart lesion

A

Surgical procedure called an anterior stabilisation

36
Q

What does an ALPSA lesion stand for

A

Anterior labral periosteal sleeve avulsion

37
Q

What is an ALPSA

A

Displaced bankart tear where the labrum has displaced around the glenoid neck

Associated with higher risk of recurrent instability then an undisplaced bankart tear where

38
Q

What is a HAGL tear

A

Humeral avulsion of glenohumeral ligament

39
Q

What is a bony bankartt

A

Fragments of bone break off with a bankart tear where

40
Q

What is a hill sachs lesion

A

Dent in the back of the humeral head that occurs during dislocation as the humeral head impacts against the front of the glenoid

41
Q

What is a slap tear

A

A tear at the bottom of the labrum