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PTH 603 - Patho for Neuro > Vestibular Disorders > Flashcards

Vestibular Disorders Flashcards

1
Q

What makes up the central portion of the Vestibular System?

A
  • 4 vestibular nuclei
  • Cortex/Thalamus
  • Cerebellum
  • Tracts connecting to the muscles and nerves, reticular formation
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2
Q

What makes up the peripheral portion of the Vestibular System?

A
  • Bony Labyrinth
  • Membranous Labyrinth
  • 5 Sensory Organs
  • Receptors
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3
Q

What do the 3 semicircular canals do?

A

Detect angular acceleration

-Mediates dynamic functions

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4
Q

What do the 2 Otolithic Organs do?

A

Mediate STATIC FUNCTIONS

Saccule and Utricle

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5
Q

What does the Saccule do?

A

Detect VERTICAL translation of the head

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6
Q

What does the Utricle do?

A

Detect Horizontal translation of the head

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7
Q

What is innervated by the Superior Division of Cranial Nerve VIII?

A
  • Utricle
  • Anterior Semicircular Canal
  • Horizontal Semicircular Canal
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8
Q

What is innervated by the Inferior Division of Cranial Nerve VIII?

A
  • Saccule

- Posterior Semicircular Canal

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9
Q

What is the entrance and exit of CN VIII? (Sensory only)

A

Stemming from the lateral inferior margin of the left and right side of the pons and the cerebellum, exiting the skull via the internal acoustic meatus.

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10
Q

Role of Vestibular System?

A

SENSORY and MOTOR System

  • Provides awareness of spatial orientation
  • Helps influence muscle tone and balance
  • Controls the position of the eye in orbit

Input –> Integration –> Perception –> Selection of Motor –> Refinement –> Output

Positional information for visual, vestibular, and proprioceptive input go through central processing in cerebellum and reticular system. The inputs are influenced and controlled by eye movements and posture & motor skills.

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11
Q

What is responsible for input?

A

Somatosensory, Labyrinths, and Vision

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12
Q

What is responsible for Integration?

A

Vestibular Nuclei and Cerebellum

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13
Q

What is responsible for Perception?

A

Cortex and Thalamus

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14
Q

What is responsible for Selecting Motor Responses?

A

Cortex

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15
Q

What is responsible for Refinement of Movement?

A

Vesibular nuclei and cerebellum

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16
Q

What is responsible for Output?

A

Vestibular spinal, vesibular occular, occular tilt response

17
Q

What is the Push-Pull Mechanism?

A

Each Canal has a mate on the opposite side that lies in the same plane.

When your head is rotated, the cupula of the horizontal canal on the side you turn towards is facilitory, causing an increased rate of firing of Scarpa’s ganglion to the vestibular nuclei.

On the opposite side (the side you are turning away from), the cupula is inhibitory, causing Scarpa’s ganglion to SLOW is rate of fire to that vestibular nuclei

(Scarpa’s Ganglion = Constanty firing at 100 spikes/sec. at rest)

–

Head move R, Fluid move L (excite side going towards)

When one canal is stimulated, its corresponding partner on the other side is inhibited, and vice versa. This push-pull system allows us to sense all directions of rotation: while the right horizontal canal gets stimulated during head rotations to the right (Fig 2), the left horizontal canal gets stimulated (and thus predominantly signals) by head rotations to the left.

18
Q

What are some key concepts of nystagmus?

A

Involuntary rhythmic oscillation of the eyes

  • Slow phase - generated by the vestibular system
  • Quick phase - generated by the saccade system

Named for QUICK PHASE
Causes retinal slip, giving the illusion of movement

Horizontal, Vertical, or Torsional movements

  • Direction of torsional nystagmus is identified by the direction that the superior pole of the eye is moving towards
  • Most of the time, you see a combo of the 3

Anterior canal = down-beat nystagmus
Nystagmus is normal in supine in the dark
Named by the direction of the FAST-PHASE

19
Q

What are key concepts of Spontaneous Nystagmus?

A

Vestibular hypofunction - fast phase of nystagmus beats towards the strong and unaffected side (eye beats R, means L side is weak - may be opposite if a virus is involved)

Alexander’s Law: Gaze in direction of fast component causes increased frequency, gaze away from fast component has opposite effect.

Usually a peripheral lesion (can suppress with fixation)

20
Q

What are key concepts of Central Nystagmus?

A

VERTICAL is often CENTRAL in origin

Purely vertical, horizontal, or torsional is central (pure=central)

UNABLE TO SUPPRESS WITH FIXATION

(Torsion + Downbeat = Peripheral) - combo is rare?

21
Q

What is “vertigo?”

A

Sensation of movement of oneself or of the environment (rotation, translation, tilt)

-Sensation that the room is spinning (usually over-diagnosed)

22
Q

What is “dizziness?”

A

Non-specific catch all

Have the patient use a different term

23
Q

What is a “Head Fog?”

A

Lightheadedness (pea soup in head?)

24
Q

What is dysequilibrium?

A

Static and Dynamic unsteadiness

25
Q

Temporal Characteristics of Clinical Presentation?

A

Frequency, Duration (constant, episodic, seconds, minutes)

26
Q

What provokes symptoms?

A

Spontaneous

  • Head position
  • Specific movement
  • Environment (light and sound)
  • Fatigue
27
Q

Auditory Characteristics of Clinical Presentation?

A
  • Hearing loss

- Tinnitus (ringing in the ears - high-pitched, does not go away)

28
Q

What are the eight different differential diagnoses discussed in class?

A
  1. Vestibular Labyrinthitis and Neuritis
  2. Bilateral vestibulopathy
  3. Meniere’s Disease
  4. Vestibular Migrane
  5. Cervical Vertigo
  6. Acoustic Neuroma
  7. Superior Semicircular Canal Dehiscence or Perilymphatic Fistula
  8. Benign Paroxysmal Positional Vertigo

(will be briefly discussed in future notecards. Consult Rijo or PowerPoint for more details)

29
Q

What is Vestibular Labyrinthitis and Neuritis?

A

Viral infection (Herpes Zoster - Chicken Pox)
-Patholoy to vestibular nerve
_Balance, gait, gaze all affected
Can last for 1-4 days?

30
Q

What is Bilateral vestibulopathy?

A

Pathology to bilateral labyrinth/nerve
Ototoxic antibiotics or Chemotherapy agents
Partial or complete neuritis
Increased dysequilibrium and decreased c/o dizziness
Oscillopsia; “bouncing vision”
Rotory Chair - most reliable diagnostic test for incomplete bilateral vestibulopathy
-Use antihistamines or antiemetic (1-3 days)
-Vestibular rehab to spur compensation
-Steroids for early hearing loss
-Suppressive meds for acute symptoms (may interfere with vestibular rehab)

CANNOT DRIVE AT NIGHT

31
Q

What is Meniere’s Disease?

A

Five characteristics
1. Episodic True spinning vertigo (TSV)
2. Fluctuating Hearing Loss (unilateral - on affected side)
3. Tinnitus (roaring, LOW freq)
4. Progressive Low Frequency hearing loss
5. Aural Fullness
(diagnosis made over time, 3 episodes)

Treatment:

  • Low Sodium diet
  • Diuretics
  • Vestibular rehab
  • Gentamicin ablation (last resort)
32
Q

What are Vestibular Migranes?

A
  • Usually without headaches!
  • Disorder of electrical activity of brain
  • Visual aura preceding attacks
  • Set off by triggers
  • Treat as you would a treat a Migraine Headache

Diagnostic criteria:

  1. Episodic vertigo
  2. One migraine symptom during 2 vertigo attacks
  3. Often other central involvement presentation

Treatment:
Diet, exercise, regular sleep, hydration
-Avoid triggers
Meds:
-Abortive Triptans; Imitrex; Maxalt; Zomig)
-Preventative (Amitriptyline, Inderal, nSAIDs)`

33
Q

What is Cervical Vertigo?

A

Non-specific sensation of altered orientation in space originating from abnormal afferent activity from the neck. Mismatch between vestibular, visual, and proprioceptive input. Many causes

DIAGNOSIS OF EXCLUSION

  • Imbalance TEMPORARILY related to neck pain
  • All other possible causes of dizziness eliminated

Treat the neck
-Sustained natural apophyseal glides (SNAGs) are an effective treatment for cervicogenic dizziness.

34
Q

What is Acoustic Neuroma?

A

Lesion site = vestibular portion of CN VIII, Cerebellopontine Angle (space occupying lesion)

Presents with progressive, ASYMMETRICAL, sensorineural hearing loss, tinnitus (3rd most common intercranial tumor)

Treatment: Watchful waiting, microsurical removal, sterotactic Radiosurgery, vestibular rehab (if abnormal balance or VOR)

35
Q

What is Superior Semicircular Canal Dehiscence or Perilymphatic Fistula?

A

Perilymphatic Fistula = Hole or leak in the round window

Superior Canal Dehiscence = Hole or thinning of bone between cranium and anterior SCC

Hx: barotrauma, pressure changes, head trauma
Pt c/o: symptoms with coughing, sneezing, straining
Hearing own voice, footseps as unusually loud

Fistula Pressure Test, Tragus Testing, Valslva with blocked glottis or nostrils
Looking for mixed vertical or torsional nystagmus
Dx with High Resolution CT Scan of temporal bone

Med Mangement: Surgical repair, loud sound management, vestibular rehab pending abnormal balance and VOR testing (conductive vs sensorineural)

36
Q

What is Benign Paroxysmal Positional Vertigo?

A

MOST COMMON vestibular disorder - cause of “dizziness”
Brief episodes of vertigo, lasting seconds
-Provoked by specific head positions
Incidence is 64 in 100,000
75% happen in those over 40.

Canalithiasis - Otoconia displaced from utricle free floating in canals (nystagmus less than 60 seconds)

Cupulolithiais - Attached to cupula (more than 60 seconds)

-Usually robust and vigorous nystagmus
Latency of 3-10 seconds

37
Q

What are the 3 types of BPPV?

A

Posterior Semicircular Canal:

  • 75% of all BPPV
  • Dix-Hallpike Test
  • Up beating, torsional nystagmus

Anterior Semicircular Canal:

  • Dix-Hallpike Test
  • Down beating, torsional nystagmus

Horizontal Semicircular Canal:

  • Horizontal Roll Test
  • Horizontal Nystagmus
38
Q

Management of BPPV?

A

Anterior/Posterior Canalithiasis:

  • Canalith Repositioning maneuvers (Epley)
  • Habituation Manuevers

Anterior/Posterior Cupulolithiasis:
-Liberatory Manuever

Horizontal Canalithiasis/Cupulolithiasis:
-Repositioning Manuevers (BBQ Roll)

PTH 603 - Patho for Neuro (7 decks)

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