Vertigo/ dizziness Flashcards

1
Q

Define vertigo

A

Vertigo is the sensation that the environment is spinning around relative to oneself (objective vertigo) or vice versa (subjective vertigo).

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2
Q

Pathophysiology of BPPV?

A

presence of debris in the semicircular canals of ears cause vertigo upon head movement

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3
Q

Diagnostic test of BPPV?

A

Dix-hallpike manœuvre- rotatory nystagmus brings on symptoms

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4
Q

What resolves BPPV?

A

Epley manœuvre

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5
Q

How does BPPV present?

A

Vertigo on turning head
Gradual onset
Episodes last 10-20 secs

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6
Q

What is acute labyrinthitis?

A

Inflammation of the vestibular nerve causes acute severe vertigo, may be associated with vomiting/

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7
Q

How does acute labyrinthitis present?

A

Recent viral infection
Vertigo
N&V
hearing may be affected/tinnitus

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8
Q

Vestibular neuritis presentation?

A

Recent viral infection
recurrent vertigo attacks lasting hours or days
Nausea and vomiting
Horizontal nystagmus
No hearing loss

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9
Q

How do you differentiate between vestibular neuritis and acute labyrinthitis?

A

Vestibular neuritis will not have any hearing loss but acute labyrinthitis might

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10
Q

Treatment for acute labyrinthitis?

A

Self-resolving in about a month, conservative treatment.
prochlorperazine or antihistamines may help reduce the sensation of dizziness

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11
Q

Meniere’s disease features?

A

Recurrent episodes of vertigo, sensorineural hearing loss, tinnitus and feeling of aural fullness
Can be unilateral or bilateral

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12
Q

Treatment of Menieres disease?

A

Antihistamines and bedrest

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13
Q

Presentation of vestibular schwannoma?

A

The classical history of vestibular schwannoma includes a combination of:
* vertigo
* hearing loss
* tinnitus
* absent corneal reflex.

Features can be predicted by the affected cranial nerves:
* cranial nerve VIII: vertigo, unilateral sensorineural hearing loss, unilateral tinnitus
* cranial nerve V: absent corneal reflex
* cranial nerve VII: facial palsy

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14
Q

What condition is vestibular schwanoma associated with?

A

Neurofibromatosis type 2

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15
Q

What is Ramsay Hunt syndrome?

A

Herpetic infection of the facial N–> causes facial nerve palsy
With or without vertigo, tinnitus and hearing loss

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16
Q

How do you treat Ramsay Hunt?

A

Aciclovir and prednisolone

17
Q

What causes ototoxicity?

A

Aminoglycoside abs
Loop diuretics

18
Q

Features of carbon monoxide poisoning?

A

Headache
Nausea and vomiting
Vertigo
Confusion
Subjective weakness

In severe toxicity: ‘pink’ skin and mucosae, hyperpyrexia, arrhythmias, extrapyramidal features, coma, death

19
Q

Investigations in carbon monoxide poisoning?

A

Pulse ox may be FALSELY high
VBG/ABG
ECG- for cardiac ischaemia

20
Q

What are typical carboxyhemoglobin levels?

A

< 3% non-smokers
< 10% smokers
10 - 30% symptomatic: headache, vomiting
> 30% severe toxicity

21
Q

Management of carbon monoxide poisoning?

A

100% high-flow oxygen via a non-breath mask, target sats are 100%
Treatment until all symptoms have resolved

22
Q

Indications of hyperbaric oxygen use in carbon monoxide poisoning?

A

Discussion with specialist should be considered in more severe cases: >25%

LOC
Neuro signs other than headache
Myocardial ischaemia or arythmie
Pregnancy

23
Q

What is a TIA?

A

a transient episode of neurologic dysfunction caused by focal brain, spinal cord, or retinal ischaemia, without acute infarction.

24
Q

Features of TIA?

A

Usually resolve within one hour

Unilateral weakness or sensory loss
aphasia/dysarthria
Ataxia, vertigo, loss of balance
Visual problems (amaurosis fugax), diplopia, homonymous hemianopia

25
Q

Management of Vestibular neuronitis?

A

buccal or intramuscular prochlorperazine is often used to provide rapid relief for severe cases
a short oral course of prochlorperazine, or an antihistamine (cinnarizine, cyclizine, or promethazine) may be used to alleviate less severe cases
vestibular rehabilitation exercises are the preferred treatment for patients who experience chronic symptoms

26
Q

What is essential to determine about the cause of vertigo when assessing a pt? WHy?

A

Whether the vertigo is due to a central or peripheral cause.

why? cerebellar infarction / haemorrhage (central) is life threatening as opposed to non life threatening e.g. BPPV or mernieres (peripheral)

27
Q

What are DDx for a patient presenting with acute vertigo i.e. causes of vertigo

A

Mechanical
- BPPV (otoliths moving in semicirucalr canals)
- Menieres

Infectious (viral)
- Acute vestibular neuritis (semicircular canal function)

Vascular :
- Infarction in anterior vestibular artery territory (presents like v. neuritis - think pts with RF for stroke e.g. DM, HTN, AF)

  • Brainstem stroke (accompanied by other signs, horners, dysarthuria, incoordination, diplopia, facial numbness)
  • Inferior cerebellar artery infarction (present only with vertigo, nystagmus and postural instability)

MS
- plaque around VIII cranial nerve

Migraine
- diagnosis of exclusion

Migrainous vertigo

28
Q

what are clinical features of acute vertigo ?

A

vertgio
nausea
vomitting

29
Q

Compare clincial features of BPPV vs Menieres vs migraine

A

BPPV vertigo lasts for seconds

Meniere’s vertigo lasts for mins/hours

Migraine vertigo last for hours

30
Q

compare central vs peripheral vertigo charachteristics

A
31
Q

What examination can you do to determine if peripheral or central origin of vertigo?

A

HINNTs exam

involves:
Head impulse test
evaluation of nystagmus
test of skew

https://www.youtube.com/watch?v=1q-VTKPweuk