Vertigo Flashcards

1
Q

What is vertigo?

A
  • vertigo is an illusion of movement
  • includes sensation of rotation of self or of the environment as well as sensations of being pulled downwards/sideways or the room tilting - ie. ‘spinning’
  • always worsened by movement
  • often associated w/ difficulty in walking or standing with relief on lying or standing still
  • other common symptoms → nausea, vomiting, pallor, sweating
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2
Q

Dizziness is a generic term that may refer to lightheadedness, faintness, giddiness, swimming or floating sensation, unsteadiness, imbalance, mental confusion, minor seizures or even true vertigo.

What is the impact of dizziness?

A
  • 12x more likely to fall
  • falls commonest cause of accidental death in over 75s
  • psychological → dizziness increases anxiety + depression which interferes w/ recovery, reluctance to go out as unsteadiness mimics drunkenness
  • socio-economic → not able to go back to work, may not be able to do certain types of work, not able to drive in certain conditions, poor QoL
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3
Q

Causes of vertigo

A
  • Otological → meniere’s disease, vestibular neuronitis, labyrinthitis, BPPV, cerumen impaction, perilymphatic fistula, trauma, infection
  • Central → CN VIII nerve disorders, MS, stroke, haemorrhage, migraine, acoustic neuroma, malignancy, trauma, infection, epilepsy
  • Other → DM, hypoglycaemia, alcohol, anaemia, dysrhythmias, iatrogenic, multisensory disease syndrome
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4
Q

Peripheral vertigo (85%) is the result of a problem with your inner ear or CN VIII, which controls balance. Central vertigo refers to problems within your brain or brainstem.

How do these present differently?

A
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5
Q

What is physiologic vertigo?

A
  • “motion sickness”
  • mismatch between visual, proprioceptive and vestibular inputs
  • not a diseased cochleovestibular system or CNS
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6
Q

What is Benign Paroxysmal Positional Vertigo (BPPV)?

A

Most common cause of vertigo (a peripheral cause)

Acute attacks of transient vertigo lasting seconds-mins initiated by certain head position accompanied by torsional (rotatory) nystagmus

​Commonly occurs on getting out of bed / looking up / rolling

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7
Q

What is the Dix-Hallpike maneouvre?

A
  • rapidly moving pt from a sitting position to supine position
  • w/ head hanging over end of table, turned 45o to one side
  • hold for 15-20s to elicit nystagmus
  • onset of vertigo + rotatory nystagmus indicate positive test for dependent side
  • top pole of the eyes rotates towards the undermost (affected) ear
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8
Q

What is the pathophysiology BPPV?

A

Otoliths

Tiny crystals of calcium carbonate (normal part of inner ear) detach from otolithic membrane in utricle + collect in one of the semicircular canals (most commonly posterior)

Head still → gravity causes otoliths to clump + settle

Head moves → otoliths shift → stimulates cupula to send false signal to brain → vertigo and nystagmus occur

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9
Q

Treatment for BPPV

A
  • reassure patient that process resolves spontaneously
  • particle repositioning maneouvres
    • main aim → reposition the otoliths back to utricle
    • 3-step PRM / Epley manouvre
    • Semont manouvre
    • Brandt-Daroff exercise (by patient)
  • surgery for refractive causes
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10
Q

What is vestibular neuritis?

A
  • inflammation of the nerve - affects branch associated w/ balance, resulting in dizziness or vertigo but no change in hearing
  • sudden onset
  • only vestibular symptoms + worst symptoms in the beginning
  • prodromal viral URTI may be present
  • symptomatic recovery by central compensation
  • recovery influenced by age, vision, proprioception, mental health + medications
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11
Q

What is labyrinthitis?

A
  • commonly used to denote vestibular neuritis
  • labyrinth is structure in inner ear, consisting of semicircular canals, vestibule and cochlea
  • it is inflammation of the labyrinth
  • occurs when an infection affects whole structure of inner ear (labyrinth)
  • affects both vestibular apparatus AND cochlea
  • results in hearing changes as well as dizziness or vertigo
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12
Q

Management of labyrinthitis

A
  • Vestibular sedatives (benzos) should be used only for short duration
    • act by inhibiting normal side to reduce asymmetry
    • hence it interfered w/ central compensation
    • may prolong recovery from symptoms
  • vestibular rehabilitation exercises mainstay of treatment
  • anti-emetics for N+V
  • sudden hearing loss → steroids
  • IV antibiotics if bacterial
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13
Q

What is Meniere’s disease?

A
  • AKA endolymphatic hydrops
  • idiopathic, XS endolymphatic fluid pressure
  • triad of fluctuating low freq hearing loss, severe vertigo + roaring tinnitus
  • RFs → high salt intake, caffeine, stress, nicotine, alcohol
  • recurrent episodes, initial episodes usually worse
  • preceding aural fullness common
  • occasionally bilateral
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14
Q

Investigations for Meniere’s disease

A
  • pure-tone air + bone conduction w/ masking
  • speech audiometry
  • tympanometry
  • oto-acoustic emissions
  • MRI internal auditory canals
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15
Q

Management of Meniere’s disease

A
  • life-style changes → limiting salt + caffeine
  • MEDICAL:
    • symptomatic relief of an acute attack → cinnarizine, prochloperazine, cyclizine
    • prophylactic preventive → betahistine, diuretics (bendroflumethiazide)
  • SURGICAL:
    • trans-tympanic treatment → gentamicin, steroids
    • endolymphatic sac surgery
    • vestibular neurectomy
    • labyrinthectomy
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16
Q

What is an acoustic neuroma (vestibular schwanomma)?

A
  • schwannoma of the vestibular portion of CN VIII
  • acoustic neuroma most common intracranial tumour causing SNHL + most common cerebellopontine angle tumour
  • starts in internal auditory canal + expands into cerebellopontine angle (CPA), compressing cerebellum and brainstem
  • presentation → unilateral SNHL or tinnitus, dizziness and unsteadiness may be present but true vertigo rare as tumour growth slow + compensation occurs
  • facial nerve palsy + trigeminal (V1) sensory deficit (corneal reflex) are late complications
17
Q

How is acoustic neuroma diagnosed?

A
  • DIAGNOSIS
    • MRI w/ gadolinium
    • audiogram
    • poor speech discrimination relative to hearing loss
    • stapedial reflex absent or significant reflex decay
    • ABR - increase in latency of 5th wave
    • vestibular test → normal or asymmetric caloric weakness
18
Q

Causes of BPPV

A

Idiopathic (50%)

Head injury

Whiplash

Post-vestibular neuritis

19
Q

Risk factors for Meniere’s disease

A

High salt intake

Caffeine

Stress

Nicotine

Alcohol

20
Q

Management of acoustic neuroma

A

Surgical exicison

Other → gamma knife, radiation