Valves Flashcards
Splitting of S2
Physiological
Parodoxical
during inspiration, A2 before P2, increased venous return delays P2, decreased return to the L quickens A2
during expiration, delayed closure of the aortic valve (AS, HOCM, LBBB)
Persistent S2 splitting
splitting throughout the respiratory cycle, increased during inspiration
early A2 or delayed P2 (severe MR/VSD or RBBB, pulm htn, pulmonic stenosis)
Fixed S2 splitting
always split, not increased in inspiration
delayed P2 only (increased flow across valve)- ASD
mitral stenosis in American elderly
senile calcific MS
calcification of mitral annulus and leaflets-> narrow annulus, rigid leaflets, no commissural fusion
no role for percutaneous mitral balloon or surgical commissurotomy
delay intervention until symptoms are severely limiting
treat with diuresis and HR control
causes of MS
rheumatic heart disease
senile calcific MS
congenital disease (parachute mitral valve, mitral chordae attached to a single or dominant papillary muscle-> a component of the Shone complex: supramitral rings, valvular or subvalvular AS, and aortic coarctation)
tumors, obstruction
treatment of acute severe MR
surgery
temporize with diuresis and afterload reduction (vasodilator->nitroprusside, ACEI/ARB/ARNI/nifedipine, IABP)
Do not use vasoconstrictors
prominent v waves
TR
brief, high-pitched sound after S2 followed by a low-pitched rumble, best heard at the apex at held expiration.
opening snap with MS
Percutaneous balloon mitral commissurotomy vs surgical
preferred as long as the valve is mobile, relatively thin, and free of calcium and there is no left atrial clot or more than mild mitral regurgitation (MR)
TEE before to eval MR and r/o clot
surgery if perc is CI+ low-intermediate risk for surgery+ severe MS
exercise testing with doppler or invasive hemodynamics in MS when
discrepancy between resting Doppler echocardiographic findings and clinical symptoms or signs.
Percutaneous mitral balloon commissurotomy indicated in
symptomatic severe MS or asymptomatic with pulmonary hypertension, moderate or severe stenosis and favorable valve morphology in the absence of left atrial thrombus or moderate to severe mitral regurgitation.
meds are preferred if preggo
high gradient+ small EOA
functional-> patient prosthesis mismatch
pathologic (thrombus/pannus)
prosthetic valve obstruction
indexed EOA <0.65, acceleration time < 100ms, DVI>0.25
acceleration time > 100 ms
flushing, diarrhea, hypervolemia, elevated 5-HIAA
carcinoid heart
carcinoid
TR, hepatic vein flow reversal if severe TR
Causes of primary TR
Ebstein’s (apical displacement of leaflets >8 in comparison to mitral leaflets)
endocarditis
implanted devices (eccentric, restricted septal leaflet)
carcinoid (thickened leaflets)
radiation
rheumatic heart disease
Systolic notching of the pulmonic valve
pulm htn
Pulmonary vein flow reversal
severe MR
severe MS
MVA <1.5, T1/2> 150ms
warfarin preg
if <5 mg, can continue
switch to UFH for delivery
if >5, discontinue in first trimester for any heparin
medical mgmt of MS
diuretic, beta blocker
apex is HYPERDYNAMIC, grade 2/6 systolic murmur and grade 2/4 diastolic murmur along the left sternal border
m-mode
AR
fluttering of anterior mitral leaflet, early closure of mitral valve due to increased LV pressure in diastole
chronic vs acute AR
wide pulse pressure
treatment of severe AR
surgery
DO NOT USE IABP OR BETA BLOCKERS (tachycardia is appropriate) OR phenylephrine (increases afterload)