Valve disease & Cardiac Inflammatory conditions Flashcards

1
Q

5 questions on inflammatory and valve disease on exam 3

A
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2
Q

what is the pericardium ?

A

protect the heart, it covers the heart

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3
Q

what does pericarditis mean ?

A

inflammation of the pericardium causing pericardium to be less elastic and compress the heart

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4
Q

what is pericarditis mainly caused by ?

A

bacteria or virus

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5
Q

pericarditis is often progressive, frequent severe chest pain but worsen when ?

A

inspiration and when lying flat
( deep breath and learning forward )

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6
Q

why do you think patients who have pericarditis avoid taking deep breaths and leaning forward?

A

youre filling your chest, which puts pressure on the heart

and when you lean forward, heart moves and rubs against your chest wall

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7
Q

what are some clinical manifestations of pericarditis ?

A

dyspnea
fever - its an infection
chills
fatigue

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8
Q

what are you going to hear in pericarditis ?

A

pericardial friction rub

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9
Q

if pericarditis is chronic, you may have what ?

A

right ventricular failure - goes into the pitting edema

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10
Q

how do we manage pericarditis ?

A

high fowlers
blood culture - infection
oxygen

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11
Q

what is the medication for pericarditis ?
and why ?

A

NSAIDS or steroids
- this an inflammatory conditions

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12
Q

what is cardiac tamponade?

A

the entire heart fills up with fluid, remember its like drowning

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13
Q

cardiac tamponade is an emergency, so what are some procedures we are going to do in order to help aid all the fluid build up ?

A

pericardiocentesis
pericardial window
pericardectomy

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14
Q

when youre listening for heart sounds, what is it going to sound like in a patient with cardiac tamponade?

A

muffled heart sounds

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15
Q

what are some medications we are going to give to patients with cardiac tamponade ?

A

NSAIDS

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16
Q

what is infective endocarditis ?

A

disease of the endocardium, including the heart valves - usually aortic and mitral

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17
Q

how does infective endocarditis come to be?

A

bacteria enters blood stream, adheres to endocardium and valves, with previous damage and develops into vegetation which can become emboli

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18
Q

left sided vegetation can move to where ?
for infective endocarditis

A

brain, kidneys, spleen

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19
Q

right sided vegetation can move to where?
for infective endocarditis

A

lungs (pe)

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20
Q

notes
some risk factors for infective endocarditis
- age
- prosthetic valves
- hemodialysis
- iv drug abuse
- iv access/infection
-hemodialysis

tongue piercing !

A
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21
Q

what are some clinical manifestation of infective endocarditis ?

A

clubbing - chronic
fever
chills
weakness
malaise
back pain
weight loss

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22
Q

typically in patients who have infective endocarditis, typically you will hear a new or worsening what?

A

systolic murmur

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23
Q

if infective endocarditis is not treated, they may end up getting what?

remember think of how the vegetation plays a part in the body

A

heart failure

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24
Q

what are some manifestations secondary to septic embolism we can potentially see in a patient with a infective endocarditis ? (4)

josr!!

A

splinter hemorrhages in nail beds
( black longitudinal streaks )

osler nodes on fingertips or toes
( painful, red pea size lesson )

janeways lesions on pads of the fingers/toes
( flat, red spots )

Roths sports
( retinal lesions )

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25
what are some diagnostic studies for patients who have infective endocarditis ?
history blood cultures fluids rest valve replacement
26
typically the end treatment for patients who have infective endocarditis is to give them what? additional information if you are having procedures, you need to give antibiotics before hand because the risk of progressive disease is at risk
prophylactic antibiotics treatment
27
nursing assessment notes Objective data Fever Osler’s nodes Splinter hemorrhage Janeway’s lesions Petechiae, purpura Peripheral edema, clubbing Tachypnea, crackles Dysrhythmia, tachycardia, murmurs, S3, S4 Retinal hemorrhages
28
nursing diagnosis notes Decreased cardiac output Activity intolerance Hyperthermia Impaired comfort Knowledge deficit nursing management notes Assess for risk factors and physical evidence Patient will be on IV antibiotics for weeks Teach patient to rest DVT prophylaxis-compression stockings, ROM, Prevent atelectasis: turn, cough, deep breathe Q2 hours Patient to monitor temperature, look for signs of complications, finish antibiotic treatment
29
what is rheumatic fever and caused by? occurs ____(when) after____(what) how do we prevent rheumatic fever?
acute inflammation of the heart resulting from group a stream infection 2-3 weeks after strep infection proper treatment of strep infections
30
what is rheumatic heart disease ? what is the treatment for this and for how long?
chronic scarring and deformity of the heart valves 5 years and may need lifelong prophylaxis
31
valve disease section time !
32
you can have two types of valve conditions, which are?
stenosis regurgitation or insufficiency
33
what does stenosis mean ?
valve are stiff and do not open easily ( think a set of doors that sticks and must be pushed hard to open ) openings are smaller
34
what does regurgitation or insufficient mean ?
valve do not close completely
35
how does blood flow work for each stenosis regurgitation or insufficiency
forward blood flow backward blood flow
36
she mentions how we need to know the valve locations and where it is to listen to before we do that what are the 5 valves ?
aortic pulmonary erbs point tricupsid mitral
37
now give me the landmark location to where we are going to need to know where to listen to aortic pulmonary erbs point tricuspids mitral
2nd intercostal space to the right sternal border 2nd intercostal space to the left sternal border 3rd intercostal space to the left sternal border 4th intercostal space to the left sternal border (apex), 5th intercostal space, midclavical line (point of maximum impulse)
38
between stenosis and regurgitation or insufficiency, which one is worse and why ? and why
stenosis - because its very hard for the blood to even move compared to insufficiency - because it is at least moving, but just moving backwards
39
mitral valve stenosis goes from the what ?
left atrium to left ventricle
40
what is the most common cause for rheumatic heart disease ?
mitral valve stenosis
41
what is the result from mitral valve stenosis?
decreased blood flow from left atrium to left ventricle, blood can back up into pulmonary vasculature
42
so with mitral valve stenosis, we understand that the blood from the left atrium and left ventricle is decreased causing what to increase and putting risk of getting ?
increased in left atrial pressure/volume increase in pulmonary vasculature atrial fibrillation
43
what is the most important clinical manifestion for patient who have mitral valve stenosis ?
exertion dyspnea due to decreased lung compliance
44
what is mitral valve regurgitation ?
when the mitral valve doesn't close completely
45
what are some examples/causes of mitral valve regurgitation ?
myocardial infarction chronic rheumatic heart disease mitral valve prolapse ischemic papillary muscle dysfunction infective endocarditis
46
what is some acute mitral valve regurgitation clinical manifestations? remember with acute, the valve is not closing properly, so instead the blood is going backwards, so the patents are going to end up with ?
therady peripheral pulses tachycardia pulmonary edema - crackles - frothy
47
if we do not fix mitral valve regurgitation, what time of shock can they develop ?
cardiogenic shock
48
what are some chronic mitral vital regurgitation clinical manifestions ?
left ventricular failure after a long time - weakness, fatigue, palpitation
49
what is the treatment of mitral value regurgitation ?
fix the valve
50
what is mitral valve prolapse?
abnormality of mitral valve leaflets allowing leaflets to prolapse back into left atrium during systole - they can close, but they aren't in the space where its suppose to be
51
most of the time people who have mitral valve prolapse dont have what?
symptoms
52
how are we going to manage mitral valve prolapse ? (3)
antibiotics beta blockers - help with chest pain avoid stimulants
53
the aortic valve goes from you ___to your ?
left ventricle to your aorta
54
what is aortic valve stenosis ?
obstruction of blood flow from left ventricle to aorta
55
aortic valve stenosis clinical manifestations ?
angina dyspnea on exertion syncope on exertion
56
aortic valve insufficiciency manifestion?
dysonea fatigue angina
57
remember stenosis aortic is an emergency and needs to be fixed insufficiency can be delayed
58
what are some diagnostic things we can do?
ct scan echocardiogram cardiac catherterization
59
with aortic valve stenosis, we want to avoid giving nitroglycerin because why ?
reduces preload and blood pressure can worsen chest pain
60
what are some medications we are going to give to aid aortic problems ?
vasodilators positive inotropes diuretics beta blockers sodium restriction anticoagulation therapy antidysrhythmias
61
what is a procedure we can do for valvular heart disease ?
percutaneous transluminal ballon valvuloplasty
62
if we can't repair the valve, we have to replace it. mechanical (artifical) last longer, requires anicoagulations development of clot biologic (tissue) bovine (cow) porcine(pork) human no anticoagulation is needed, only last 10 years