Urgent Care Flashcards

1
Q

Systemic or Pulmonary insult that leads to pulmonary edema, alveolar collapse, and hypoxemia is known as _____.

A

ARDS

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2
Q

Sepsis, severe trauma, and aspiration of gastric contents are leading causes of ______ (hypoxemia refractory to O2 + organ failure).

A

ARDS

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3
Q

_____ (Prone/Supine) position helps to improve oxygenation in patients with ARDS.

A

Prone

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4
Q

The MC cause of cardiac failure is _____ (chronic condition).

A

CAD

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5
Q

Chronic causes of cardiac failure include:

A
  1. Dilated cardiomyopathy

2. Valvular dz

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6
Q

The most common rhythms seen in cardiac arrest are:

A
  1. V tach
  2. V fib

(also PEA and asystole)

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7
Q

Reversible causes of cardiac arrest include:

*Hint- H’s & T’s

A
  • Hypovolemia
  • Hypoxia
  • Hydrogen ion (acidosis)
  • Hypo-/hyperkalemia
  • Hypothermia
  • Tension PTX
  • Tamponade, cardiac
  • Toxins
  • Thrombosis, pulmonary
  • Thrombosis, coronary
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8
Q

____ is a normal GCS and ____ indicates severe TBI and the need for intubation.

A
  • 15–> normal

- < 8–> INTUBATE!

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9
Q

Give _____ then glucose when a patient is hypoglycemic with hx of EtOH abuse or malnutrition.

A

Thiamine

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10
Q

If you suspect an elevated ICP then elevate the head of bed 30 degrees and give _____ (drug).

A

Mannitol

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11
Q

Treatment of a local allergic reaction involves:

A

Ice, elevation, remove stinger, tylenol, ibuprofen

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12
Q

Treatment of urticaria involves:

A

Antihistamines, PO steroids (prednisone, medrol), consider EPI

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13
Q

Treatment of bronchospasm involves:

A
  1. Mild–> Moderate: Albuterol, parenteral steroids (methylprednisolone/solumedrol)
  2. Moderate–> Severe: Parenteral beta agonist (epinephrine), consider intubation
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14
Q

Treatment of Hypotension involves:

A
  • Etiology: histamine, PGE, and leukotrienes vasodilate peripheral vessels, inc capillary endothelial permeability and 3rd spacing
  • Treatment: IV NS or LR bolus, EPI or NE infusion; consider vasopressors (DA) for refractory hypotension
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15
Q

Treatment of Anaphylaxis involves:

A

-Etiology: IgE mediated

Treatment:
1. RSI, 2 large-bore IVs, continuous pulse ox, cardiac monitor

  1. Parenteral beta agonist (EPI), antihistamines, and glucocorticoids, IVF
  2. Obstructive Angioedema: Cricothyrotomy
    - Contraindicated if < 8 y/o
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16
Q

How is free air on CXR under the diaphragm and free fluid that is not ascites treated?

A

Urgent surgical referral!

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17
Q

What ABX are used for acute abdomen?

A
  • Levaquin-Flagyl combo
  • Zosyn
  • Also give aggressive IVF (1 L NS or LR per hr)
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18
Q

____ burns are the MC type.

A

Scald

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19
Q

_____ (treatment) is recommended for circumferential burns to prevent compartment syndrome.

A

Escharotomy

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20
Q

____ is used to treat superficial burns while _____ is used to treat 2nd-3rd degree burns (not including pregnant, <2mo, and face).

A
  1. Superficial- aloe vera and bacitracin

2. 2nd-3rd degree- Silvadene

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21
Q

What is the Parkland Formula?

A

Use to calculate IV Fluids for burns > 10%.

LR 4cc x Wt. (kg) x %BSA= amount given in 24hrs
-Half over first 8 hrs, half over subsequent 16 hrs

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22
Q

The treatment for chemical burns entails:

A

Continuous irrigation with H2O until pH= 7

*Ocular burns- Irrigate ASAP with NS or LR w/ morgan lens for at least 30 min. ABX- moxifloxacin and cycloplegics

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23
Q

Small surface injuries that may be associated with devastating internal injuries are related to which type of burn?

A

Electrical

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24
Q

Lichtenberg (fern like dendritic pattern on skin) is associated with what type of injury?

A

Lightning strike

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25
____ is the MC cause of death in smoke inhalation pts
PNA
26
Injury ABOVE the glottis is MC caused by ____.
Heat
27
Injury BELOW the glottis is MC caused by _____.
Chemical injury *Dx- fiberoptic bronchoscopy
28
How are inhalation injuries managed?
Mostly supportive: humidified O2, pulm physiotherapy, mucolytics, and bronchodilators Consider intubation IF- copious secretions, laryngeal edema
29
Cherry red skin color, agitation, and AMS with a potentially normal PaO2 and SaO2 are signs of _____.
Carbon monoxide poisoning
30
How do you treat CO poisoning?
100% O2
31
What are common OB causes of 3rd trimester bleeding (after 28 weeks)?
1. Abruptio placentae (MC)- premature separation of placenta--> abdominal pain and dark red blood 2. Placenta previa- placenta covers opening of cervix--> profuse, painless, bright red bleeding 3. Vasa previa- fetal blood vessels near opening of uterus--> painless vaginal bleeding w/ fetal distress 4. Uterine rupture 5. Premature labor
32
What are non-OB causes of 3rd trimester bleeding?
Genital tract lesions, infections, intercourse, friable cervix, cervical carcinoma
33
Rules for suturing areas of bites...
1. YES if: clinically NOT infected, <12 hrs old (<24 hrs on face), NOT on hand or foot 2. NO if: high risk for infection (crush injury, puncture wound, human/cat bite (unless on face), immunocompromised patient)
34
What is the treatment of choice for foreign body aspiration?
Bronchoscopy- used for object removal
35
What's the study of choice for an occult hip fx?
MRI
36
What's the study of choice for a pelvic, facial, or intraarticular fx?
CT
37
What IV abx are used for open fractures?
1st-2nd gen cephs and aminoglycosides *Don't forget tetanus shot!!
38
Common sites of dislocations?
Anterior shoulder, posterior hip, posterior elbow
39
Key features of Stable Angina are:
1. Substernal CP (pressure, burning, tightness) 2. Brought on by exertion 3. Nonpleuritic 4. Usually < 30 min, typically 1-5 min
40
How is stable angina managed?
Rest or NTG
41
Unstable angina, NSTEMI, and STEMI all fall under the umbrella of ______.
ACS- Acute Coronary Syndrome
42
UA and NSTEMI can be distinguished from STEMI by:
1. UA and NSTEMI- subtotal occlusion; STEMI- TOTAL OCCLUSION 2. UA and NSTEMI- ST depressions and/or T wave inversions; STEMI- ST elevations
43
Cardiac enzymes are _____ (positive/negative) in cases of UA and are ______ (positive/negative) in cases of NSTEMI.
UA= negative cardiac enzymes NSTEMI= positive cardiac enzymes
44
The MC cause of MI is:
Atherosclerosis- acute plaque rupture causing coronary thrombosis
45
The S/s of ACS include:
- Retrosternal "pressure" usually > 30 min! | - NOT RELIEVED WITH REST/NITRO
46
Silent MIs....
About 25% of MIs - More common in women, elderly, DM, and obese - S/s= abdominal, jaw pain, or dyspnea w/o CP
47
Natural STEMI progression on EKG=
Peaked T waves--> ST elevations--> Q waves--> T wave inversions
48
The most sensitive and specific cardiac marker for NSTEMI/STEMI is _____.
Troponin *Can also be elevated in cases of renal failure, advanced HF, acute PE, and CVA
49
How are cases of UA and NSTEMI managed?
1. Antithrombotic Therapy - Antiplatelet Meds a. ASA b. ADP Inhibitors (Clopidogrel) c. GP II/IIIa Inhibitors (Eptifibatide) - Anticoags a. Unfractioned Heparin b. LMWH (ADR: thrombocytopenia; renal dose) 2. Adjunctive Therapy and Assess RFs (TIMI) a. BBs (see p. 10 for CIs) b. Nitrates c. Morphine d. CCBs (DOC for vasospastic disorders)
50
How are STEMIs managed?
1. Reperfusion treatment - Do w/in 12 hours; either with PCI or thrombolytics - PCI best within 3 hours of symptom onset- ideally 90 min. - Door to thrombolytics- 30 min. 2. Antithrombotics - ASA- chew, dec. mortality by 20% - Heparin 3. Adjunctive therapy - BBs - ACEI - Morphine - K+ and Mg+ repletion, Statin therapy, Monitor BP, glucose, reduce RFs
51
What are the indications for CABG v. PCI?
CABG: - > 3 vessel dz. - L main coronary artery - Decreased LV EF
52
____ is DOC in cocaine-induced MI and _____ (drug) should be avoided.
CCB= DOC -AVOID BB b/c of vasospasm
53
You want to avoid IV ____ and _____ in cases of R Ventricular (Inferior) MI because these drugs may drop preload.
Nitrates and Morphine
54
If CP began 12+ hours ago you want to proceed with conservative management which entails:
ASA +/- Clopidogrel x 9 months, statin, BB, ACEI, Nitro PRN
55
What is a TIMI score and what is considered a high risk?
Risk score to assess risk of death and ischemic events in pts with UA/NSTEMI to determine benefit of invasive angiography to reduce mortality Score ≥ 3 = high risk!
56
Absolute CIs for use of Thrombolytics in ACS are:
- Intracranial hemorrhage - Non-hemorrhagic stroke w/in 6 months - Head/face trauma w/in 3 months - Intracranial neoplasm - Aneurysm - AVM -p. 11 for relative CIs
57
Transient ST segment elevations often seen in females > 50y/o and smokers and are associated with hyperventilation, emotional stress, and cold weather are known as ______.
Variant Angina
58
Management of Variant Angina and Cocaine-induced MI include:
CCBs or Nitro -ASA, heparin, and benzos (for cocaine-induced MI) may be given until atherosclerotic dz is ruled out
59
Increased BP with NO acute end organ damage is known as a ______.
Hypertensive Urgency
60
HTN Urgency is treated with:
Clonidine or Captopril to decrease BP by 25% over 24-48 hours Goal= < 160/100mmHg
61
Increased BP WITH acute end organ damage and BP > 180/120 is known as a ______.
HTN Emergency!
62
HTN Emergency managed by:
Decreased BP by 25% w/in the 1st hours and additional 5-15% over next 23 hours with IV meds
63
What medications do you want to use for an acute aortic dissection?
Esmolol, Labetalol
64
What medications do you want to use for HTN encephalopathy?
Nicardipine, Clevidipine, Labetalol
65
Classic triad for PE includes:
Dyspnea, Pleuritic CP, and Hemoptysis
66
MC predisposing condition for PE is _____.
Factor V Leiden
67
BREATHS acronym for suspected PE
``` Blood in sputum Room air sat < 95% Estrogen or OCP use Age > 50 y/o Thrombotic event in past- PE or DVT, Trauma HR > 100bpm Surgery in last 4 weeks ``` *PERC Criteria- if all the above are negative you effectively r/o PE
68
Workup for PE:
PE likely--> CT scan! *If CT scan is indeterminate then Pulmonary Angiography is the GOLD STANDARD (can also do LE US--> 70% of patients will also have DVT) PE UNlikely--> D-dimer -If negative then VTE/PE excluded
69
EKG findings associated with PE:
- Sinus tach and nonspecific ST/T changes MC | - S1Q3T3 most specific--> wide deep S in lead I; isolated Q and T wave inversion in lead III
70
Management of Stable PE:
IV UFH or SQ LMWH--> PO Warfarin or NOAC at least 3 months *No PTT monitoring with LMWH (protamine sulfate is antidote to UFH and LMWH) **If anticoag CI--> IVC filter
71
Management of UNstable PE:
Thrombolytic treatment: tPA (CI if CVA or internal bleed within 2 months) If Thrombolytics CI--> embolectomy
72
When is PE prophylaxis warranted?
Preop patients w/ prolonged immobilization, pregnant, hx of prior DVT/PE *LMWH: used if undergoing ortho, neuro sx, trauma
73
What are the 3 types of PTX?
1. Spontaneous- Due to bleb rupture a. Primary- NO underlying lung dz; tall and thin men that are 20-40, smokers, FHx of PTX b. Secondary- underlying lung disease w/o trauma (COPD, asthma) 2. Traumatic- iatrogenic: CPR, thoracentesis, PEEP, subclavian line placement, trauma 3. Tension- increased JVP, pulsus paradoxus, HYPOtension
74
Management of PTX involves:
- Observe at least 6 hours w/ repeat CXR to affirm no progression +24-48 hr f/u - If primary, spontaneous, and small then observation is okay. Usually resolves w/in 10 days. *Chest tube placement if large or severe symptoms **NEEDLE ASPIRATION for Tension PTX--> chest tube
75
Ingesting harmful substances
p. 14-15!
76
Orbital cellulitis...
- Usually secondary to sinus infxn (ethmoid 90%)--> S. aureus, S. pneumo, GABHS, H. flu - May be caused by dental/facial infxns - MC in kids 7-12 - S/s: decreased vision, pain with eye movement, proptosis, eyelid erythema, and edema
77
How is orbital cellulitis diagnosed?
CT scan or MRI
78
How is orbital cellulitis treated?
IV ABX- Vancomycin, clindamycin, cefotaxime
79
What is preseptal cellulitis?
Infection of eyelid and periocular tissue. May have ocular pain and swelling but NO visual changes and NO pain with eye movement
80
How is preseptal cellulitis treated?
Amoxicillin