Unit 8-Environmental Heath Ecotoxicology Flashcards

1
Q

What is ecotoxicology?

A

The study of toxins and their effects on organisms, and the environmental factors that influence exposure

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2
Q

What is one of the most common uses of toxins and is a basis for ecotoxicological studies?

A

Pesticides: It’s development, testing, and use

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3
Q

Pesticides are used to?

A

Protect crops, animals, and humans from damage and disease caused by “pests” and to remove weeds.

ex: herbicide, rodenticide, fungicide, insecticide

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4
Q

What is a pest?

A

An undesirable competitor that interferes with human welfare/activities

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5
Q

**What are the factors that affect toxicity of substances in the environment?

A
  1. Persistance
    -How long a chemical takes to break down in the environment
  2. Solubility
    -The ability of a chemical to dissolve in liquid
  3. Bioaccumulation and biomagnification
    -The build up of fat soluble chemicals in the body over time
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6
Q

_____ can be excreted from your body but may easily enter and accumulate in aquatic ecosystems

A

Water-soluble

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7
Q

_____ are absorbed into fatty tissues and build up in bodies

A

Fat -soluble

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8
Q

How is the food-chain/food-web affected by bioaccumulation/magnification?

A

Bioaccumulation leads to build up in each trophic level

Animals higher in the food web accumulate more toxins

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9
Q

**What factors affect toxicity?
1. Acute exposure

A

-Large exposure
-Symptoms develop rapidly

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10
Q

**What factors affect toxicity?
2. Chronic exposure

A

-Low-level exposure
-Takes place over a long period of time
-prolonged exposure

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11
Q

**What factors affect toxicity?
3. Age and sex

A

-Young and old are more susceptible
-Hormones interact with toxins

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12
Q

**What factors affect toxicity?
4. Antagonistic Effect

A

-Chemicals interact and cancel out/lessen the toxicity

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13
Q

**What factors affect toxicity?
5. Synergistic Effect

A

-Chemicals interact to create a pronounced effect and greater response than would be expected

ex: Nicotine + Asbestos increase cancer

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14
Q

Do toxins differ from their intended pathway in an ecosystem?

A

YES

A very low % reaches its target
98% can end up in air surface water, groundwater, or bottom sediments

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15
Q

What is the Mobility and Long Range Transport of (Air) Pollutants (LRTAP) also called? What is this effect?

A

The Grasshopper Effect because pollutants leap towards the poles with air currents

Substances are transported by wind and water and end up far away from the source
-ex: the arctic having the highest mercury concentrations

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16
Q

What does broadcast spraying/crop dusting result in?

A

Exposure of non-target species

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17
Q

What are indirect ecotoxicological stresses?

A

Changes in habitat due to purposely released toxins

-ex: herbicides kill plants, depriving herbivores of their preferred foods

18
Q

What is Genetic Resistance/Genetic Based Tolerance?

A

Mutant individuals are resistant to a pesticide. The mutant survives and contributes to the new population until eventually the mutant gene becomes dominant. Higher concentrations/new pesticides are required to affect the population

19
Q

How do you reduce resistance?

A

With active ingredients, rotating herbicides, and having multiple modes of action

20
Q

What is a risk?

A

The probability that exposure to a substance will be harmful

21
Q

**What are the factors of a risk assessment?

A
  1. Dose (determined in lab)
    -The amount that enters the body
  2. Exposure assessment (tested in the environment)
  3. Risk characterization (whether it poses a risk or not)
22
Q

What is a lethal dose?

A

Causes death

ex: LD50 causes death to 50% of the population

23
Q

What is a sub-lethal dose?

A

Has a measurable effect that is not death

24
Q

What is an effective dose?

A

Causes a specific response

ex: ED50 causes 50% of the population to exhibit a response

25
Which dose do you determine first?
Start with high doses then work your way down
26
What is the threshold level?
Where there is no observable effect aka a safe dose For some toxins there is no threshold, meaning there is no safe dose
27
How to measure risk- Hazard Quotient (HQ)
How toxic a substance is HQ= Exposure concentration (EEC)/Effect concentration (TBC) EEC: Expected environmental concentration determined from environmental samples/models TBC: Benchmark concentration determined from toxicity data (concentration where an effect is observed in an organism)
28
Values of HQ > 1 are considered to be?
Of concern. Toxic effects are expected to occur
29
Values of HQ < 1 are considered to be?
Low concern Toxic effects are not expected to occur
30
****What are Persistent Organic Pollutants? (POPs) Why are they risky?
Synthetic compounds resistant to degradation, that biomagnify, are mobile, and organic (fat soluble)
31
Most common example of a POP?
DDT Dichlorodiphenyltrichloroethane
31
POPs (DDT and PCB) were used in?
Manufacturing as insulators and coolants (in transformers)
32
What did Rachel Carson's book Silent Spring raise awareness of?
The harmful effects of DDT
33
What was the purpose of the Stockholm Convention? (2001)
To restrict the release and the clean up of the Dirty Dozen (the 12 worst POPs) Helped developing nations get rid of POPs
34
Is mercury naturally occuring?
Yes-meaning it is fat soluble
35
_____ enhances the release of mercury?
Human activities such as industry and gold mining
36
Once released, mercury accumulates where?
In the mud at the bottom of lakes and rivers
37
What transforms the mercury into organic methylmercury?
Bacteria
38
Is Methylmercury fat soluble?
Yes-making it difficult to excrete causing bioaccumulation and biomagnification in the food web
39
Mercury poisoning first occurred where?
In Japan -due to wastewater discharge -central nervous system disease
40
What is mercury poisoning from methylmercury also called?
Minimata disease
41
Why are mercury emissions in 2010 87% lower than 1990?
New technologies to capture mercury before it is released into the environment Provincial and federal legislation