Unit 6: Systems within Organisms Flashcards

1
Q

what are the 2 types of movement

A

movement within cells
locomotion (moving from one place to another)

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2
Q

motile vs sessile

A

motile = organisms that can do locomotion
sessile = organisms that remain in one location

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3
Q

what is the sliding filament model of muscle contraction

A

striated muscle fibres has the stripes. this is caused by something called a sarcomere

see B3.3.2 for structure

when contract, myosin pulls the actin filaments + z-line closer

see iPad for the model

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4
Q

what is titin

A

titin = connects myosin to the z-line
- biggest polypeptide
it is very elastic (has spring motion)
prevents z-lines from getting too far apart
makes muscle contractions stronger

when muscle is relaxed, titin is stretched
contract = spring recoil

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5
Q

role of antagonistic muscles in muscle relaxation

A

muscles that accomplish opposite movements
(i.e. bicep and tricep)

when one contracts, the other stretches, stretching the titin, which can recoil to make contraction stronger

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6
Q

structure and function of motor units in skeletal muscle

A

neuromuscular junction: where a motor neuron meets a muscle fiber

acetylcholine = transfers messages from neurons to muscles

motor unit = motor neuron + all of the muscle fibers it connects to (B3.3.4 iPad)
–> for coordinated contraction

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7
Q

what is the role of skeletons

A

muscles provide pulling force, skeletons provide ANCHORAGE points
- levers

Fulcrum = pivot point of a lever

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8
Q

movement at a synovial joint

A

see B3.3.6 on iPAD

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9
Q

range of motion in a joint

A
  1. hinge joint = can only flex or extend
  2. ball and socket joint = capable of rotation, adduction/abduction (side to side), protraction/retraction (up and down)

can be measured with a goniometer

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10
Q

internal and external intercostal muscles

A

inhaling:
ribcage up and out
external contract

exhaling:
ribcage in and down
internal contract

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11
Q

reasons for locomotion

A

finding food
finding a mate
escaping predators
migration

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12
Q

adaptations for swimming in marine mammals

A
  1. streamlined body (teardrop shape, no hair to reduce friction)
  2. airways (blowhole, mouth not connected to lungs)
  3. locomotion (fins, flippers, tails, blubber for buoyancy)
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13
Q

gas exchange in animals

A

universal function in organisms

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14
Q

properties of gas-exchange surfaces

A
  1. thin
  2. permeable to gases
  3. large surface area to volume ratio
  4. moist
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15
Q

maintenance of conc. gradients at exchange surfaces in animals

A

higher gradient = faster diffusion
needs constant blood flow
ventilation = moving air into and out of the lungs through dense networks of blood vessels
fish = move fresh water through gills

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16
Q

adaptations of mammalian lungs

A

diaphragm
intercostal muscles
abdominal muscles
ribs

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17
Q

ventilation of the lungs

A

inhaling:
1. diaphragm and external IM contract
2. abdominal and interior IM relax
3. inc. chest cavity volume
4. pressure decreases
5. air is forced into our lungs

exhaling:
1. diaphragm and external IM relax
2. abdominal and interior IM contract
3. dec. chest cavity volume
4. pressure increases
5. air is forced out of our lungs

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18
Q

measurements of lung volumes

A

ventilation rate: number of inhalations/exhalations per minute
tidal volume: volume of air inhaled/exhaled in each breath
vital capacity: maximum amount of air the lungs can hold
inspiratory reserve vole: amount of air a person can inhale after a normal breath
expiratory reserve volume: amount of air a person can exhale after a normal breath

can use a spirometer or a bell jar to measure lung volumes

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19
Q

adaptations for gas exchange in leaves

A

stomata = openings for gas exchange and water loss

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20
Q

distributions of tissues in a leaf

A

see b3.1.8

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21
Q

consequences of gas exchange in a leaf

A

transpiration = loss of water vapour from the leaves
higher temp = more transpiration
higher humidity = less transpiration

guard cells can open and close stomata to control water loss

can measure transpiration using a potometer

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22
Q

how to measure stomatal density

A

stomata/cm^2

23
Q

what is cooperative/allosteric binding of O2/CO2 to haemoglobin?

A

haemoglobin can transport 4 oxygen molecules
when oxygen binds to one of the haem groups, it causes a conformational change, increasing its affinity for oxygen
= cooperative binding

this is reversible

haemoglobin w/ 3 O molecules will have the greatest affinity for oxygen. w/ 4 O molecules = no affinity. w/ 0 O molecules = least affinity (besides 0)

allosteric binding
CO2 will bind to the polypeptide regions of the molecule, known as the allosteric site of the polypeptide
binding of CO2 = release of O molecules (BOHR SHIFT)

24
Q

adaptations of capillaries for exchange of materials between blood

A

capillaries = site of diffusion into and out of the blood
–> have a large surface area
tissues that need lots of oxygen or nutrients have a high-density capillary network

-pores = to increase permeability
- fluid that comes out of the capillaries = tissue fluid (water, oxygen, glucose, ions)
—> tissue fluid leaves the capillaries and flower between tissues
—> materials diffuse into tissues, waste diffuses into tissue fluid
—> fluid returns to capillaries

25
Q

foetal haemoglobin adaptations for transport of oxygen

A

Foetal haemoglobin has a higher affinity for oxygen than adult haemoglobin.

The capillaries in the placenta of a pregnant woman come very close to the capillaries of the foetus, allowing for molecular exchanges between the mother and foetus. Diffusion occurs due to the concentration gradient between the blood of the mother and foetus and the foetal haemoglobin’s greater affinity for oxygen, encourages diffusion of the mother’s oxygen to the foetus.

26
Q

what is the Bohr shift?

A

high carbon dioxide conc. reduces haemoglobin oxygen affinity. this is good for respiring tissues

27
Q

structure of arteries and veins

A

ARTERY:
- thick wall
- narrow lumen
- circular
- bumpy on the inside (inner corrugation)
- wall fibres visible

VEIN:
- thin wall
- wide lumen
- sometimes circular, but most of the time they are flattened
- no inner surface corrugation
- no wall fibres visible

28
Q

adaptations of arteries for the transport of blood away from the heart

A

narrow lumen = high pressure

thick, muscular wall = contract/recoil

elastic fibres = elastic/strong, help push blood through, less energy required than a full contraction

muscular walls = contract to push blood through

29
Q

adaptations of veins for the return of blood to the heart

A

wide lumen = low pressure, towards the heart

thin muscle wall = relies on the contraction of skeletal muscles
veins prevent backflow (valves)

30
Q

role of a coronary artery

A

bring oxygenated blood from the aorta to the heart tissue itself

if there is an occlusion = coronary heart disease
(blocked with plaque)
–> can lead to a myocardial infarction)

31
Q

adaptations of xylem vessels for water transport

A

dead hollow cells = maintenancee of a continuous water colume

lignin = prevents collapse

pits = water can pass through

xylem is on the INSIDE

32
Q

exchange of substances between tissue fluid and cells in tissues

A

oxygen diffuses into cells
glucose moves into cells through sodium-glucose cotransporters)
carbon dioxide and waste moves out of cells

33
Q

drainage of excess tissue fluid

A

85% of tissue fluid returns to capillaries, the other 15% drains into the lymphatic system, eventually draining back into the blood

34
Q

single vs double circulation

A

mammals = need a separate low-pressure loop to the lungs (pulmonary loop)

fish = can pump blood at high pressure to the gills

35
Q

what are pathogens

A

Pathogens are disease-causing organisms → typically bacteria, viruses, fungi, protists

36
Q

what is the role of skin and mucous membranes in defence against disease?

A

skin = physical AND chemical barrier

mucous membrane = sticky mucus that traps pathogens

37
Q

sealing of cuts in skin by blood clotting

A
  1. platelets (cell fragments) attach to site
  2. platelets release clotting factors
  3. prothrombin is converted to thrombin
  4. thrombin converts fibrinogen to fibrin
  5. fibrin forms a mesh to trap platelets and blood cells
38
Q

innate vs adaptive immune system

A

innate = phagocytes (constant throughout the organism’s life), not specific

adaptive = lymphocytes (builds memory/immunity through life), specific

39
Q

types of blood cells

A
  1. erythrocytes (red blood cells)
  2. leucocytes
    –> phagocytes
    –> lymphocytes
    ——> T-cells
    ——> B-cells
    ———-> plasma cells
    ———-> memory cells
40
Q

infection control by phagocytes

A

phagocytes, engulf pathogens via endocytosis

enzymes inside lysosomes destroy pathogens

41
Q

infection controlled by lymphocytes

A

lymphocytes produce antibodies

antibodies bind to antigens on pathogens
- tag it for destruction
- prevent it from binding w other host cells

antibodies are specific to antigens so we need a lot of diff. types of lymphocytes to make lots of diff types of antibodies

42
Q

what are antigens and their role?

A

recognition molecule on the surface of a cell/virus (are glycoproteins)

–> antigens stimulate immune responses

–> antibodies are specific to antigens

43
Q

blood group antigens

A

A: Anti-B, A antigen
B: Anti-A, B antigen
AB: no antibodies, A and B antigen
O: anti-A and anti-B, no antigens

44
Q

activation of B-cells by T-cells

A
  1. pathogens engulfed by phagocyte
  2. antigen presentation on the outside of macrophage
  3. t-cells bind to the antigen and become activated
  4. activated t-cell binds to specific lymphocyte
  5. b-cell activates
  6. b-cell will clone (mitosis)
  7. differentiate into plasma cells
    –> grow
    –> produce organelles for antibody production
  8. will secrete antibodies
45
Q

what is HIV

A

human immunodefiency virus

46
Q

what is AIDS

A

HIV –> AIDS
after HIV destroys helper T-cells
most people die from opportunistic infections, because no more T-cells

47
Q

what is an antibiotic

A

chemicals that disrupt prokaryotic metabolism (so, good for bacterial infection, not viral infections)

48
Q

evolution of resistance to several antibiotics in strains of pathogenic bacteria

A

resistance caused by mutation, antibiotics kills competitors, resistant strains become dominant

to prevent = only take antibiotics if needed, develop new antibiotics, restrict use of antibiotics for farm animal growth

49
Q

zoonoses

A

infectious diseases that can transfer from other species to humans

i.e. tuberculosis = cattle (from raw milk),
rabies = dogs (from bite/scratch),
covid-19 = bats to humans
japanese encephalitis = pigs or birds passed by mosquitos

50
Q

when does transpiration stop

A

outside air is humid
stomata closes at night
if the plant has lost its leaves

51
Q

root pressure generation

A

active transport of mineral ions into the root, water will move into the xylem. increase in pressure

52
Q

what is a companion cell

A

helps pump carbon compounds into sinks and into phloem out of source

53
Q

what is a phloem sieve tube

A

allows carbon compounds through