Unit 5 -Neuro Flashcards

1
Q

seizure

A

Brief abnormal nurst of electrical activity in the brain that alters one or more of the following:
movement, sensation, behaviour, awareness

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2
Q

what causes someone be diagnosed with a seizure disorder

A

2 or more seizures 24 hours apart. Or if they have one that puts them at high risk

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3
Q

Pre ictal phase of a seizure

A

Warning signs before a seizure occurs.
Ora: Sensory, motor, emotional symptoms

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4
Q

Ictal phase

A

seizure activity seconds to minutes, abnormal activity

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5
Q

Post ictal phase

A

recovery, confusion, fatigue, mins to hours

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6
Q

age related seizure causes

A

Children: Birth trauma, infection, febrile, congenital abnormalities
Middle years: Trauma, inf, alcohol, drugs
Elderly: CVA and metastatic brain tumour

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7
Q

seizure causes

A

brain tumour, trauma, vascular disease, borth hypoxia

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8
Q

Possible seizure triggers

A

specific time of day or night, sleep deprivation, fever or other illness, alcohol, stress, hormonal/metabolic, specific foods, meds taking/not taking, accident/injury, senses (light, smell), hypoglycemia, dehydration, over exertion

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9
Q

seizure lab work

A

decreased NA and Cam Hypoglycemia, kidney function,

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10
Q

Partial seizure

A

electrical disturbance limited to 1 area of the brain.
Simple: awareness, consciousness, and memory perserved less then 1 minute.
Complex: altered consiousness for more then 1 minute. Post-ictal. Often involve lip smoking and repetitive movement.

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11
Q

Generalized seizure

A

Electrical disturbance affects the entire brain.
Tonic: Decreased LOC, tonic stiffening, clonic jerking, entire body stiffens as the mucles contract. Absence: rarely beyond adolescence.

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12
Q

tonic phase presentation adn duration

A

LOC. May cry out. Fall down. Entire body stiffens as the muscle contract. cyanosis.
10-20 sec

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13
Q

Clonic phase presentation and duration

A

Jerking of extremities, cyanosis, excessice salivation, tongue/cheek biting, incontinance

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14
Q

Post seizure presentation

A

groggy, muscle soreness, no recall of event, tired, sleepy, can be aggressive. varies in how long it lasts,

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15
Q

How to protect your patient from injury during seizure

A

if they are upright ease to floor, portect head with padding, push aside any furnature that is in the way.
Raise side rails, bed at lowest position, DO NOT open jaws that are clenched and insert anything in, restain the pateint in any way

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16
Q

Seizures mimic

A

migraine, syncope, stroke, non epipetic spell, movement disorder, night terrors, panic attacks

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17
Q

seizure precautions

A

working O2, suction, oral airway HOB, IV access, rectal temp, bed in lowest position, padded rails, brain on, patient on side, privacy

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18
Q

Observe and record what during seizure

A

time, what they are doing, aura, triggering events, seizure progression and symmetry, eyes

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19
Q

Post ictal nursing management

A

ensure patent airway, check for injury especally tongue and oral cavity, suction PRN, IV access, NPO, remove loosen tight clothing, document, offer emotional support

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20
Q

Epilepsy

A

2 or more unprovoked seizures seperated by atleast 24 hours, recurring seizure. Only 1% of people will suffer from epilesy

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21
Q

when is a seizure an emergency

A

longer then 5 mins, 2nd without regaining consciousness, repeated seizure over a 30 minute perios

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22
Q

AED

A

Anti epileptic drug, effective in 70% of patients, take as perscribed, do not suddently stop,

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23
Q

Nursing managment for epilepsy

A

Health promotion, ensure treatment, assess progress, involve the family, have a predetermined plan for seizure

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24
Q

Nursing priorites for epilepsy

A

ABC
-prevent or control seizure, protect pateint from injury, maintain airway or resp function, promote positive self esteem, provide info about disease prognosis

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25
Q

How long should someone be seizure free until driving

A

3-12 months

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26
Q

water safety for seizures

A

take shwers not baths, shower chair or sit in the bottom of an unfilled tip. swimming safety

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27
Q

OPA

A

used for airway through the mouth. Measure jawline angle across, invert, insert, noninvasive. For people with no gauge reflex.

28
Q

NPA

A

Used for intact gag , though nose, better tolerated.

29
Q

MS

A

Chronic, progressive, degenerative autoimmune disorder of the CNS characterized by disseminated demyelination of nerve fibres of the brain, spinal cord, optic nerve

30
Q

Immunologic causes of MS

A

MS Causes damage to the protective fatty coating that surrounds nerve cells, causing signals from the cells to slow down or stop all together

31
Q

Environemental causes of MS

A

People exposed to more vit D help prevent MS

32
Q

MS women to men

A

3:1 women to men

33
Q

When do people develop symptoms

34
Q

Neuro effect of MS

A

difficulty speaking, involuntary eye movement, tremors, double vision, weakness, decreased bowel and bladder function, blurred vision

35
Q

patho of MS

A

autoimmune disorder, brain and spinal cord lined by myelin sheath. Body perceives its own myeline as an intruder adn attacks it. Causes inflammation, degeneration and scarring of the myelin, electrical impulses impaired

36
Q

Types of MS

A

Benign: minimal disability between attacks 10-20%
Relapsing-remitting: Attacks may leave permanent deficits followed by periods of remission 75%
Primary progressive: steady increase in disability. Older age of onset and no relapse. 10-20%
Secondary progressive. Initail RRMS that begins to decline wihout perios of remission
Chronic preogressive stead decline since disease onset

37
Q

favorable prognosis

A

women, under 40, low relapse yearly, long interval between attacks, young age of onset, low disability at 2-5 yeats from disease onset, later cerebral involvment

38
Q

Unfavorable prognosis

A

Male, over 40, high rate of relapse, short interval between attacks, symptoms from efferent (motor tract), Older age at onset, earlier cerebellar involvement

39
Q

Clinical manifestation for MS

A

Involuntary eye movement, vision loss, abnormal eye alignment, double vision, muscular atrophy, spasm, lack of muscle coordination, heightened pain, reduced sense of touch, constant tingling and burning, fatigue, cognitive impairment, depression, unstable mood, hearing loss, stuttering, diffculty swallowing, incontinance, diarreah.

40
Q

steps to diagnose MS

A

History, clinical manifestation, MRI, (this is the gold standard) Presence of multiple lesions over time, Spinal can also be done.

41
Q

How MS progresses

A

1.0: No disability, minimal signs of ms
3.0: Moderate disability in one system, or mild disability in up to 4 systems. No impairment of walking
5.0: Disability severe ebough to impair full daily activities able to walk without aid for 200 m
6.0: Requires a walk aid, cane, or crutch to walk 100 m
7.5: Unable to take more then a few steps. Restricted to wheelcahri usage.
9.0: Confined to bed. Can still communicate and eat.
10: DEATH

42
Q

Frontal lobe symptoms of MS

A

Speech, mood, though, planning, language

43
Q

Occiptal lobe symtpoms of MS

44
Q

Symptoms of MS in temporal lobe

A

Memory and emotion

45
Q

Symptoms of MS in Brain Stem

A

Breathing, regulating heart rate, seeing and hearing, sweating, BP, Digestion, Temp regulation

46
Q

Symptoms of MS in Cerebellum

A

Coordination and body movement, regu;ates balance and equilibrium

47
Q

Goals of MS treatment

A
  1. Stop acute relapse by steroid use
  2. DMT (Disease modifying therapies) Disease progression
  3. Symptoms management
    -maintain immune system. find therapies that supress the immune system enought to decrease immune attack.
48
Q

Can you cure MS

A

No, only treat symptoms

49
Q

Nursing management in MS

A

Maximise neuromuscular function, maintain indépendance for as long as possible, manage associated symptoms

50
Q

Assessment for MS

A

Symptoms, Trigger (Infection), trauma, immunization, post partum, stress, change in climate

51
Q

Drug therapy for MS

A

Corticosteriods, immunosupressice drugs, immunomodular drugs

52
Q

Nutrition for MS

A

Well balanced, high protein (helps with tissue repair), Extra Vit C to keep urine acidity, Hifh roughage,

53
Q

Physical therepies for MS

A

prevent contractures, water exercise, stretching, balance, assistive device

54
Q

Patho of parkinsons disease

A
  1. progressice neurodengerative disorder
  2. progressive destruction of dopaminergic nerurons in the basal ganglia and substantial nigra
  3. Results in domapine deficency and degeneration of muscular function affectign movement, mood, cognition, swallowing, and communication.
55
Q

Bradykinesia

A

slow movement and speed (common in parkinsons)

56
Q

Dyskinesia

A

Irradic involuntary movement,

57
Q

Non modifiable and modifiable risk factors for Parkinson’s

A

Non-modifiable:
age, mal, fam hx
Modifiable:
exposure to toxins, head trauma

58
Q

Assessment of TRAP for parkinsons

A

T: Tremor (pill rolling hand movement, often asymmetric, inhitibited during sleep) Progressive
R: Rigidty (jerky, facial, can’t swallow)
A: Akinesia/Bradykinesia (Slow/absent movement)
P: Postural instability (Unable to stand, hold posture)

59
Q

Symptoms of parkinsons to assess/monitor

A

-Aspiration risk
-Motor exam
-Psychosocail
-HEENT
-Nutrition
-ABDO (constapation and incontinance)
-Derm
-Neuro exam (LOC, language, cranial change)
-Coordination
-sensory
-gait/posture assessment

60
Q

Drug therapy for parkinsons

A

Dopaminergic (enahance the release or supply of dopamine to decrease tremors, rigidity, bradykinesia) Levodopa with cardipo are common

Anticholergic (Antagonize or block the effect of acetylcholine) and decrease tremors (cogentin is common)

Antihistamins (bendryl) Can manage tremors

MOA inhibitor (selegilene) used to slow down the breaksown of dopamine thereby increase teh doapmine levels

61
Q

Investiagtions that must be done before diagnosis of parkinsons

A

Blood work (rule out any other reason they may be experiencing tremor/other symptoms) Radiology/imaging looking for lesions, Psych (depression is high in these pateints(

62
Q

How long do parkinson medication usually work

A

for around 4-6 years and then they wear off

63
Q

Surgical therapies for parkinsons

A

Deep brain stimulation (surgical implanted medical device that delivers electrical stimulation to the brain)
Transplantation fetal neural tissue stem cells transplanted

64
Q

Nurse teaching for parkinsons

A

med: adherance, side effects
Nutrition: position when eating, smaller more frequent, soft foods or thickened fluids, supplimenets
Fall risk
Psychosocial

65
Q

Indications for hosptialization for parkinsons

A

Neuropsychiatric disturbances, delerium, frequent falls, failure to cope at home

66
Q

Complications with parkinsons

A

psychiatric: depression, hallucinations, psychosis
Cognitive: dementia
Autonomic dysfunction: hypotension, urinary urgency, constapation
Sleep disturbances: REM behaviour disorder, insomnia
Posture instability: balance and fall risk
Med related: dyskinesia