Unit 3.3: CV Pathophysiology Flashcards
Review
perioperative risk factors for MI
Review
risk of perioperative MI
Review
NYHA Heart Failure classification
what degree of surgical risk is: Breast Surgery
A. High risk
B. Intermediate Risk
C. Low risk
C
what degree of surgical risk is: cataract Surgery
A. High risk
B. Intermediate Risk
C. Low risk
C
what degree of surgical risk is: endoscopic Surgery
A. High risk
B. Intermediate Risk
C. Low risk
C
what degree of surgical risk is: peripheral vascular Surgery
A. High risk
B. Intermediate Risk
C. Low risk
A
what degree of surgical risk is: orthopedic Surgery
A. High risk
B. Intermediate Risk
C. Low risk
B
what degree of surgical risk is: CEA Surgery
A. High risk
B. Intermediate Risk
C. Low risk
B
what degree of surgical risk is: intrathoracic Surgery
A. High risk
B. Intermediate Risk
C. Low risk
B
what degree of surgical risk is: emergency Surgery
A. High risk
B. Intermediate Risk
C. Low risk
A
what degree of surgical risk is: head and neck Surgery
A. High risk
B. Intermediate Risk
C. Low risk
B
what degree of surgical risk is: open aortic Surgery
A. High risk
B. Intermediate Risk
C. Low risk
A
30%
0.3%
Review
O2 delivery vs o2 demand
Review
O2 delivery vs o2 demand chart
Review
MYOCARDIAL ISCHEMIA: BIOMARKERS
Detection of LV ischemia: combination of leads ___, ____, and _____ has an ischemic detection rate of up to 96%
II, V4, V5
Review
treatment of ischemia
CK-MB
- V3
- V4
- V5
hypocapnia will
A. decrease myocardial O2 delivery
B. increase myocardial O2 demand
A
increase in wall tension will
A. decrease myocardial O2 delivery
B. increase myocardial O2 demand
B
SNS stimulation will
A. decrease myocardial O2 delivery
B. increase myocardial O2 demand
B
decreased aortic. pressure will
A. decrease myocardial O2 delivery
B. increase myocardial O2 demand
A
anemia will
A. decrease myocardial O2 delivery
B. increase myocardial O2 demand
A
hypertension will
A. decrease myocardial O2 delivery
B. increase myocardial O2 demand
B
decreased P50 will
A. decrease myocardial O2 delivery
B. increase myocardial O2 demand
A
increased contractile force will
A. decrease myocardial O2 delivery
B. increase myocardial O2 demand
B
Review
diastolic compliance
Review
myocardial ischemia
A. Increases myocardial compliance
B. decreases myocardial compliance
B
chronic aortic insufficiency
A. Increases myocardial compliance
B. decreases myocardial compliance
A
dilated cardiomyopathy
A. Increases myocardial compliance
B. decreases myocardial compliance
A
aortic stenosis
A. Increases myocardial compliance
B. decreases myocardial compliance
B
pericardial tamponade
A. Increases myocardial compliance
B. decreases myocardial compliance
B
old age
A. Increases myocardial compliance
B. decreases myocardial compliance
B
hypertrophic obstructive cardiomyopathy
A. Increases myocardial compliance
B. decreases myocardial compliance
B
Review
decreased compliance
Review
HF classification and etiology
Review
pathophysiologic changes that accompany CHF
Review
Anesthetic management of HF
Review
conditions that increase PVR
Review
treatment of RV Failure
Review
HTN diagnosis
REVIEW
Cerebral aurtoregulation
texts recommend delaying surgery if:
SBP:
DBP:
SBP: > 180 mmHg
DBP: > 110 mmHg
Hypertensive crisis is a BP of
180/120
Review
HTN: Secondary Causes
what is the etiology of the clinical finding:
Upper limb BP > lower limb BP
A. Cushingsd syndrome
B. Pheochromocytoma
C. Coarctation of aorta
D. Conn’s Disease
C
what is the etiology of the clinical finding:
Diaphoresis
A. Cushingsd syndrome
B. Pheochromocytoma
C. Coarctation of aorta
D. Conn’s Disease
B
what is the etiology of the clinical finding:
truncal obesity
A. Cushingsd syndrome
B. Pheochromocytoma
C. Coarctation of aorta
D. Conn’s Disease
A
what is the etiology of the clinical finding:
systolic bruit
A. Cushingsd syndrome
B. Pheochromocytoma
C. Coarctation of aorta
D. Conn’s Disease
C
what is the etiology of the clinical finding:
hypokalemia
A. Cushingsd syndrome
B. Pheochromocytoma
C. Coarctation of aorta
D. Conn’s Disease
D
what is the etiology of the clinical finding:
headache
A. Cushingsd syndrome
B. Pheochromocytoma
C. Coarctation of aorta
D. Conn’s Disease
B
what is the etiology of the clinical finding:
Moon Face
A. Cushings syndrome
B. Pheochromocytoma
C. Coarctation of aorta
D. Conn’s Disease
A
what is the etiology of the clinical finding:
alkalosis
A. Cushingsd syndrome
B. Pheochromocytoma
C. Coarctation of aorta
D. Conn’s Disease
D
Review
Drugs that target the ANS
Review
Drugs that target the Myocardium and Vascular smooth muscle
Review
Drugs that target the kidney
what drug class:
prasozin
A. Alpha 1 antagonist
B. Alpha 2 agonist
C. CCB
A
what drug class:
diltiazem
A. Alpha 1 antagonist
B. Alpha 2 agonist
C. CCB
C
what drug class:
phenoxybenzamine
A. Alpha 1 antagonist
B. Alpha 2 agonist
C. CCB
A
what drug class:
clevidipine
A. Alpha 1 antagonist
B. Alpha 2 agonist
C. CCB
C
what drug class:
clonidine
A. Alpha 1 antagonist
B. Alpha 2 agonist
C. CCB
B
what drug class:
phentolamine
A. Alpha 1 antagonist
B. Alpha 2 agonist
C. CCB
A
what drug class:
Verapamil
A. Alpha 1 antagonist
B. Alpha 2 agonist
C. CCB
C
what drug class:
dexmedetomidine
A. Alpha 1 antagonist
B. Alpha 2 agonist
C. CCB
B
what drug class:
captopril
A. ACE-I
B. ARBs
C. K sparing diuretic
A
what drug class:
enalapril
A. ACE-I
B. ARBs
C. K sparing diuretic
A
what drug class:
benazapril
A. ACE-I
B. ARBs
C. K sparing diuretic
A
what drug class:
valsartan
A. ACE-I
B. ARBs
C. K sparing diuretic
B
what drug class:
amiloride
A. ACE-I
B. ARBs
C. K sparing diuretic
C
what drug class:
triamterene
A. ACE-I
B. ARBs
C. K sparing diuretic
C
what drug class:
azilsartan
A. ACE-I
B. ARBs
C. K sparing diuretic
B
what drug class:
losartan
A. ACE-I
B. ARBs
C. K sparing diuretic
B
what are the 3 types of voltage-gated calcium channels
Review
MOA of CCBs
Pines Die Very Smoothly
Aka the CCB ending in “-pine” are “di”hydropyridines and act on vascular smooth muscle
CCB impair contractility in the following order (highest to lowest):
- diltiazem
- nifedipine
- verapamil
- nicardepine
verapamil > nifedipine > diltiazem > nicardpine
Review
CCBs and Vascular tone
Review
CCBs and heart rate
t or F: clevidipine does not impair cardiac contractility
F
sort to the drug class
verapamil
A. Dihydropyridines
B. Non-Dihydropyridines
B
sort to the drug class
diltiazem
A. Dihydropyridines
B. Non-Dihydropyridines
B
sort to the drug class
nimodipine
A. Dihydropyridines
B. Non-Dihydropyridines
A
sort to the drug class
nifedipine
A. Dihydropyridines
B. Non-Dihydropyridines
A
REVIEW
Pericarditis pathophysiology
Pericardial knock is a sign/symptom of
A. Constrictive pericarditis
B. Acute pericarditis
A
Kussmaul’s Sign is a sign/symptom of
A. Constrictive pericarditis
B. Acute pericarditis
A
Pulsus Paradoxus is a sign/symptom of
A. Constrictive pericarditis
B. Acute pericarditis
A