Unit 2 Exam Flashcards

1
Q

Interventions based off of Acute Phase of healing

  • Acute/Protective phase 7-10days
A
  • Intervention goals are to :
  • control inflammation
  • avoid painful positions
  • Minimize pain and edema
  • restore full passive ROM
  • Maintain soft tissue joint integrity
  • Reduce muscle atrophy through gentle isometric muscle setting
  • maintain aerobic fitness
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2
Q

Interventions based off of Subacute Phase of Healing

  • Subacute/controlled motion 10 days - 6 weeks
A
  • Progressively stressing the healing tissue structures
  • Modify faulty joint mechanics
  • protect forming collagen
  • direct orientation parallel to the lines of force it must withstand
  • prevent crosslinking and scar contracture
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3
Q

Interventions based off of Chronic phase of healing

Chronic/return to function phase 6weeks-months

A
  • Gradual return to full pain free ROM
  • progressively increasing movement speed
  • Developing neuromuscular control
  • By end of the phase the full and unrestricted ROM should be present
  • More aggressive work related and sport specific movements and activities should be incorporated as appropriate
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4
Q

Low to high potential for healing tissues

A
  1. cartilage
  2. meniscus
  3. ligament
  4. Tendon
  5. bone
  6. muscle
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5
Q

Acute/Protective phase timeline

A

7-10days

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6
Q

Subacute/Controlled Motion phase timeline

A

10 days to 6 weeks

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7
Q

Chronic/ Return to Function

A

6 weeks to months

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8
Q

Strain grades

A
  • Grade 1 = pain only limited swelling, few tears, no loss of function
  • Grade 2= Increased pain, some loss of strength/function, disruption of moderate amount of fibers
  • Grade 3= complete rupture of musculotendinous units, loss of function
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9
Q

What is a strain?

A
  • Distractive strain is excess pull/overstretch
  • Risk factors include= inadequate flexibility/ strength/rehab from previous injury, muscle imbalances, insufficent warm up, fatigue
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10
Q

Tendinitis

A
  • Swelling, pain, dysfunction of the tendon
  • Has no PG mediated inflammation
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11
Q

Tendinosis

A
  • Degeneration of tendon structures
  • Pain isn’t always present

4 main Histological Changes
1. Angioblast hyperplasia
2. Disorganized immature collagen
3. Vascular hyperplasia and neovascularization
4. Hypercellularity

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12
Q

NSAIDs

A
  • Nonsteroidal anti-inflammatory drug
  • Acute/Chronic MSK disorders its used for
  • Decreases inflammation/blocks vasodilation and inflammatory response
    -Nonselective
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13
Q

Osteoporosis

A

A chronic progressive disease characterized by low bone mass and microarchitectural deterioration of bone tissue leading to decreased bone strength enhanced bone fragility and increase in fracture incidence

  • High bone turnover rate leads to weakening due to weaker trabecular/cancellous bone
  • New bone formation falls behind resorption due to declining osteoblast function
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14
Q

What are the treatments and preventions of Osteoporosis

A
  1. Nutrition= calcium/vitamin D
  2. Vitamin K can help bone metabolism
  3. Magnesium may increase BMD in the elderly
  4. Exercising, gait training, pain management
  5. Weightbearing exercises, flexibility exercises, postural/balance
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15
Q

What exercises are contraindicated for osteoporosis

A
  • Flexion exercises because it can lead to vertebral fracture by placing compressive forces on the anterior part of the vertebrae
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15
Q

Major Risk factors of osteoporosis

A
  1. Body weight < 70kg or <21 BMI
  2. Corticosteroids
  3. personal history of fractures as an adult
  4. First degree relative with osteoporosis
  5. Currently smoking
16
Q

Where does articular cartilage get its nutrition from ?

A

diffusion from the synovial fluid

17
Q

How do ligaments get their blood supply?

A

Epiligament layer

18
Q

Intraarticular Ligament

A

Inside the joint capsule
- Encased in synovium
- Blood dissipates into the synovium
Hematoma prevented
- Limited arrival of growth factors and cytokines needed to mediate inflammation and healing
- Doesn’t follow typical tri-phasic healing timeframes

19
Q

Extraarticular Ligament

A

Outside of the joint capsule
- Epiligament layer= highly vascular/cellular/ sensory and proprioceptive nerves
- greater likelihood of healing without surgery
- Follows typical triphasic healing timeframes

20
Q

Neural Zone

A

Little to no resistance to movement

21
Q

Plastic zone

A

permanent deformation and may lead to injury

22
Q

Elastic zone

A

First barrier to motion encountered

23
Q

Hypermobile

A
  • generalized multi-joint
  • Localized hypermobility
  • Instability
  • Laxity + symptoms associated with inability to control joint during movement
24
Q

Flexibility

A

Function of contractile tissue length/resistance –> can also function of joint passive restraints

25
Q

laxity

A

State of more than typical motion present at a joint through not a problem unless associated with symptoms

26
Q

Peak growth rate of boys and girls

A

Boys 14-15
Girls 12-13

27
Q
A